scholarly journals Relationship Between the Pulmonary Artery Index and Physiological Properties of the Pulmonary Vascular Bed

1996 ◽  
Vol 60 (6) ◽  
pp. 334-340 ◽  
Author(s):  
Hideaki Senzaki ◽  
Hitoshi Kato ◽  
Michio Akagi ◽  
Toshio Hishi ◽  
Masayoshi Yanagisawa
Keyword(s):  
Pulmonary Artery ◽  
Vascular Bed ◽  
Physiological Properties ◽  
Pulmonary Vascular Bed

Effect of Ligation of Left Pulmonary Artery at Birth on Maturation of Pulmonary Vascular Bed

Respiration ◽  
1962 ◽  
Vol 19 (5) ◽  
pp. 362-369
Author(s):  
Peter E. Pool ◽  
Keith H. Averill ◽  
John H.K. Vogel
Keyword(s):  
Pulmonary Artery ◽  
Left Pulmonary Artery ◽  
Vascular Bed ◽  
Pulmonary Vascular Bed

The pulmonary vascular bed in patients with functionally univentricular physiology and a Fontan circulation

2021 ◽  
pp. 1-10
Author(s):  
Ahmed Krimly ◽  
C. Charles Jain ◽  
Alexander Egbe ◽  
Ahmed Alzahrani ◽  
Khalid Al Najashi ◽  
...  
Keyword(s):  
Pulmonary Artery ◽  
Venous Hypertension ◽  
Adult Survival ◽  
Vascular Reserve ◽  
Passive Flow ◽  
Vascular Bed ◽  
Systemic Ventricle ◽  
Pharmacologic Regulation ◽  
Fontan Palliation ◽  
Pulmonary Vascular Bed

Abstract Fontan palliation represents one of the most remarkable surgical advances in the management of individuals born with functionally univentricular physiology. The operation secures adult survival for all but a few with unfavourable anatomy and/or physiology. Inherent to the physiology is passive transpulmonary blood flow, which produces a vulnerability to adequate filling of the systemic ventricle at rest and during exertion. Similarly, the upstream effects of passive flow in the lungs are venous congestion and venous hypertension, especially marked during physical activity. The pulmonary vascular bed has emerged as a defining character on the stage of Fontan circulatory behaviour and clinical outcomes. Its pharmacologic regulation and anatomic rehabilitation therefore seem important strategic therapeutic targets. This review seeks to delineate the important aspects of pulmonary artery development and maturation in functionally univentricular physiology patients, pulmonary artery biology, pulmonary vascular reserve with exercise, and pulmonary artery morphologic and pharmacologic rehabilitation.

Pressure-Flow Relationships in the Dog Lung During Acute, Subtotal Pulmonary Vascular Occlusion

1958 ◽  
Vol 192 (3) ◽  
pp. 613-619 ◽  
Author(s):  
Michael T. Lategola
Keyword(s):  
Blood Flow ◽  
Pulmonary Artery ◽  
Pulmonary Artery Pressure ◽  
Pulmonary Blood Flow ◽  
Vascular Occlusion ◽  
Percentage Change ◽  
Artery Pressure ◽  
Vascular Bed ◽  
Change In Mean ◽  
Pulmonary Vascular Bed

The relationship of pulmonary artery pressure to pulmonary blood flow was studied in the dog by means of occlusive shifting of blood flow within the pulmonary vascular bed. All experiments were performed using the closed-chest preparation. The range of blood flow increases studied was 25–388%. A graphical plot of the percentage change in blood flow versus the percentage change in mean pulmonary artery pressure is presented. A visually estimated curve of this latter data is presented, discussed and compared to four other curves from previous pulmonary vascular studies. A comparison of these curves suggests that the relative maximum capacity of the pulmonary vascular bed of man and dog are similar. These curves plus certain assumptions allow the speculative delineation of a graphical area representing the ‘active’ vasomotor component of exercise at different levels of pulmonary blood flow increase.

scholarly journals OBSERVATIONS ON RESISTANCE TO THE FLOW OF BLOOD TO AND FROM THE LUNGS

1927 ◽  
Vol 45 (4) ◽  
pp. 655-671 ◽  
Author(s):  
Richmond L. Moore ◽  
Carl A. L. Binger
Keyword(s):  
Pulmonary Artery ◽  
Pulmonary Veins ◽  
Hydrogen Ion ◽  
Ion Concentration ◽  
Tissue Elasticity ◽  
Hydrogen Ion Concentration ◽  
Starch Grains ◽  
Vascular Bed ◽  
Shallow Breathing ◽  
Pulmonary Vascular Bed

1. Embolism of pulmonary arterioles and capillaries produced by the intravenous injection of starch grains results in a dilatation of the pulmonary artery and the right chambers of the heart. This has been demonstrated both by x-ray studies and direct inspection. 2. The dilatation of the pulmonary artery and heart occurs synchronously with the acceleration of respirations. 3. Dilatation of these structures produced by other means, such as obstruction to the flow of blood to and from the lungs, by gradually clamping either the pulmonary artery (cat and dog) or pulmonary veins (cat) does not, however, give rise to rapid and shallow breathing. 4. The effect of these maneuvers on respiration does not become apparent until respirations suddenly cease. 5. Neither does sudden restriction of the pulmonary vascular bed by clamping the left branch of the pulmonary artery give rise to rapid and shallow breathing, though this procedure may cause an increase in CO2 tension and in hydrogen ion concentration of the blood. 6. Since rapid and shallow breathing is not the result of (1) anoxemia, (2) increased pCO2 and hydrogen ion concentration of the serum, (3) restriction of pulmonary vascular bed by nearly half, (4) increase in resistance to the flow of blood to and from the lungs) (5) the presence of starch grains in the lungs acting as a local irritant, it must be the result of the secondary pathological changes which occur in the pulmonary parenchyma following embolism. 7. The nature of these changes, congestion and edema, has been discussed elsewhere. Whether they operate directly on nerve endings or through their influence on lung volume and tissue elasticity is not certain. 8. Various important clinical analogies have been emphasized.

Responses to L-163,491, a nonpeptide angiotensin II mimic, in the pulmonary vascular bed of the cat

1995 ◽  
Vol 287 (2) ◽  
pp. 163-168 ◽  
Author(s):  
Alan D. Kaye ◽  
Bobby D. Nossaman ◽  
Salah Kivlighn ◽  
Ikhlass N. Ibrahim ◽  
Bracken J. De Witt ◽  
...  
Keyword(s):  
Angiotensin Ii ◽  
Vascular Bed ◽  
Pulmonary Vascular Bed
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