Growth Hormone Responses to Administration of L-5-Hydroxytryptophan (L-5-HTP) in Manic-Depressive Psychoses

2015 ◽  
pp. 32-38 ◽  
Author(s):  
S. Takahashi ◽  
H. Kondo ◽  
M. Yoshimura ◽  
Y. Ochi
Keyword(s):  
Growth Hormone ◽  
Manic Depressive ◽  
Hormone Responses

Growth Hormone Responses to Administration of L-5-Hydroxytryptophan (L-5-HTP) in Manic-Depressive Psychoses

1973 ◽  
Vol 27 (3) ◽  
pp. 197-206
Author(s):  
Saburo Takahashi ◽  
Hisao Kondo ◽  
Manabu Yoshimura ◽  
Yukio Ochi ◽  
Teruya Yoshimi
Keyword(s):  
Growth Hormone ◽  
Manic Depressive ◽  
Hormone Responses

GROWTH HORMONE RESPONSES TO FRUCTOSE IN DIABETES MELLITUS

1974 ◽  
Vol 75 (3) ◽  
pp. 497-502
Author(s):  
Mayer B. Davidson ◽  
Roger M. Steele
Keyword(s):  
Diabetes Mellitus ◽  
Growth Hormone ◽  
Fasting Glucose ◽  
Area Under The Curve ◽  
Oral Glucose ◽  
Gh Secretion ◽  
Significant Difference ◽  
Duration Of Disease ◽  
Hormone Responses

ABSTRACT Since fructose is normally metabolized in diabetics and has recently been shown to stimulate GH secretion, it was used to assess GH responses in diabetics. Fourteen diabetics (9 on insulin) and 8 controls matched for weight were studied. Fructose, infused over 10 min, was compared to arginine, infused over 30 min, both at 0.5 g/kg. Samples were collected at 0, 30, 60, 90 and 120 min and GH responses assessed as area under the curve minus the fasting area. There was no significant difference between the GH responses in diabetics and controls to either agent. Responses to arginine and fructose were significantly correlated (r = 0.60, P < 0.01) in all subjects, but not related to therapy, duration of disease or fasting glucose (75–287 mg/100 ml) in the diabetics. Oral glucose blunted the GH response to fructose in 2 controls. It is concluded that 1) fructose can stimulate GH secretion in male diabetics; 2) however, fructose-stimulated GH responses are not increased in diabetes mellitus.

scholarly journals Growth hormone responses to repeated maximal cycle ergometer exercise at different pedaling rates

2002 ◽  
Vol 92 (2) ◽  
pp. 602-608 ◽  
Author(s):  
K. A. Stokes ◽  
M. E. Nevill ◽  
G. M. Hall ◽  
H. K. A. Lakomy
Keyword(s):  
Growth Hormone ◽  
Area Under The Curve ◽  
Cycle Ergometer ◽  
Mean Power ◽  
Ergometer Exercise ◽  
Hormone Responses ◽  
Sprint Cycling ◽  
Mean Power Output ◽  
Male Subjects ◽  
Serum Gh

The present study examined the growth hormone (GH) response to repeated bouts of maximal sprint cycling and the effect of cycling at different pedaling rates on postexercise serum GH concentrations. Ten male subjects completed two 30-s sprints, separated by 1 h of passive recovery on two occasions, against an applied resistance equal to 7.5% (fast trial) and 10% (slow trial) of their body mass, respectively. Blood samples were obtained at rest, between the two sprints, and for 1 h after the second sprint. Peak and mean pedal revolutions were greater in the fast than the slow trial, but there were no differences in peak or mean power output. Blood lactate and blood pH responses did not differ between trials or sprints. The first sprint in each trial elicited a serum GH response (fast: 40.8 ± 8.2 mU/l, slow: 20.8 ± 6.1 mU/l), and serum GH was still elevated 60 min after the first sprint. The second sprint in each trial did not elicit a serum GH response ( sprint 1 vs. sprint 2, P < 0.05). There was a trend for serum GH concentrations to be greater in the fast trial (mean GH area under the curve after sprint 1vs. after sprint 2: 1,697 ± 367 vs. 933 ± 306 min · mU−1 · l−1; P = 0.05). Repeated sprint cycling results in an attenuation of the GH response.

A Pilot Study of the Mechanism of Action of Desipramine

1981 ◽  
Vol 138 (3) ◽  
pp. 248-251 ◽  
Author(s):  
S. A. Checkley ◽  
A. P. Slade ◽  
E. Shur ◽  
S. Dawling
Keyword(s):  
Blood Pressure ◽  
Growth Hormone ◽  
Pilot Study ◽  
Mechanism Of Action ◽  
Depressed Patients ◽  
Sedative Effects ◽  
Before And After ◽  
Hormone Responses

SummaryTo test the hypothesis that desipramine alters α adrenoceptor function in depressed patients, the effects of clonidine upon growth hormone sedation and blood pressure have been measured in depressed patients before and after treatment with desipramine. After three weeks of treatment the hypotensive and sedative effects of clonidine were inhibited in all patients even though plasma desipramine concentrations at this time varied from 42 to 560 μg/l. Growth hormone responses to clonidine were enhanced in five of the six patients but this effect was not statistically significant. These findings are consistent with the hypothesis that in these patients desipramine altered α adrenoceptor function: other explanations are discussed.

Insulin-induced growth hormone responses in patients with prostatic carcinoma

Urology ◽  
1979 ◽  
Vol 13 (5) ◽  
pp. 490-493 ◽  
Author(s):  
S. Madajewicz ◽  
A. Bhargava ◽  
Z. Wajsman ◽  
A. Mittelman ◽  
G.P. Murphy
Keyword(s):  
Growth Hormone ◽  
Prostatic Carcinoma ◽  
Hormone Responses
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