scholarly journals Aluminium tolerance in barley (Hordeum vulgare L.): Physiological mechanisms, genetics and screening methods

2006 ◽  
Vol 7 (10) ◽  
pp. 769-787 ◽  
Author(s):  
Jun-ping Wang ◽  
Harsh Raman ◽  
Guo-ping Zhang ◽  
Neville Mendham ◽  
Mei-xue Zhou
2013 ◽  
Vol 32 (1) ◽  
pp. 155-164 ◽  
Author(s):  
Miao Bian ◽  
Irene Waters ◽  
Sue Broughton ◽  
Xiao-Qi Zhang ◽  
Meixue Zhou ◽  
...  

BMC Genomics ◽  
2016 ◽  
Vol 17 (1) ◽  
Author(s):  
Yanling Ma ◽  
Chengdao Li ◽  
Peter R. Ryan ◽  
Sergey Shabala ◽  
Jianfeng You ◽  
...  

2020 ◽  
Vol 4 (7) ◽  
pp. 92-101
Author(s):  
Zied Hammami ◽  
Nawel Ahmed ◽  
Nejia Ben Hmida ◽  
Soumaya Tounsi ◽  
Sawsen Ayadi ◽  
...  

2006 ◽  
Vol 57 (1) ◽  
pp. 113 ◽  
Author(s):  
Junping Wang ◽  
Harsh Raman ◽  
Barbara Read ◽  
Meixue Zhou ◽  
Neville Mendham ◽  
...  

Aluminium (Al) phytotoxicity is one of the major constraints for barley (Hordeum vulgare L.) production on acid soils. Genetic variation for Al tolerance is present in barley germplasm. Previous studies have shown that Al tolerance in cv. Dayton and WB229 is controlled by a single locus on 4H. The present study was conducted to investigate the genetic control of Al tolerance in Japanese barley cv. Honen, and validate the linkage relationship between the Al tolerance locus and the molecular markers established previously. An F3 progeny representing 94 F2s from the cross of F6ant28B48-16 (Al sensitive) × Honen (Al tolerant) was evaluated for Al tolerance by staining roots with eriochrome cyanine R. A single Al tolerance locus Alt was validated on the long arm of chromosome 4H in Honen. Single sequence repeat (SSR) markers Bmag353 and HVM68 flanked the Alt locus at a distance of 1.6 ± 0.9 and 5.1 ± 1.7 cM, respectively. Our results have shown that the linkage between SSR markers and the Al tolerance locus can be used to characterise the ‘unknown source’ for Al tolerance.


Author(s):  
R.H.M. Cross ◽  
C.E.J. Botha ◽  
A.K. Cowan ◽  
B.J. Hartley

Senescence is an ordered degenerative process leading to death of individual cells, organs and organisms. The detection of a conditional lethal mutant (achloroplastic) of Hordeum vulgare has enabled us to investigate ultrastructural changes occurring in leaf tissue during foliar senescence.Examination of the tonoplast structure in six and 14 day-old mutant tissue revealed a progressive degeneration and disappearance of the membrane, apparently starting by day six in the vicinity of the mitochondria associated with the degenerating proplastid (Fig. 1.) where neither of the plastid membrane leaflets is evident (arrows, Fig. 1.). At this stage there was evidence that the mitochondrial membranes were undergoing retrogressive changes, coupled with disorganization of cristae (Fig. 2.). Proplastids (P) lack definitive prolamellar bodies. The cytoplasmic matrix is largely agranular, with few endoplasmic reticulum (ER) cisternae or polyribosomal aggregates. Interestingly, large numbers of actively-budding dictysomes, associated with pinocytotic vesicles, were observed in close proximity to the plasmalemma of mesophyll cells (Fig. 3.). By day 14 however, mesophyll cells showed almost complete breakdown of subcellular organelle structure (Fig. 4.), and further evidence for the breakdown of the tonoplast. The final stage of senescence is characterized by the solubilization of the cell wall due to expression and activity of polygalacturonase and/or cellulose. The presence of dictyosomes with associated pinocytotic vesicles formed from the mature face, in close proximity to both the plasmalemma and the cell wall, would appear to support the model proposed by Christopherson for the secretion of cellulase. This pathway of synthesis is typical for secretory glycoproteins.


Author(s):  
А.В. ЖЕЛЕЗНОВ ◽  
◽  
Н.Б. ЖЕЛЕЗНОВА ◽  
Т.В. КУКОЕВА ◽  
Н.В. БУРМАКИНА ◽  
...  

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