Flame wall interaction of an H2/02 flame

2002 ◽  
Author(s):  
Francois Dabireau ◽  
Olivier Vermorel ◽  
Benedicte Cuenot ◽  
Thierry Poinsot
Keyword(s):  
Wall Interaction

Evidence for an Increased Generation of Prostacyclin in the Microvasculature and an Impairment of the Platelet α-Granule Release in Chronic Renal Failure

1988 ◽  
Vol 60 (02) ◽  
pp. 205-208 ◽  
Author(s):  
Paul A Kyrle ◽  
Felix Stockenhuber ◽  
Brigitte Brenner ◽  
Heinz Gössinger ◽  
Christian Korninger ◽  
...  
Keyword(s):  
Renal Failure ◽  
Chronic Renal Failure ◽  
Blood Clotting ◽  
Normal Subjects ◽  
Chronic Uraemia ◽  
Wall Interaction ◽  
End Stage ◽  
Granule Release

SummaryThe formation of prostacyclin (PGI2) and thromboxane A2 and the release of beta-thromboglobulin (beta-TG) at the site of platelet-vessel wall interaction, i.e. in blood emerging from a standardized injury of the micro vasculature made to determine bleeding time, was studied in patients with end-stage chronic renal failure undergoing regular haemodialysis and in normal subjects. In the uraemic patients, levels of 6-keto-prostaglandin F1α (6-keto-PGF1α) were 1.3-fold to 6.3-fold higher than the corresponding values in the control subjects indicating an increased PGI2 formation in chronic uraemia. Formation of thromboxane B2 (TxB2) at the site of plug formation in vivo and during whole blood clotting in vitro was similar in the uraemic subjects and in the normals excluding a major defect in platelet prostaglandin metabolism in chronic renal failure. Significantly smaller amounts of beta-TG were found in blood obtained from the site of vascular injury as well as after in vitro blood clotting in patients with chronic renal failure indicating an impairment of the a-granule release in chronic uraemia. We therefore conclude that the haemorrhagic diathesis commonly seen in patients with chronic renal failure is - at least partially - due to an acquired defect of the platelet a-granule release and an increased generation of PGI2 in the micro vasculature.

Further Evidence that Glycoprotein IIb-IIIa Mediates Platelet Spreading on Subendothelium

1991 ◽  
Vol 65 (02) ◽  
pp. 202-205 ◽  
Author(s):  
Harvey J Weiss ◽  
Vincet T Turitto ◽  
Hans R Baumgartner
Keyword(s):  
Platelet Adhesion ◽  
Surface Coverage ◽  
Rabbit Aorta ◽  
Conclusive Evidence ◽  
Initial Contact ◽  
Single Exposure ◽  
Vessel Segment ◽  
Multiple Exposures ◽  
Wall Interaction ◽  
Platelet Spreading

SummaryIn order to explore further the mechanism by which glycoprotein GPIIb-IIIa promotes platelet vessel wall interaction, platelet adhesion to subendothelium was studied in an annular chamber in which subendothelium from rabbit aorta was exposed at a shear rate of 2,600 s−1 to blood from patients with thrombasthenia. Perfusions were conducted for each of 5 exposure times (1 ,2,3, 5 and 10 min), and the percent surface coverage of the vessel segment with platelets in the contact (C) and spread (S) stage was determined. Increased values of platelet contact (C) were obtained in thrombasthenia at all exposure times; this finding is consistent with a defect in platelet spreadirg, based on a previously described kinetic model of platelet attachment to subendothelium. According to this model of attachment, increased values of platelet contact (C) at a single exposure time may be indicative of either a defect in spreading (S) or initial contact (C), but multiple exposures will result in increased contact only for defects which are related to defectiye platelet spreading (s).The results obtained over a broad range of exposure times provide more conclusive evidence that GPIIb-IIIa mediates platelet spreading than those previously obtained at single exposure times.

Thrombin Inhibition and Thrombin Generation by Cultured Aortic Endothelial and Smooth Muscle Cells from Minipig

1982 ◽  
Vol 48 (01) ◽  
pp. 101-103 ◽  
Author(s):  
B Kirchhof ◽  
J Grünwald
Keyword(s):  
Endothelial Cells ◽  
Smooth Muscle ◽  
Smooth Muscle Cells ◽  
Vessel Wall ◽  
Thrombin Generation ◽  
Muscle Cells ◽  
Thrombin Inhibition ◽  
Generating Capacity ◽  
Wall Interaction ◽  
Cells Cultured

SummaryEndothelial and smooth muscle cells cultured from minipig aorta were examined for their inhibitory activity on thrombin and for their thrombin generating capacity.Endothelial cells showed both a thrombin inhibition and an activation of prothrombin in the presence of Ca++, which was enhanced in the presence of phospholipids. Smooth muscle cells showed an activation of prothrombin but at a lower rate. Both coagulation and amidolytic micro-assays were suitable for studying the thrombin-vessel wall interaction.

DEVELOPMENT OF DROP/WALL INTERACTION MODEL FOR APPLICATION IN ENGINE CONDITIONS

2020 ◽  
Vol 30 (3) ◽  
pp. 153-170
Author(s):  
Yaoyu Pan ◽  
Xiufeng Yang ◽  
Song-Charng Kong ◽  
Chol-Bum M. Kweon
Keyword(s):  
Interaction Model ◽  
Wall Interaction

Application of Laser Resonance Scattering to the Study of High-temperature Plasma-wall Interaction

1981 ◽  
Vol 9 (3) ◽  
pp. 237-252 ◽  
Author(s):  
Mitsuo MAEDA ◽  
Katsunori MURAOKA ◽  
Makoto HAMAMOTO ◽  
Masanori AKAZAKI ◽  
Yasushi MIYAZOE
Keyword(s):  
High Temperature ◽  
Resonance Scattering ◽  
Temperature Plasma ◽  
Laser Resonance ◽  
High Temperature Plasma ◽  
Wall Interaction

scholarly journals MHD natural convection flow of Casson fluid in an annular microchannel containing porous medium with heat generation/absorption

2020 ◽  
Vol 9 (1) ◽  
pp. 223-232 ◽  
Author(s):  
B.J. Gireesha ◽  
S. Sindhu
Keyword(s):  
Porous Medium ◽  
Natural Convection ◽  
Skin Friction ◽  
Interaction Parameter ◽  
Heat Generation ◽  
Velocity Slip ◽  
Casson Fluid ◽  
Convection Flow ◽  
Natural Convection Flow ◽  
Wall Interaction

AbstractThis study has been conducted to focus on natural convection flow of Casson fluid through an annular microchannel formed by two cylinders in the presence of magnetic field. The process of heat generation/absorption is taken into consideration. Combined effects of various parameters such as porous medium, velocity slip and temperature jump are considered. Solution of the present mathematical model is obtained numerically using fourth-fifth order Runge-Kutta-Fehlberg method. The flow velocity, thermal field, skin friction and Nusselt number are scrutinized with respect to the involved parameters of interest such as fluid wall interaction parameter, rarefaction parameter, Casson parameter and Darcy number with the aid of graphs. It is established that higher values of Casson parameter increases the skin friction coefficient. Further it is obtained that rate of heat transfer diminishes as fluid wall interaction parameter increases.

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