Significance of Impaired Conduction between the Sinus Node and Right Atrium for Appearance of Absolute Arrhythmia in Mitral Stenosis

2015 ◽  
pp. 208-210
Author(s):  
L. Georgiev ◽  
A. Atanassov
Keyword(s):  
Right Atrium ◽  
Mitral Stenosis ◽  
Sinus Node

scholarly journals Dual-Chamber Pacemaker After Sinus Node Dysfunction and Enlarged Right Atrium

Circulation ◽  
2021 ◽  
Vol 143 (17) ◽  
pp. 1725-1728
Author(s):  
Emilio Arbas-Redondo ◽  
Álvaro Montes-Muñiz ◽  
Carlos A. Álvarez-Ortega
Keyword(s):  
Right Atrium ◽  
Sinus Node ◽  
Sinus Node Dysfunction ◽  
Dual Chamber ◽  
Dual Chamber Pacemaker

scholarly journals The arterial supply to the right atrium and the sinus node in classic tricuspid atresia

1988 ◽  
Vol 96 (5) ◽  
pp. 816-822 ◽  
Author(s):  
Sergio A. Battistessa ◽  
Siew Yen Ho ◽  
Robert H. Anderson ◽  
Audrey Smith ◽  
Philip B. Deverall
Keyword(s):  
Right Atrium ◽  
Sinus Node ◽  
Tricuspid Atresia ◽  
Arterial Supply ◽  
The Right

Cardiac Glycosides and Pacemaker Activity of the Sinus Node — A Microelectrode Study on the Isolated Right Atrium of the Rabbit

1978 ◽  
pp. 258-269 ◽  
Author(s):  
Gerhard Steinbeck ◽  
Felix I.M. Bonke ◽  
Maurits A. Allessie ◽  
Wim J.E.P. Lammers
Keyword(s):  
Right Atrium ◽  
Cardiac Glycosides ◽  
Sinus Node ◽  
Pacemaker Activity

Tumor Thrombus Extending from the Inferior Vena Cava into the Right Atrium: First Manifestation of Renal Neoplasia in a Patient with Mitral Stenosis

2005 ◽  
Vol 22 (8) ◽  
pp. 693-694 ◽  
Author(s):  
Stella Brili ◽  
Vassilis I. Barberis ◽  
Apostolos Drollias ◽  
John Barbetseas ◽  
Christodoulos Stefanadis
Keyword(s):  
Inferior Vena Cava ◽  
Right Atrium ◽  
Mitral Stenosis ◽  
Tumor Thrombus ◽  
Inferior Vena ◽  
Vena Cava ◽  
The Right ◽  
Renal Neoplasia

Calcitonin gene-related peptide as potential neurotransmitter in guinea pig right atrium

1986 ◽  
Vol 250 (4) ◽  
pp. H693-H698 ◽  
Author(s):  
A. Saito ◽  
S. Kimura ◽  
K. Goto
Keyword(s):  
Guinea Pig ◽  
Right Atrium ◽  
Sinus Node ◽  
Calcitonin Gene Related Peptide ◽  
Dependent Manner ◽  
Chronotropic Response ◽  
Related Peptide ◽  
Calcitonin Gene ◽  
The Right

The potential neurotransmitter role of calcitonin gene-related peptide (CGRP) in cardioacceleratory nonadrenergic noncholinergic (NANC) nerves was examined in the guinea pig right atrium in vitro. In the presence of atropine, a muscarinic antagonist, and atenolol, a beta-adrenoceptor antagonist, transmural nerve stimulation (TNS) of the isolated right atrium caused a positive chronotropic response, which is slow in both onset and decay. This TNS-induced slow response was assumed to be mediated by NANC nerves in the right atrium since tetrodotoxin inhibited the response. Dense distribution of CGRP-like immunoreactive (CGRP-I) nerves was demonstrated in the sinus node. Exogenously applied CGRP exerted a positive chronotropic effect on the isolated right atrium in a dose-dependent manner. Both CGRP-I nerves and NANC response induced by TNS were not affected by surgical sympathectomy and reserpine pretreatment but were abolished by the pretreatment of animals with capsaicin. The results suggest that CGRP is the neurotransmitter of cardioacceleratory NANC nerves in the right atrium of the guinea pig.

Giant right atrium in rheumatic mitral stenosis

1970 ◽  
Vol 79 (1) ◽  
pp. 28-35 ◽  
Author(s):  
William C. Roberts ◽  
J.O'Neal Humphries ◽  
Andrew G. Morrow
Keyword(s):  
Right Atrium ◽  
Mitral Stenosis ◽  
Rheumatic Mitral Stenosis

Effect of neurotransmitters on the activation sequence of the isolated atrium

1990 ◽  
Vol 258 (6) ◽  
pp. H1632-H1641 ◽  
Author(s):  
R. B. Schuessler ◽  
B. I. Bromberg ◽  
J. P. Boineau
Keyword(s):  
Right Atrium ◽  
Propagation Velocity ◽  
Sinus Node ◽  
Consistent Pattern ◽  
Differential Sensitivity ◽  
Crista Terminalis ◽  
Spontaneous Activation ◽  
Right Atria ◽  
Unipolar Electrograms ◽  
Activation Sequence

Unipolar electrograms were recorded from 360 sites in 20 isolated, perfused canine right atria. In 52% of the preparations, spontaneous activation started in the sinus node region. In 32% of the preparations, spontaneous activation was initiated at the junction of the intercaval band and the crista terminalis. Simultaneous multicentric initiation occurred from both sites in 16% of the preparations. With infusion of incrementally increasing doses of norepinephrine, the superior sinus node site consistently assumed dominance. Infusion of incrementally increasing doses of acetylcholine produced no consistent pattern of pacemaker dominance. Propagation velocity during pacing was unaffected by either neurotransmitter. The results demonstrate the presence of two different pacemaker sites in the posterior right atrium that function spontaneously, devoid of any active or significant autonomic input. Exchange of dominance between the two sites occurs in the presence of norepinephrine and acetylcholine. The two sites appear to have a differential sensitivity to norepinephrine but are not differentially sensitive to acetylcholine. Changes in the pattern of activation seen with either neurotransmitters are mediated exclusively by the site of initiation and not by changes in conduction.

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