scholarly journals The physiological uptake pattern of 18F-FDG in the left ventricular myocardium of patients without heart disease

2000 ◽  
Vol 40 (1.2) ◽  
pp. 53-58
Author(s):  
Hayato Nose ◽  
Hideki Otsuka ◽  
Yoichi Otomi ◽  
Kaori Terazawa ◽  
Shoichiro Takao ◽  
...  
2014 ◽  
Vol 61 (1.2) ◽  
pp. 53-58 ◽  
Author(s):  
Hayato Nose ◽  
Hideki Otsuka ◽  
Yoichi Otomi ◽  
Kaori Terazawa ◽  
Shoichiro Takao ◽  
...  

Author(s):  
Wojciech Tański ◽  
Paweł Gać ◽  
Angelika Chachaj ◽  
Małgorzata Sobieszczańska ◽  
Rafał Poręba ◽  
...  

Abstract Background The aim of the study was to assess the relationship between the occurrence of rheumatoid arthritis (RA) and ankylosing spondylitis (AS) and the cardiac magnetic resonance (CMR) changes in people without clinically overt heart disease. Methods The study group consisted of 74 people (48.81 ± 11.35 years): 29 patients with RA, 23 patients with AS and 22 people from control group. Blood samples were taken to assess laboratory parameters, disease activity was determined using activity scales, and CMR was performed. Results It was shown that the factors independently related to higher left ventricular mass index are AS occurrence, human B27 leukocyte antigen occurrence, higher neutrophil gelatinase–associated lipocalin concentration (NGAL) and higher body mass index (BMI). The lower right ventricular ejection fraction is result of an independent effect of RA, AS and higher NGAL. RA presence, methotrexate use, higher rheumatoid factor titer, higher NGAL, older age and higher BMI should be considered independent risk factors for greater left ventricular myocardium water content. RA occurrence, AS occurrence, type 2 diabetes occurrence and a higher C-reactive protein concentration can be independently associated with a higher probability of non-ischemic left ventricular myocardium injury. Larger pericardial fluid volume is result of an independent effect of higher NGAL, higher anti-cyclic citrullinated peptide antibodies titer and higher DAS28 disease activity index. Use of steroids is protective factor against larger volume of pericardial fluid. Conclusions RA and AS in people without clinically apparent heart disease are associated with the occurrence of adverse changes in CMR. Key Points•RA and AS in people without clinically apparent heart disease are associated with the occurrence of adverse changes in CMR..•The independent risk factors for higher LVEF are AS occurrence, human B27 leukocyte antigen occurrence, higher NGAL concentration and higher BMI..•RA presence, methotrexate use, higher RF, higher NGAL, older age and higher BMI are independent risk factors for higher LV T2 ratio..•RA occurrence, AS occurrence, type 2 diabetes occurrence and a higher CRP are independently associated with a higher risk of non-ischemic LV myocardium injury..


2017 ◽  
Vol 2017 ◽  
pp. 1-3
Author(s):  
Selcuk Ozturk ◽  
Ertan Yetkin

Ice pick headache is a momentary, transient, repetitive headache disorder and manifests with the stabbing pains and jolts. The exact mechanism causing this disease is unknown. Premature ventricular contractions are early depolarization of the ventricular myocardium and in the absence of a structural heart disease, it is considered to be a benign disease. In this report, we describe a male patient presenting with the symptom of momentary headache attacks accompanied with instant chest pain which is associated with premature ventricular contraction.


2015 ◽  
Vol 43 (3) ◽  
pp. 513-518 ◽  
Author(s):  
Lisa McArthur ◽  
Lisa Chilton ◽  
Godfrey L. Smith ◽  
Stuart A. Nicklin

Gap junctions are channels which allow electrical signals to propagate through the heart from the sinoatrial node and through the atria, conduction system and onwards to the ventricles, and hence are essential for co-ordinated cardiac contraction. Twelve connexin (Cx) proteins make up one gap junction channel, of which there are three main subtypes in the heart; Cx40, Cx43 and Cx45. In the cardiac myocyte, gap junctions are present mainly at the intercalated discs between neighbouring myocytes, and assist in rapid electrical conduction throughout the ventricular myocardium. Fibroblasts provide the structural skeleton of the myocardium and fibroblast numbers significantly increase in heart disease. Fibroblasts also express connexins and this may facilitate heterocellular electrical coupling between myocytes and fibroblasts in the setting of cardiac disease. Interestingly, cardiac fibroblasts have been demonstrated to increase Cx43 expression in experimental models of myocardial infarction and functional gap junctions between myocytes and fibroblasts have been reported. Therefore, in the setting of heart disease enhanced cardiac myocyte: fibroblast coupling may influence the electrical activity of the myocyte and contribute to arrhythmias.


2019 ◽  
Vol 38 (3) ◽  
pp. 328-331 ◽  
Author(s):  
Pierre-Emmanuel Noly ◽  
Peter Dorfmüller ◽  
Elie Fadel ◽  
Olaf Mercier

Kardiologiia ◽  
2015 ◽  
Vol 4_2015 ◽  
pp. 5-13
Author(s):  
A.M. Chernyavsky Chernyavsky ◽  
O.S. Efanova Efanova ◽  
V.U. Efendiyev Efendiyev ◽  
D.A. Sirota Sirota ◽  
E.M. Alyapkina Alyapkina ◽  
...  

2005 ◽  
Vol 15 (4) ◽  
pp. 434-436 ◽  
Author(s):  
Dursun Alehan ◽  
Omer Faruk Dogan

Ventricular noncompaction is a rare unclassified cardiomyopathy occurring because of arrest of the normal intrauterine compaction of the loose luminal component of the ventricular myocardium. There is limited data regarding its diagnosis and outcome in children. It is recognised, however, that right ventricular involvement is extremely rare. We report a case in which only the right ventricular myocardium was noncompacted, a situation which led to heart failure soon after birth.


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