scholarly journals Role of Nitric Oxide in Regulation of Retinal Blood Flow in Response to Hyperoxia in Cats

2008 ◽  
Vol 49 (10) ◽  
pp. 4595 ◽  
Author(s):  
Naohiro Izumi ◽  
Taiji Nagaoka ◽  
Eiichi Sato ◽  
Kenji Sogawa ◽  
Hiroyuki Kagokawa ◽  
...  
Keyword(s):  
Nitric Oxide ◽  
Blood Flow ◽  
Retinal Blood Flow

Role of Nitric Oxide in Mediating Retinal Blood Flow Regulation in Cats

1999 ◽  
Vol 15 (4) ◽  
pp. 295-303 ◽  
Author(s):  
SEIYO HARINO ◽  
KAZUO NISHIMURA ◽  
KUNIKO KITANISHI ◽  
MINA SUZUKI ◽  
PETER REINACH
Keyword(s):  
Nitric Oxide ◽  
Blood Flow ◽  
Flow Regulation ◽  
Retinal Blood Flow ◽  
Blood Flow Regulation

Role of Neuronal Nitric Oxide Synthase in Regulating Retinal Blood Flow During Flicker-Induced Hyperemia in Cats

2015 ◽  
Vol 56 (5) ◽  
pp. 3113 ◽  
Author(s):  
Takafumi Yoshioka ◽  
Taiji Nagaoka ◽  
Youngseok Song ◽  
Harumasa Yokota ◽  
Tomofumi Tani ◽  
...  
Keyword(s):  
Nitric Oxide ◽  
Blood Flow ◽  
Nitric Oxide Synthase ◽  
Neuronal Nitric Oxide Synthase ◽  
Retinal Blood Flow ◽  
Oxide Synthase

Role of Nitric Oxide in Regulation of Retinal Blood Flow during Hypercapnia in Cats

2003 ◽  
Vol 44 (11) ◽  
pp. 4947 ◽  
Author(s):  
Eiichi Sato ◽  
Takashi Sakamoto ◽  
Taiji Nagaoka ◽  
Fumihiko Mori ◽  
Kaoru Takakusaki ◽  
...  
Keyword(s):  
Nitric Oxide ◽  
Blood Flow ◽  
Retinal Blood Flow

Role of Nitric Oxide on Papillary Blood Flow and Pressure Natriuresis

Hypertension ◽  
1995 ◽  
Vol 25 (3) ◽  
pp. 408-414 ◽  
Author(s):  
Francisco J. Fenoy ◽  
Paloma Ferrer ◽  
Luis Carbonell ◽  
Miguel García-Salom
Keyword(s):  
Nitric Oxide ◽  
Blood Flow ◽  
Pressure Natriuresis

scholarly journals The role of nitric oxide and endothelin on optic nerve head blood flow autoregulation

2012 ◽  
Vol 13 (Suppl 1) ◽  
pp. A28
Author(s):  
Doreen Schmidl ◽  
Agnes Boltz ◽  
Semira Kaya ◽  
René Werkmeister ◽  
Reinhard Told ◽  
...  
Keyword(s):  
Nitric Oxide ◽  
Blood Flow ◽  
Optic Nerve ◽  
Optic Nerve Head

Disruption of renal peritubular blood flow in lipopolysaccharide-induced renal failure: role of nitric oxide and caspases

2005 ◽  
Vol 289 (6) ◽  
pp. F1324-F1332 ◽  
Author(s):  
Manish M. Tiwari ◽  
Robert W. Brock ◽  
Judit K. Megyesi ◽  
Gur P. Kaushal ◽  
Philip R. Mayeux
Keyword(s):  
Nitric Oxide ◽  
Renal Failure ◽  
Blood Flow ◽  
Saline Group ◽  
No Production ◽  
Capillary Perfusion ◽  
Peritubular Capillary ◽  
Synthase Inhibitor ◽  
Peritubular Capillary Perfusion

Acute renal failure (ARF) is a frequent and serious complication of endotoxemia caused by lipopolysaccharide (LPS) and contributes significantly to mortality. The present studies were undertaken to examine the roles of nitric oxide (NO) and caspase activation on renal peritubular blood flow and apoptosis in a murine model of LPS-induced ARF. Male C57BL/6 mice treated with LPS ( Escherichia coli) at a dose of 10 mg/kg developed ARF at 18 h. Renal failure was associated with a significant decrease in peritubular capillary perfusion. Vessels with no flow increased from 7 ± 3% in the saline group to 30 ± 4% in the LPS group ( P < 0.01). Both the inducible NO synthase inhibitor l- N6-1-iminoethyl-lysine (l-NIL) and the nonselective caspase inhibitor benzyloxycarbonyl-Val-Ala-Asp fluoromethylketone (Z-VAD) prevented renal failure and reversed perfusion deficits. Renal failure was also associated with an increase in renal caspase-3 activity and an increase in renal apoptosis. Both l-NIL and Z-VAD prevented these changes. LPS caused an increase in NO production that was blocked by l-NIL but not by Z-VAD. Taken together, these data suggest NO-mediated activation of renal caspases and the resulting disruption in peritubular blood flow are an important mechanism of LPS-induced ARF.

Role of nitric oxide in angiotensin IV-induced increases in cerebral blood flow

1998 ◽  
Vol 74 (2-3) ◽  
pp. 185-192 ◽  
Author(s):  
Enikö A. Kramár ◽  
Radhika Krishnan ◽  
Joseph W. Harding ◽  
John W. Wright
Keyword(s):  
Nitric Oxide ◽  
Blood Flow ◽  
Cerebral Blood Flow ◽  
Angiotensin Iv
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