A CLUSTER OF CASES OF THYROID HYPERPLASIA IN AQUARIUM-HOUSED TROPICAL MARINE TELEOSTS FOLLOWING A CHANGE OF SALT MIX BRAND

2020 ◽  
Vol 51 (3) ◽  
Author(s):  
Marion Jalenques ◽  
Claire Vergneau-Grosset ◽  
Noémie Summa ◽  
Wahiba Ait Youcef ◽  
Jean-Francois St-Cyr ◽  
...  
Keyword(s):  
Thyroid Hyperplasia

THYROID VASCULARITY - DOCUMENTATION OF THE IODIDE EFFECT IN THYROTOXICOSIS

1977 ◽  
Vol 86 (2) ◽  
pp. 317-322 ◽  
Author(s):  
B. E. W. Brownlie ◽  
J. G. Turner ◽  
M. A. Ellwood ◽  
T. G. H. Rogers ◽  
D. I. Armstrong
Keyword(s):  
Potassium Iodide ◽  
Gamma Camera ◽  
Dramatic Reduction ◽  
Multinodular Goitre ◽  
Normal Limits ◽  
Thyroid Hyperplasia ◽  
Minicomputer System

ABSTRACT Thyroid vascularity was measured in 101 thyrotoxic patients by analysis of 99mTc pertechnetate thyroid flow studies obtained with a gamma camera - minicomputer system. The diffusely hyperplastic goitres tended to have higher vascularity than the toxic multinodular goitres, and many of the solitary toxic nodules had vascularity results within normal limits. Potassium iodide therapy, 60 mg b. d. for 10–14 days results in a dramatic reduction in thyroid vascularity in diffuse thyroid hyperplasia and toxic multinodular goitre but the effect on toxic nodules was marginal.

scholarly journals THE MORPHOLOGICAL CHANGES IN THE TISSUES OF THE RABBIT AS A RESULT OF REDUCED OXIDATION

1918 ◽  
Vol 27 (3) ◽  
pp. 399-412 ◽  
Author(s):  
H. G. Martin ◽  
A. S. Loevenhart ◽  
C. H. Bunting
Keyword(s):  
Carbon Dioxide ◽  
Oxygen Content ◽  
Morphological Changes ◽  
Low Oxygen ◽  
High Carbon ◽  
Red Bone Marrow ◽  
Hydropic Degeneration ◽  
Thyroid Hyperplasia ◽  
High Carbon Dioxide ◽  
Stimulation Of

Exposure of rabbits to an atmosphere of low oxygen content results in a stimulation of the cardiorespiratory systems, in an extension (hyperplasia) of red bone marrow and probably of a thyroid hyperplasia, with the further production of hydropic and hyaline degeneration in the cells of the parenchymatous organs. An atmosphere of high carbon dioxide and normal oxygen content produces, however, a stimulation of the cardiorespiratory systems, but no marrow extension and, in the concentrations used, but slight hydropic degeneration in the parenchyma of the glandular organs.

CLINICAL CHEMISTRY OF GRAIN-FED CATTLE.: III. PLASMA THYROID PROFILES WITH NOTES ON INTERFERENCE BY EDDI AND HYPOTHYROID VALUES IN HEREFORDS

1976 ◽  
Vol 56 (3) ◽  
pp. 457-465 ◽  
Author(s):  
R. W. BIDE
Keyword(s):  
Free Choice ◽  
Clinical Chemistry ◽  
Control Diet ◽  
Test Group ◽  
Control Group ◽  
Feeding Period ◽  
Total Plasma ◽  
Alfalfa Hay ◽  
Beet Pulp ◽  
Thyroid Hyperplasia

