Allergen-Induced Increase in Non-Allergic Bronchial Responsiveness to Ultrasonic Mist

Linkage between bronchial responsiveness to methacholine and gene markers of IL-4 cytokine gene cluster and T-cell receptor alpha/delta gene complex in Korean nuclear families

Clinical & Experimental Allergy ◽

10.1046/j.1365-2222.2001.00952.x ◽

2001 ◽

Vol 31 (1) ◽

pp. 103-109 ◽

Cited By ~ 2

Author(s):

S.-H. Cho ◽

J.-W. Son ◽

Y.-Y. Koh ◽

K.-U. Min ◽

Y.-Y. Kim ◽

...

Keyword(s):

T Cell ◽

Gene Cluster ◽

T Cell Receptor ◽

Cell Receptor ◽

Gene Complex ◽

Cytokine Gene ◽

Gene Markers ◽

Bronchial Responsiveness ◽

Nuclear Families ◽

Cell Receptor Alpha

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Central Deposition of Aerosols as a Detemining Factor in Bronchial Responsiveness

Clinical Science ◽

10.1042/cs071082p ◽

1986 ◽

Vol 71 (s15) ◽

pp. 82P-82P

Author(s):

M K Gillett ◽

B A Briggs ◽

P D Snashall

Keyword(s):

Bronchial Responsiveness

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Comparison of 4 AM and 4 PM Bronchial Responsiveness to Hypertonic Saline in Asthma

Lung ◽

10.1007/s00408-006-0017-0 ◽

2006 ◽

Vol 184 (6) ◽

pp. 341-346 ◽

Cited By ~ 16

Author(s):

Erica Ferraz ◽

Marcos C. Borges ◽

J. Terra-Filho ◽

José A. B. Martinez ◽

Elcio O. Vianna

Keyword(s):

Hypertonic Saline ◽

Bronchial Responsiveness

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Serum Eosinophil Cationic Protein and Bronchial Responsiveness in Pediatric and Adolescent Asthma Patients

CHEST Journal ◽

10.1378/chest.116.2.301 ◽

1999 ◽

Vol 116 (2) ◽

pp. 301-305 ◽

Cited By ~ 10

Author(s):

Wilfried Gruber ◽

Ernst Eber ◽

Andreas Pfleger ◽

Manfred Modl ◽

Ingrid Meister ◽

...

Keyword(s):

Eosinophil Cationic Protein ◽

Cationic Protein ◽

Bronchial Responsiveness ◽

Asthma Patients

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Circadian Variation in Allergen and Nonspecific Bronchial Responsiveness in Asthma

Chronobiology International ◽

10.3109/07420529908998732 ◽

1999 ◽

Vol 16 (5) ◽

pp. 631-639 ◽

Cited By ~ 27

Author(s):

Nizar N. Jarjour

Keyword(s):

Circadian Variation ◽

Bronchial Responsiveness

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Effect of the Cyclooxygenase-2 Inhibitor Celecoxib on Bronchial Responsiveness and Cough Reflex Sensitivity in Asthmatics

Pulmonary Pharmacology & Therapeutics ◽

10.1006/pupt.2000.0274 ◽

2001 ◽

Vol 14 (2) ◽

pp. 93-97 ◽

Cited By ~ 14

Author(s):

Peter V. Dicpinigaitis

Keyword(s):

Cyclooxygenase 2 ◽

Cough Reflex Sensitivity ◽

Bronchial Responsiveness ◽

Cough Reflex

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Relation between the bronchial obstructive response to inhaled lipopolysaccharide and bronchial responsiveness to histamine.

Thorax ◽

10.1136/thx.47.4.288 ◽

1992 ◽

Vol 47 (4) ◽

pp. 288-291 ◽

Cited By ~ 36

Author(s):

O Michel ◽

R Ginanni ◽

R Sergysels

Keyword(s):

Bronchial Responsiveness

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Bronchial Responsiveness to Eucapnic Hyperventilation and Methacholine Following Exposure to Organic Dust

CHEST Journal ◽

10.1378/chest.122.1.363 ◽

2002 ◽

Vol 122 (1) ◽

pp. 363-368 ◽

Cited By ~ 8

Author(s):

Britt-Marie Sundblad ◽

Lena Palmberg ◽

Kjell Larsson

Keyword(s):

Organic Dust ◽

Bronchial Responsiveness

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Lack of interaction of hyperpnoea with methacholine and histamine in asthma

Clinical Science ◽

10.1042/cs0950611 ◽

1998 ◽

Vol 95 (5) ◽

pp. 611-619 ◽

Cited By ~ 2

Author(s):

Suree SOMPRADEEKUL ◽

Rana HEJAL ◽

Melissa McLANE ◽

K. A. LENNER ◽

J. A. NELSON ◽

...

Keyword(s):

Blood Flow ◽

Forced Expiratory Volume ◽

Additive Effects ◽

Positive Interaction ◽

Bronchial Responsiveness ◽

Individual Effects ◽

The Individual ◽

Airway Calibre ◽

Effector Mechanisms

1.The thermal precipitants of asthma (exercise and hyperventilation) appear to have a unique pathogenesis that does not alter bronchial responsiveness. In the present work, we tested whether hyperpnoea interacts with other constrictor stimuli. 2.To provide data on this issue, we exposed 17 subjects with asthma to isocapnic hyperventilation of frigid air (HV), methacholine (METH) and histamine (HIS) alone and in combination. 3.With HV (mean ventilation = 55.6±7.7 litres/min), METH (2.20±0.7 ;mmol/l) and HIS (10.35±5.04 ;mmol/l) alone, the decrements in forced expiratory volume in 1 ;s (FEV1) from baseline were 27.4±3.4, 27.4±3.8 and 32.4±3% respectively (n = 9). Giving the agonists simultaneously did not produce additive effects (ΔFEV1 HV+METH = 32.8±3.6%; HV+HIS = 28.7±5.1%). None of the individual or combined responses was significantly different from each other. Changing the sequence of the experiments and giving METH at the height of the HV-induced bronchial narrowing, instead of during hyperpnoea, did not alter the findings (n = 8). The maximum fall in FEV1 after both bronchoconstrictors in this experiment (ΔFEV1 = 32.3±4.3%) was not significantly different from either alone (HV = 22.8±1.0%; METH = 27.3±1.9%). When METH and HIS were administered together, however (n = 5), a positive interaction ensued (METH = 1.53±0.56 ;mmol/l, ΔFEV1 = 15.6±4.6%; HIS = 4.77±2.07 ;mmol/l, ΔFEV1 = 18.8±3.1%; METH+HIS ΔFEV1 = 33.4±5.2%; P< 0.001 compared with the individual effects). 4.These results indicate that HV does not interact with stimuli that directly or indirectly modulate airway calibre. It is unclear if this effect represents protection conferred from increased bronchial blood flow or derives from differences in effector mechanisms between the thermal and pharmacological agonists.

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