Are Cytokines Linked to Collagen Breakdown During Periodontal Disease Progression?

2003 ◽  
Vol 74 (2) ◽  
pp. 196-201 ◽  
Author(s):  
Anne-Laure Ejeil ◽  
Frederick Gaultier ◽  
Sylvie Igondjo-Tchen ◽  
Karim Senni ◽  
Bernard Pellat ◽  
...  
Keyword(s):  
Periodontal Disease ◽  
Disease Progression

Regenereation in periodontics

2018 ◽  
Author(s):  
Murtaza Kaderi ◽  
Mohsin Ali ◽  
Alfiya Ali ◽  
Tasneem Kaderi
Keyword(s):  
Tissue Engineering ◽  
Clinical Practice ◽  
Periodontal Disease ◽  
Disease Progression ◽  
Tissue Regeneration ◽  
Surgical Procedures ◽  
Periodontal Regeneration ◽  
Guided Tissue Regeneration ◽  
Periodontal Tissues ◽  
Extensive Documentation

The goals of periodontal therapy are to arrest of periodontal disease progression and to attain the regeneration of the periodontal apparatus. Osseous grafting and Guided tissue regeneration (GTR) are the two techniques with the most extensive documentation of periodontal regeneration. However, these techniques offer limited potential towards regenerating the periodontal tissues. Recent surgical procedures and application of newer materials aim at greater and more predictable regeneration with the concept of tissue engineering for enhanced periodontal regeneration and functional attachment have been developed, analyzed, and employed in clinical practice

Association of ALDH2 Genotypes with Periodontitis Progression

2009 ◽  
Vol 89 (2) ◽  
pp. 138-142 ◽  
Author(s):  
N. Nishida ◽  
M. Tanaka ◽  
S. Sekine ◽  
T. Takeshita ◽  
K. Nakayama ◽  
...  
Keyword(s):  
Alcohol Consumption ◽  
Periodontal Disease ◽  
Disease Progression ◽  
Odds Ratio ◽  
Alcohol Sensitivity ◽  
Pocket Depth ◽  
Probing Pocket Depth ◽  
Significant Odds Ratio ◽  
Progression Risk ◽  
Prospective Association

The progression of periodontitis may be affected by ALDH2 genotypes with respect to the oxidation of acetaldehyde to acetate, which leads to the accumulation of acetaldehyde in plasma and potential toxic effects. We examined the prospective association of ALDH2 genotypes in terms of alcohol sensitivity between alcohol consumption and periodontal disease progression. In 2003, 224 of 256 (87.5%) individuals examined at baseline (1999) completed probing pocket depth measurements for the evaluation of periodontitis progression. Missing data on self-reported questionnaires and blood samples were excluded; therefore, 183 samples were analyzed. Individuals who consumed ≥ 33.0 g/day of alcohol exhibited high periodontal disease progression risk (OR = 3.54). ALDH2 *1/*2 individuals who consumed ≥ 33 g/day of alcohol displayed a significant odds ratio (OR = 4.28) of periodontitis progression risk, in contrast to ALDH2 *1/*1 individuals. These results suggested that alcohol consumption as well as alcohol sensitivity may be a risk factor for periodontitis progression.

scholarly journals Metabolic Syndrome and Periodontal Disease Progression in Men

2016 ◽  
Vol 95 (7) ◽  
pp. 822-828 ◽  
Author(s):  
E.K. Kaye ◽  
N. Chen ◽  
H.J. Cabral ◽  
P. Vokonas ◽  
R.I. Garcia
Keyword(s):  
Metabolic Syndrome ◽  
Periodontal Disease ◽  
Disease Progression

scholarly journals Senescent cells exacerbate chronic inflammation and contribute to periodontal disease progression in old mice

2021 ◽  
Author(s):  
Ruben Aquino‐Martinez ◽  
Brittany A. Eckhardt ◽  
Jennifer L. Rowsey ◽  
Daniel G. Fraser ◽  
Sundeep Khosla ◽  
...  
Keyword(s):  
Periodontal Disease ◽  
Disease Progression ◽  
Chronic Inflammation ◽  
Old Mice

A 10-year retrospective study of periodontal disease progression

1989 ◽  
Vol 16 (7) ◽  
pp. 403-411 ◽  
Author(s):  
Panos N. Papapanou ◽  
Jan L. Wennstrom ◽  
Kerstin Grondahl
Keyword(s):  
Retrospective Study ◽  
Periodontal Disease ◽  
Disease Progression

scholarly journals CD26 Immuno-Expression and Periodontal Disease Progression

2001 ◽  
Vol 1 (2) ◽  
pp. 91-94 ◽  
Author(s):  
P. M. L. Manna ◽  
J. E. Costa ◽  
R. S. Gomez
Keyword(s):  
Periodontal Disease ◽  
Disease Progression ◽  
Cytokine Production ◽  
Membrane Glycoprotein ◽  
Th1 Cell ◽  
Clinical Attachment Loss ◽  
Attachment Loss ◽  
Immunological Mechanisms ◽  
Periodontal Attachment Loss ◽  
Immunohistochemical Identification

Immunological mechanisms participate in the pathogenesis of human chronic inflammatory periodontal disease (CIPD). Human CD4+lymphocytes express functionally heterogeneous profiles of cytokine production. CD26 is an integral membrane glycoprotein, that is, a marker of Th1-like cytokine development. The purpose of the present study was to compare the immuno-expression of CD26 receptor in periodontal sites with and without clinical attachment loss (CAL). Five patients with rapidly progressing periodontitis and one with juvenile periodontitis were investigated. Each patient presented at least one site with and without CAL. Ten sites with CAL and nine without any CAL were biopsied, followed by the immunohistochemical identification of the CD26 receptor using the MIB-DS2/7 antibody. The results demonstrated that the percentage of positive cells for this antigen in the periodontal sites with CAL was not significantly different from those without attachment loss. Therefore, Th1 cell impairment may not be directly involved with periodontal attachment loss.

Periodontal disease progression in subjects with orofacial clefts over a 25-year follow-up period

2009 ◽  
Vol 36 (10) ◽  
pp. 836-842 ◽  
Author(s):  
Guy Huynh-Ba ◽  
Urs Brägger ◽  
Marcel Zwahlen ◽  
Niklaus P. Lang ◽  
Giovanni E. Salvi
Keyword(s):  
Periodontal Disease ◽  
Disease Progression ◽  
Orofacial Clefts

Utility of Ramfjord index teeth to assess periodontal disease progression in longitudinal studies

1993 ◽  
Vol 20 (2) ◽  
pp. 147-150 ◽  
Author(s):  
Thomas E. Rams ◽  
Jacqueline Oler ◽  
Max A. Listgarten ◽  
Jorgen Slots
Keyword(s):  
Periodontal Disease ◽  
Disease Progression ◽  
Longitudinal Studies
Sign in / Sign up

Export Citation Format

Share Document