Linear thyroid profiles consisting of total plasma I (TI), protein-bound iodine (PBI), thyroxine iodine (T4I), tri-iodothyronine uptake (T3U) and the corresponding indices of T7I = T4I* T3U/100 and thyroid binding globulin estimate TBGE = T3U−1 + T4r−0.25 were prepared for 15 grain-fed (test) and 15 hay-fed (control) Hereford steers over a 186-day feeding period. The grain diet, which contained 20 mg/kg of I2 as ethylenediaminedihydriodide (EDDI), consisted of 90% steam-rolled barley, 5% beet pulp pellets and 5% protein–mineral concentrate, supplemented with local alfalfa hay fed at the rate of 0.9 kg/head/day. The control diet was local alfalfa hay given ad libitum. CoI salt blocks containing 0.028% Ca (IO3)2 were available to both groups, free choice. In the test group, TI increased threefold immediately upon introduction of the grain diet, and remained elevated. Plasma PBI increased from 5.0 to 12 μg/dl on day 50 and then fell slowly to 9.0 μg/dl by day 180. T4I, T7I and TBGE decreased during the first 30 days and returned to the original values of 4.6 ± 1.2, 2.3 ± 0.8 and 1.48 ± 0.11 μgI/dl. T3U decreased from 51 to 48% over the feeding period. The PBI and T4I values were not related, probably because EDDI or a metabolite of it interferes with the PBI test. In the control group, the profile values at the beginning and end of the feeding period were in μgI/dl: TI, 7.3 ± 1.5 and 5.6 ± 1.0; PBI, 5.9 ± 1.0 and 5.3 ± 0.7; T4I, 4.2 ± 0.4 and 2.8 ± 0.5; T3U%, 55.8 ± 8.0 and 54.4 ± 6.9. These values provided corresponding index values of T7I, 2.3 ± 0.4 and 1.5 ± 0.4 and TBGE, 1.44 ± 0.04 and 1.31 ± 0.06. The control diet did not provide enough available I2; the thyroid profiles showed a trend towards hypothyroidism, and histologic examination post-mortem revealed thyroid hyperplasia.

Expression of fibroblast growth factor and its receptor in thyroid hyperplasia and neoplasia

1997 ◽  
Vol 9 (2) ◽  
pp. 128
Author(s):  
S.D. Thompson ◽  
J.C. Watkinson ◽  
J. Verhaeg ◽  
M.C. Sheppard ◽  
J.A. Franklyn ◽  
...  
Keyword(s):  
Growth Factor ◽  
Fibroblast Growth Factor ◽  
Fibroblast Growth ◽  
Thyroid Hyperplasia

Hypertrophy and hyperplasia during goitre growth and involution in rats separate bioeffects of TSH and iodine

1987 ◽  
Vol 116 (4) ◽  
pp. 537-548 ◽  
Author(s):  
D. Stübner ◽  
R. Gärtner ◽  
W. Greil ◽  
K. Gropper ◽  
G. Brabant ◽  
...  
Keyword(s):  
Serum Level ◽  
Iodine Deficiency ◽  
Tap Water ◽  
Iodine Content ◽  
Total Dna ◽  
Thyroid Hyperplasia ◽  
Tsh Levels

Abstract. Goitre growth was investigated in rats receiving a low iodine diet (< 0.1 μg iodine/g) and either 1 g/l KClO4 or 1 g/l propylthiouracil (PTU), or a combination of KClO4 or PTU with 50.82 nmol/l T3 in tap water for 3 weeks. To investigate goitre involution, rats with iodine-deficient goitres were treated for 3 weeks either with T3 (0.5 μg T3/day = 0.768 nmol/day), iodide (0.5 or 2.7 μg KI/day) or a combination of T3 with both iodide doses. Histology together with total DNA distinguished between hypertrophy and hyperplasia of the gland. During goitre growth there was a highly significant correlation between goitre weight and TSH serum level (r = 0.93, P < 0.001). Thyroid total DNA, however, was only weakly correlated to TSH but was inversely related to the degree of iodine deficiency. During goitre regression, TSH levels were normalized, histological signs of hypertrophy had disappeared, and thyroid weight was nearly normalized in all therapy groups. Total DNA, however, was normalized only with 2.7 μg KI/day (95 ± 18 μg DNA/gland), and still elevated in all other groups. The highest DNA levels were found under T3 therapy (143 ± 21 μg DNA/gland) and under 0.5 μg KI/day (161 ± 19 μg DNA/gland). Reduction of total DNA was independent of TSH, but followed replenishment of the iodine content of the glands. We conclude that TSH mainly induces hypertrophy, whereas thyroid hyperplasia is mainly regulated by the intracellular iodine content.

RHESUS SENSITISATION, AMNIOGRAPHY, AND THYROID HYPERPLASIA

The Lancet ◽  
1976 ◽  
Vol 308 (7996) ◽  
pp. 1191-1192
Author(s):  
D BECROFT
Keyword(s):  
Thyroid Hyperplasia
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