scholarly journals Interpersonal Trauma Exposure and Cognitive Development in Children to Age 8 Years: A Longitudinal Study

2015 ◽  
pp. 135-154
2012 ◽  
Vol 66 (11) ◽  
pp. 1005-1010 ◽  
Author(s):  
Michelle Bosquet Enlow ◽  
Byron Egeland ◽  
Emily A Blood ◽  
Robert O Wright ◽  
Rosalind J Wright

2016 ◽  
Vol 29 (3) ◽  
pp. 197-204 ◽  
Author(s):  
Rohan H. C. Palmer ◽  
Nicole R. Nugent ◽  
Leslie A. Brick ◽  
Cinnamon L. Bidwell ◽  
John E. McGeary ◽  
...  

2018 ◽  
pp. 088626051881987 ◽  
Author(s):  
Jessica L. Schleider ◽  
Jacqueline Woerner ◽  
Cassie Overstreet ◽  
Ananda B. Amstadter ◽  
Carolyn E. Sartor

2017 ◽  
Vol 38 (1) ◽  
pp. 72-94 ◽  
Author(s):  
Miguel Pérez-Pereira ◽  
Raquel Cruz

The vocabulary size and composition of one group of full-term and three groups of low risk preterm children with different gestational ages (GA) were longitudinally compared at 10, 22 and 30 months of age. Expressive vocabulary development was assessed through the CDI. Cognitive development was also assessed at 22 months (Batelle Developmental Inventory), and data concerning biological and environmental characteristics of the children were also obtained. Growth curve analyses indicated that there were no significant differences in vocabulary size or percentage of word categories among GA groups. Regression analyses showed that word production and cognitive scores measured at 22 months were the main predictors of total vocabulary and word categories at 30 months. Gender, maternal education and GA did not contribute in a significant way to the variance of use of the vocabulary categories or vocabulary size. Therefore, GA does not seem to affect vocabulary development and composition when biomedical complications associated to prematurity are excluded.


2020 ◽  
Author(s):  
Emily E. Bray ◽  
Margaret E. Gruen ◽  
Gitanjali E. Gnanadesikan ◽  
Daniel J. Horschler ◽  
Kerinne M. Levy ◽  
...  

2019 ◽  
Vol 52 ◽  
pp. 100805 ◽  
Author(s):  
Smaragda Kazi ◽  
Elena Kazali ◽  
Nikolaos Makris ◽  
George Spanoudis ◽  
Andreas Demetriou

2018 ◽  
Vol 53 (1) ◽  
pp. 48-58 ◽  
Author(s):  
Denise A Chu ◽  
Richard A Bryant ◽  
Justine M Gatt ◽  
Anthony WF Harris

Objective: Posttraumatic stress disorder and childhood trauma frequently co-occur. Both are associated with abnormal neural responses to salient emotion stimuli. As childhood trauma is a risk factor for posttraumatic stress disorder, differentiating between their neurophysiological effects is necessary to elucidate the neural pathways by which childhood trauma exposure contributes to increased posttraumatic stress disorder risks. Methods: Face-specific N170 evoked response potentials for backward-masked (non-conscious) and conscious threat (fear, angry) and non-threat (happy) faces were measured in 77 adults (18–64 years old, 64% women, 78% right-handed) symptomatic for posttraumatic stress disorder. Differences in N170 peak amplitudes for fear-versus-happy and angry-versus-happy faces at bilateral temporo-occipital (T5, T6) sites were computed. The effect of cumulative exposure to childhood interpersonal trauma, other childhood trauma, adult trauma, depression and posttraumatic stress disorder symptom severity on the N170 response was assessed using hierarchical multiple regression analyses. Results: T5 N170 peak amplitudes for non-conscious fear-versus-happy faces were inversely related to cumulative childhood interpersonal trauma after accounting for socio-demographic, clinical symptom and other trauma factors. Posttraumatic stress disorder Avoidance was positively associated with N170 peak amplitudes for non-conscious fear-versus-happy faces, primarily due to reduced N170 responsivity to happy faces. Conclusion: Childhood interpersonal trauma exposure is associated with reduced discrimination between fear and happy faces, while avoidance symptom severity is associated with dampened responsivity to automatically processed happy faces in posttraumatic stress disorder adults. Results are discussed in terms of the likely contributions of impaired threat discrimination and deficient reward processing during neural processing of salient emotion stimuli, to increased risks of posttraumatic stress disorder onset and chronicity in childhood interpersonal trauma–exposed adults.


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