scholarly journals Thermoregulatory consequences of salt loading in the lizard Pogona vitticeps

2015 ◽  
Vol 218 (8) ◽  
pp. 1166-1174 ◽  
Author(s):  
C. da Silveira Scarpellini ◽  
K. C. Bicego ◽  
G. J. Tattersall
2001 ◽  
Vol 708 ◽  
Author(s):  
Mathew K. Mathai ◽  
Keith A. Higginson ◽  
Bing R. Hsieh ◽  
Fotios Papadimitrakopoulos

ABSTRACTIn this paper we report a method for tuning the extent of hole injection into the active light emitting tris- (8-hydroxyquinoline) aluminum (Alq3) layer in organic light emitting diodes (OLEDs). This is made possible by modifying the indium tin oxide (ITO) anode with an oxidized transport layer (OTL) comprising a hole transporting polycarbonate of N,N'-bis(3-hydroxymethyl)-N,N'-bis(phenyl) benzidine and diethylene glycol (PC-TPB-DEG) doped with varying concentrations of antimonium hexafluoride salt of N,N,N',N'-tetra-p-tolyl-4,4'-biphenyldiamine (TMTPD+ SbF6-). The conductivity of the OTL can be changed over three orders of magnitude depending on salt loading. The analysis of hole and electron current variations in these devices indicates that optimizing the conductivity of the OTL enables the modulation of hole injection into the Alq3 layer. The bipolar charge transport properties for OLEDs in which the interfacial carrier injection barriers have been minimized, are governed by the conductivities of the respective layers and in this case it is shown that the variable conductivity of the OTL does allow for better control of the same. Accordingly, varying the concentration of holes in the device indicates that beyond an optimum concentration of holes, further hole injection results in the formation of light quenching cationic species and the initiation of oxidative degradation processes in the Alq3 layer, thus accelerating the intrinsic degradation of these devices. The variable conductivity of the OTL can hence be used to minimize the occurrence of these processes.


Nutrients ◽  
2021 ◽  
Vol 13 (5) ◽  
pp. 1502
Author(s):  
Katarzyna Łabno-Kirszniok ◽  
Agata Kujawa-Szewieczek ◽  
Andrzej Wiecek ◽  
Grzegorz Piecha

Increased marinobufagenin (MBG) synthesis has been suggested in response to high dietary salt intake. The aim of this study was to determine the effects of short-term changes in sodium intake on plasma MBG levels in patients with primary salt-sensitive and salt-insensitive hypertension. In total, 51 patients with primary hypertension were evaluated during acute sodium restriction and sodium loading. Plasma or serum concentrations of MBG, natriuretic pro-peptides, aldosterone, sodium, potassium, as well as hematocrit (Hct) value, plasma renin activity (PRA) and urinary sodium and potassium excretion were measured. Ambulatory blood pressure monitoring (ABPM) and echocardiography were performed at baseline. In salt-sensitive patients with primary hypertension plasma MBG correlated positively with diastolic blood pressure (ABPM) and serum NT-proANP concentration at baseline and with serum NT-proANP concentration after dietary sodium restriction. In this subgroup plasma MBG concentration decreased during sodium restriction, and a parallel increase of PRA was observed. Acute salt loading further decreased plasma MBG concentration in salt-sensitive subjects in contrast to salt insensitive patients. No correlation was found between plasma MBG concentration and left ventricular mass index. In conclusion, in salt-sensitive hypertensive patients plasma MBG concentration correlates with 24-h diastolic blood pressure and dietary sodium restriction reduces plasma MBG levels. Decreased MBG secretion in response to acute salt loading may play an important role in the pathogenesis of salt sensitivity.


1995 ◽  
Vol 81 (2) ◽  
pp. 281 ◽  
Author(s):  
Chris T. McAllister ◽  
Steve J. Upton ◽  
Elliott R. Jacobson ◽  
Wayne Kopit

2000 ◽  
Vol 18 (11) ◽  
pp. 1657-1664 ◽  
Author(s):  
Michel Burnier ◽  
Marie-Laure Monod ◽  
Arnaud Chioléro ◽  
Marc Maillard ◽  
Jurg Nussberger ◽  
...  

1970 ◽  
Vol 27 (6) ◽  
pp. 1123-1130 ◽  
Author(s):  
W. Dall

Lobsters took 72 hr to adapt to a salinity of 20‰, with urine becoming markedly hypoosmotic to the blood during the first 25 hr, then increasing in concentration over the next 48 hr, though remaining significantly hypoosmotic to the blood. Adaptation to a salinity of 37‰ took 24 hr: the urine became almost isosmotic with the blood: the gastric and rectal fluids became hyperosmotic to the blood. Blood freezing-point determinations showed that over the salinity range 20–37‰ the lobster is able to osmoregulate to a limited extent only towards the lower end of the range, being otherwise an "osmocon-former." Salt-loading experiments indicated that the excess salts were rapidly excreted into the gut. It is concluded that the antennal glands are at least partly responsible for elimination of excess water, but that the gut is the site of salt excretion, and that there is rapid adjustment of salt imbalance.


1997 ◽  
Vol 130 (1) ◽  
pp. 76-82 ◽  
Author(s):  
Volker Vallon ◽  
Dorothee Kirschenmann ◽  
Lucinda M. Wead ◽  
Mark J. Lortie ◽  
Joe Satriano ◽  
...  

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