Case of Moral Insanity or Congenital Moral Defect, with Commentary

1885 ◽  
Vol 31 (135) ◽  
pp. 360-366 ◽  
Author(s):  
D. Hack Tuke
Keyword(s):  
Male Patient ◽  
Moral Insanity ◽  
Medical Superintendent ◽  
Patient’S History ◽  
Moral Defect

When I was at the Kingston Asylum, Ontario, last August, a male patient had just escaped from the institution, and made a criminal assault upon a little girl in the neighbourhood. From the inquiries I made, I found that the case was one of much interest in its bearing on moral insanity (or imbecility), and I was allowed to read the notes in the well-kept case-book of the asylum. Dr. Metcalf, the medical superintendent, has very kindly permitted me to make use of them, and the farther particulars of this patient's history and acts, which he has given me. A photograph was subsequently taken, which I have had reproduced by the London Autotype Company. (See Plate.).

Severe refeeding syndrome after human chorionic gonadotropin diet: a potentially lethal complication

2021 ◽  
Vol 14 (11) ◽  
pp. e244011
Author(s):  
Max J Schunemann ◽  
Martina Bertschinger ◽  
Christian Trachsel ◽  
Esther Bachli
Keyword(s):  
Male Patient ◽  
Chorionic Gonadotropin ◽  
Human Chorionic Gonadotropin ◽  
Young Male ◽  
History Taking ◽  
Patient History ◽  
Refeeding Syndrome ◽  
Serum Electrolytes ◽  
Potential Harm ◽  
Patient’S History

We present the case of a young male patient who presented with paralysing muscle weakness due to severe hypokalaemia and hypophosphataemia. The initial patient history evaluations could not establish the aetiology. Only after we reviewed the patient’s history did he reveal that he had been following a severe calorie-restricted regime, the human chorionic gonadotropin diet, which had ended 2 days prior to developing symptoms. This information then allowed us to diagnose severe refeeding syndrome. As a further complication, the patient developed rhabdomyolysis. After correction of serum electrolytes, symptoms resolved completely. This case emphasises the potential harm of severely calorie-restricted diets, often recommended by online ‘experts’. Furthermore, we underline the importance of thorough history taking.

A Case of Moral Insanity with Pyromania

1911 ◽  
Vol 57 (236) ◽  
pp. 124-126 ◽  
Author(s):  
H. D. MacPhail
Keyword(s):  
Male Patient ◽  
Interesting Case ◽  
Moral Insanity

The following are a few notes of an interesting case of a male patient, who was admitted here in July, 1910, after giving himself up to the police, because he had an uncontrollable desire to set fire to haystacks:

Remission of Recurrent Cochlear Hydrops Associated With Bromocriptine Treatment for Macroprolactinoma

2019 ◽  
Vol 28 (3) ◽  
pp. 548-552
Author(s):  
Andro Košec ◽  
Ivan Kruljac ◽  
Jakov Ajduk
Keyword(s):  
Dopamine Receptors ◽  
Male Patient ◽  
Endolymphatic Hydrops ◽  
Young Male ◽  
Oral Steroid ◽  
Case Description ◽  
Diuretic Treatment ◽  
Potential Therapeutic Role ◽  
Transient Improvement ◽  
Systemic Glucocorticoids

Objective Current recommendations for cochlear hydrops treatment include systemic glucocorticoids and diuretics. Cochlear cells express dopamine receptors, although their role is unknown in the pathophysiology of cochlear hydrops. Case Description We report the case of remission of recurrent right-sided cochlear hydrops in a young male patient treated with bromocriptine due to pituitary macroprolactinoma. Transient improvement was observed after oral steroid and diuretic treatment, but cochlear hydrops recurred until the dose of bromocriptine was increased to 10 mg daily. Conclusion Bromocriptine may stimulate dopamine receptors in cochlear cells with potential therapeutic role in patients with cochlear hydrops. There are no widely accepted and effective treatments for endolymphatic hydrops, and identifying potential new and efficacious therapeutics is of high relevance.

