scholarly journals Role of Follicle-Stimulating Hormone and Androgens on the Sexual Inversion of Sevenband GrouperEpinephelus septemfasciatus

2008 ◽  
Vol 70 (2) ◽  
pp. 266-272 ◽  
Author(s):  
Richard J. Kline ◽  
Izhar A. Khan ◽  
Kiyoshi Soyano ◽  
Megumi Takushima
Keyword(s):  
Follicle Stimulating Hormone ◽  
Stimulating Hormone

Follicle-stimulating hormone-dependent estrogen secretion by rat Sertoli cells in vitro: Modulation by calcium

1991 ◽  
Vol 125 (3) ◽  
pp. 280-285 ◽  
Author(s):  
J. Alan Talbot ◽  
Ann Lambert ◽  
Robert Mitchell ◽  
Marek Grabinski ◽  
David C. Anderson ◽  
...  
Keyword(s):  
Sertoli Cells ◽  
Culture Medium ◽  
Follicle Stimulating Hormone ◽  
Dibutyryl Camp ◽  
Immature Rat ◽  
Estradiol Secretion ◽  
Old Rats ◽  
Stimulating Hormone

Abstract We have investigated the role of Ca2+ in the control of FSH-induced estradiol secretion by Sertoli cells isolated from 8-10 days old rats. Exogenous Ca2+ (4-8 mmol/1) inhibited FSH-stimulated E2 secretion such that, with 8 mmol/l Ca2+ and FSH (8 IU/l) E2 secretion decreased from 2091±322 to 1480±84 pmol/l (p<0.002), whilst chelation of Ca2+ in the culture medium with EGTA (3 mmol/l) increased E2 secretion from 360±45 to 1242±133 pmol/l) in the absence of FSH. Further, EGTA (3 mmol/l) markedly potentiated FSH (8 IU/l), forskolin (1 μmol/l) and dibutyryl cAMP (1 mmol/l)-stimulated E2 secretion. Addition of the Ca2+ ionophores, ionomycin (2-5 μmol/l) and A23187 (2 μmol/l), inhibited FSH (8 IU/l)-stimulated E2 secretion by >80%. The effect of ionomycin was totally reversible, whereas that of A23187 was irreversible. Ionomycin (5 μmol/l) had no effect on EGTA-induced E2 secretion in the absence of FSH, but reduced EGTA-provoked E2 secretion by 59% in the presence of FSH (8 IU/l). Similarly, forskolin- and dibutyryl cAMP-provoked E2 production was inhibited 46-50% by ionomycin (5 μmol/l). We conclude that FSH-induced E2 secretion from immature rat Sertoli cells is modulated by intra- and extracellular Ca2+.

scholarly journals Role of Kisspeptin in Puberty in Humans

2020 ◽  
pp. 92-97
Author(s):  
Ravi Kant ◽  
Mahendra Kumar Meena
Keyword(s):  
Reproductive Biology ◽  
Conventional Treatment ◽  
Hypogonadotropic Hypogonadism ◽  
Follicle Stimulating Hormone ◽  
Gonadotropin Releasing Hormone ◽  
Pilot Studies ◽  
Releasing Hormone ◽  
Infertile Couples ◽  
Stimulating Hormone

Kisspeptin or GPR-54 is a product of KISS 1 gene regulating the production of gonadotropin releasing hormone (GnRH), luteinizing (LH) as well follicle stimulating hormone (FSH). Both LH and FSH are important hormones for reproduction in animals as well in humans. The recognition of Kisspeptin has a landmark bearing in reproductive biology. Few recent pilot studies have convincingly proven it to be a promising molecule in treating infertile couples especially those having hypogonadotropic hypogonadism not responding to conventional treatment.

scholarly journals Role of E Box and Initiator Region in the Expression of the Rat Follicle-stimulating Hormone Receptor

1996 ◽  
Vol 271 (52) ◽  
pp. 33317-33324 ◽  
Author(s):  
Tamara L. Goetz ◽  
Tracy L. Lloyd ◽  
Michael D. Griswold
Keyword(s):  
Hormone Receptor ◽  
Follicle Stimulating Hormone ◽  
E Box ◽  
Stimulating Hormone

Role of Glycosylation in Function of Follicle-Stimulating Hormone

Endocrine ◽  
1999 ◽  
Vol 11 (3) ◽  
pp. 205-216 ◽  
Author(s):  
Alfredo Ulloa-Aguirre ◽  
Carlos Timossi ◽  
Pablo Damián-Matsumura ◽  
James A. Dias
Keyword(s):  
Follicle Stimulating Hormone ◽  
Stimulating Hormone
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