scholarly journals Cognitive Deficits in Patients with Gambling Disorder

2020 ◽  
Vol 28 (4) ◽  
pp. 167-185
Author(s):  
T.V. Agibalova ◽  
O.D. Tuchina ◽  
O.Zh. Buzik ◽  
R.K. Potapova ◽  
V.V. Potapov

The paper provides a narrative review of cognitive deficits in patients with gambling disorder. We discuss several types of these deficits, namely, cognitive impairments due to deficits in executive functioning, impulsive and risky decision-making, gambling-related cognitive distortions, and cognitive errors in cognitive-behavioral terms. These deficits are reviewed from the perspective of their relevance for psychotherapy and the development of differentiated psychotherapeutic interventions for gambling disorder treatment.

2018 ◽  
Vol 18 (2) ◽  
pp. 269-285 ◽  
Author(s):  
Iris M. Balodis ◽  
Jakob Linnet ◽  
Fiza Arshad ◽  
Patrick D. Worhunsky ◽  
Michael C. Stevens ◽  
...  

2020 ◽  
Vol 18 (5) ◽  
pp. 21-24
Author(s):  
E. G. MENDELEVICH ◽  
◽  
R. G. KOKURKINA ◽  

This paper presents literature review on cognitive impairments in patients with CM1, data on which began to be formed in the recent years. Data on the predominant deficit in the structure of executive functioning in patients with CM1 are presented. The genesis of cognitive deficits in patients with CM1 is most often considered in the context of cerebellar dysfunction and damage to the connections of the cerebellum with the prefrontal cortex, which leads to dysmetria of thinking and emotions. Data from other studies are presented that consider the role of additional factors of cognitive dysfunction in CM1, such as pain and emotional disorders.


2017 ◽  
Vol 34 (1) ◽  
pp. 209-223 ◽  
Author(s):  
Núria Mallorquí-Bagué ◽  
Iris Tolosa-Sola ◽  
Fernándo Fernández-Aranda ◽  
Roser Granero ◽  
Ana Beatriz Fagundo ◽  
...  

2021 ◽  
pp. 97-113
Author(s):  
Iris M. Balodis ◽  
Jakob Linnet ◽  
Fiza Arshad ◽  
Patrick D. Worhunsky ◽  
Michael C. Stevens ◽  
...  

2010 ◽  
Vol 19 (3) ◽  
pp. 70-78 ◽  
Author(s):  
Mary Purdy ◽  
Aimee Dietz

Abstract Instructing individuals with aphasia in the usage of AAC strategies and devices is a challenging endeavor. Not only does this population present with a wide range of linguistic impairments, but many individuals also demonstrate cognitive deficits, which may adversely affect communication. This paper will summarize the wide variety of cognitive deficits demonstrated by individuals with aphasia, specifically attention, memory and executive functioning problems. In addition, we will review the impact of these cognitive impairments on communicative competence. Finally, we will discuss an intervention, the Multimodality Communication Training Program (MCTP), designed to address the cognitive impairments that influence AAC intervention.


2009 ◽  
Vol 36 (11) ◽  
pp. 1213-1227 ◽  
Author(s):  
Eva M. Syngelaki ◽  
Simon C. Moore ◽  
Justin C. Savage ◽  
Graeme Fairchild ◽  
Stephanie H.M. Van Goozen

Executive functioning (EF) deficits have been implicated in antisocial behavior. EF constitutes a broad selection of functions, and it is yet to be determined what specific aspects of EF are associated with antisocial behavior. This study examines IQ, EF, and related decision-making processes in young males involved with the criminal justice system and their age-matched male controls. The Risky Choice Task (RCT), the Wisconsin Card Sorting Test (WCST), and tests taken from the Cambridge Neuropsychological Test Automated Battery (CANTAB) assessing spatial working memory, planning, and set-shifting ability were administered. Young offenders showed lower estimated IQ scores and perseveration of responding, as indicated by performance on the WCST. There were also specific EF impairments, such as problems in working memory and planning (CANTAB). Finally, young offenders showed more risky decision-making than controls did, particularly after a small win. The results on the RCT suggest altered reward mechanisms in young offenders, whereas the findings also support the notion of EF difficulties related to dorsolateral and ventromedial prefrontal cortex functioning. The implications of these findings for interventions with young offenders are discussed.


2009 ◽  
Vol 27 (21) ◽  
pp. 3526-3532 ◽  
Author(s):  
Femke K. Aarsen ◽  
Philippe F. Paquier ◽  
Willem-Frans Arts ◽  
Marie-Lise Van Veelen ◽  
Erna Michiels ◽  
...  

PurposeTo prospectively study cognitive deficits and predictors 3 years after diagnosis in a large series of pediatric patients treated for pilocytic astrocytoma (PA).Patients and MethodsSixty-one of 67 children were grouped according to infratentorial, supratentorial midline, and supratentorial hemispheric site. Intelligence, memory, attention, language, visual-spatial, and executive functions were assessed. Included predictors were sex, age, relapse, diagnosis-assessment interval, hydrocephalus, kind of treatment, and tumor variables.ResultsAll children with PA had problems with sustained attention and speed. In the infratentorial group, there also were deficits in verbal intelligence, visual-spatial memory, executive functioning, and naming. Verbal intelligence and verbal memory problems occurred in the brainstem tumor group. The supratentorial hemispheric tumor group had additional problems with selective attention and executive functioning, and the supratentorial midline tumor group displayed no extra impairments. More specifically, the dorsal supratentorial midline tumor group displayed problems with language and verbal memory. Predictors for lower cognitive functioning were hydrocephalus, radiotherapy, residual tumor size, and age; predictors for better functioning were chemotherapy or treatment of hydrocephalus. Almost 60% of children had problems with academic achievement, for which risk factors were relapse and younger age at diagnosis.ConclusionDespite normal intelligence at long-term follow-up, children treated for PA display invalidating cognitive impairments. Adequate treatment of hydrocephalus is important for a more favorable long-term cognitive outcome. Even children without initial severe deficits may develop cognitive impairments years after diagnosis, partly because of the phenomenon of growing into deficit, which has devastating implications for academic achievement and quality of life (QOL).


eNeuro ◽  
2020 ◽  
Vol 7 (3) ◽  
pp. ENEURO.0461-19.2020 ◽  
Author(s):  
Jan Peters ◽  
Taylor Vega ◽  
Dawn Weinstein ◽  
Jennifer Mitchell ◽  
Andrew Kayser

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