Observations on the Use and Limitations of Renal Vein Renins in Hypertensive Patients

2015 ◽  
pp. 146-149 ◽  
Author(s):  
M. Aurwell ◽  
K. Delin ◽  
G. Granerus
Keyword(s):  
Renal Vein ◽  
Hypertensive Patients

Renal vein prostaglandins in renovascular hypertensive patients

1985 ◽  
Vol 19 (3) ◽  
pp. 219-226 ◽  
Author(s):  
Y. Tabuchi ◽  
T. Ogihara ◽  
Y. Kumahara
Keyword(s):  
Renal Vein ◽  
Hypertensive Patients

Captopril-stimulated Renal Vein Renin in Hypertensive Patients With or Without Renal Artery Stenosis

1990 ◽  
Vol 3 (12_Pt_1) ◽  
pp. 918-926 ◽  
Author(s):  
Fumihiro Tomoda ◽  
Masanobu Takata ◽  
Shinya Ohashi ◽  
Hitoshi Ueno ◽  
Kiyoshi Ikeda ◽  
...  
Keyword(s):  
Renal Artery ◽  
Renal Artery Stenosis ◽  
Renal Vein ◽  
Artery Stenosis ◽  
Hypertensive Patients ◽  
Renal Vein Renin

scholarly journals THE VASOCONSTRICTOR ACTION OF PLASMA FROM HYPERTENSIVE PATIENTS AND DOGS

1940 ◽  
Vol 72 (3) ◽  
pp. 301-310 ◽  
Author(s):  
Irvine H. Page
Keyword(s):  
Femoral Artery ◽  
Renal Vein ◽  
Renal Hypertension ◽  
Peripheral Vein ◽  
Experimental Conditions ◽  
Chemical Mediator ◽  
Hypertensive Patients ◽  
Femoral Vessels ◽  
Experimental Renal Hypertension ◽  
Venous Plasma

1. Plasma from the renal vein, femoral artery or vein of normal dogs and plasma from the femoral artery, femoral and antecubital veins of man cause little or no vasoconstriction when added in small amounts to blood from a bilaterally nephrectomized dog used as perfusing medium in an isolated rabbit's ear. 2. Plasma from the femoral vessels and antecubital vein of patients with essential hypertension, malignant hypertension, and chronic nephritic hypertension causes marked vasoconstriction under the same circumstances. The plasma of dogs made hypertensive either by constriction of the parenchyma by the scar of silk perinephritis or by constriction of the renal artery by a clamp also causes pronounced vasoconstriction. 3. Plasma from the renal vein of normal dogs produces little or no vasoconstriction, but that of hypertensive dogs elicits vasoconstriction but usually not so marked as that elicited by plasma collected from peripheral vessels. A sample of renal venous plasma from one hypertensive patient caused severe vasoconstriction, not quite so intense as that produced by the peripheral vein plasma. 4. Since renin is liberated into the renal vein in large amounts in hypertensive dogs and reacts with renin-activator to produce angiotonin and since the conditions of the experiment are such as to enhance greatly the sensitivity of the ear preparation to angiotonin, it is believed that the vasoconstriction is the result of the presence of angiotonin in the peripheral blood. 5. Since vasoconstriction occurs under the same experimental conditions with plasma from both hypertensive patients and dogs, this is considered cogent evidence in favor of the view that the chemical mediator of both is similar and is possibly angiotonin. 6. A method is presented which is believed will distinguish between plasma from patients with normal blood pressure and that from those with hypertension, and between plasma from normal dogs and that from dogs with experimental renal hypertension.

Selective Renal-Vein Renin Sampling in Hypertensive Patients with Segmental Renal Lesions

1974 ◽  
Vol 290 (21) ◽  
pp. 1153-1157 ◽  
Author(s):  
Morris Schambelan ◽  
Morton Glickman ◽  
Jan R. Stockigt ◽  
Edward G. Biglieri
Keyword(s):  
Renal Vein ◽  
Hypertensive Patients ◽  
Renal Lesions ◽  
Renal Vein Renin

V484: Laparoscopic Spleno-Renal Venous Bypass for Left Renal Vein Nutcracker Syndrome

2007 ◽  
Vol 177 (4S) ◽  
pp. 161-162
Author(s):  
Benjamin I. Chung ◽  
Monish Aron ◽  
Nicholas J. Hegarty ◽  
Inderbir S. Gill
Keyword(s):  
Renal Vein ◽  
Left Renal Vein ◽  
Nutcracker Syndrome ◽  
Venous Bypass
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