Danon Disease: Clinical Presentation and Diagnostic Workup in a Case of a Male Patient with a Novel Mutation in the LAMP2 Gene

2016 ◽  
Vol 47 (S 01) ◽  
Author(s):  
A. Dieckmann ◽  
F. Majer ◽  
H. Hulkova ◽  
M. Farr ◽  
T. Kalina ◽  
...  
Keyword(s):  
Male Patient ◽  
Clinical Presentation ◽  
Novel Mutation ◽  
Diagnostic Workup ◽  
Danon Disease ◽  
Lamp2 Gene

Multi-dimensional Visualisation of Laboratory Findings and Functional Test Results for Analysing the Clinical Course of Disease in Medicine

1995 ◽  
Vol 34 (03) ◽  
pp. 302-308 ◽  
Author(s):  
C. J. Luz ◽  
W. Giere ◽  
R. Lüdecke ◽  
D. Jonas ◽  
A. J. W. Goldschmidt
Keyword(s):  
Clinical Course ◽  
Test Results ◽  
Geometrical Figure ◽  
Laboratory Findings ◽  
Course Of Disease ◽  
Quantitative Examination ◽  
Ward Rounds ◽  
Patient’S History ◽  
Graphical Primitive ◽  
Computerized Medical Record

Abstract:The illustration of a patient’s history by a graphical primitive is discussed. Illustration technology is presented which simultaneously represents quantitative examination findings (e. g., laboratory values) and qualitative findings (e. g., from function diagnostics) by a single geometrical figure. Depending on the medical results, this figure takes on characteristic forms which can be identified as patterns typical for a specific disease. The procedure developed is integrated in a user interface which is implemented in the form of a computerized medical record for use on a pentop computer. This portable computer assists the physician during ward rounds, supplies additional, intelligence-based information, serves quality control, and streamlines working procedures making them more efficient.

Severe Isolated Prothrombin Deficiency : an Acquired State with Complete Recovery

1962 ◽  
Vol 08 (02) ◽  
pp. 221-234 ◽  
Author(s):  
Simon Karpatkin ◽  
G.I.C Ingram ◽  
John B. Graham
Keyword(s):  
Vitamin K ◽  
Liver Damage ◽  
Complete Recovery ◽  
Factor Vii ◽  
Natural Vitamin ◽  
Patient’S History ◽  
Derivatives Of

SummaryA 62 year old man acquired a temporary, virtually complete, deficiency of prothrombin as measured by both the Iowa and Oxford 2-stage methods. The etiology agent (or agents) is not known, but one of the drugs listed in Table 1 or an interaction between several was probably responsible. There was no evidence of an inhibitor, and factors I (fibrinogen), V (AcG), VII (SPCA), VIII (AHF), IX (PTC), X (Stuart), and XII (Hageman) were demonstrated to be normal. There were other evidences of mild liver damage and the prothrombin deficiency did not respond immediately to injections of natural vitamin K. Greatly prolonged clotting times but only moderately prolonged prothrombin times and partial thromboplastin times were observed. Bleeding was very severe into many tissues. Thromboplastin generation proceeded normally in the patient’s pro-thrombin-deficient plasma, despite the appearance of only a trace of thrombin. This patient’s history and clotting profile argue against the concept that factor VII (SPCA) and IX (PTC) are derivatives of prothrombin.

Congenital Intercostal Hernia: A Rare Entity

JMS SKIMS ◽  
2018 ◽  
Vol 21 (1) ◽  
pp. 48
Author(s):  
Syed Muzamil Andrabi ◽  
Mohd Yousuf Dar ◽  
Javid Ahmad Bhat
Keyword(s):  
Male Patient ◽  
General Surgery ◽  
Rare Entity ◽  
Intercostal Hernia ◽  
History Of ◽  
Lateral Chest ◽  
Patient Department

A 35-year-old male patient presented to the General Surgery Out Patient Department with a history of swelling on the left lateral chest since birth. The swelling appeared during inspiration and disappeared during expiration. JMS 2018;21(1):48 

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