Essential Fatty Acids in Serum Lecithin of Children with Atopic Dermatitis and in Umbilical Cord Serum of Infants with High or Low IgE Levels

1987 ◽  
Vol 82 (3-4) ◽  
pp. 422-423 ◽  
Author(s):  
Inga-Lisa Strannegård ◽  
Lars Svennerholm ◽  
Örjan Strannegård
Keyword(s):  
Fatty Acids ◽  
Atopic Dermatitis ◽  
Umbilical Cord ◽  
Essential Fatty Acids ◽  
Cord Serum

Supplementary therapy in canine atopic dermatitis

2019 ◽  
Vol 24 (8) ◽  
pp. 400-407
Author(s):  
Sue Paterson
Keyword(s):  
Fatty Acids ◽  
Atopic Dermatitis ◽  
Specific Immunotherapy ◽  
Essential Fatty Acids ◽  
Topical Therapy ◽  
Allergen Specific Immunotherapy ◽  
Canine Atopic Dermatitis ◽  
Adequate Control ◽  
Inflammatory Drugs ◽  
Anti Inflammatory

Therapy for canine atopic dermatitis can be described as foundation or supplementary. Foundation therapy, which refers to allergen-specific immunotherapy, ciclosporin, glucocorticoids, lokivetmab and oclacitinib, is needed in all but the mildest of cases of canine atopic dermatitis. Supplementary therapies include drugs such as antihistamines, essential fatty acids, topical therapy and other systemic anti-inflammatory drugs. While it is uncommon for any of these supplementary drugs to provide adequate control of many cases of canine atopic dermatitis, they can reduce the frequency of relapse and reduce the amount of foundation therapy that is required when used in combination with them.

Foundation therapy in canine atopic dermatitis

2019 ◽  
Vol 24 (7) ◽  
pp. 347-352
Author(s):  
Sue Paterson
Keyword(s):  
Fatty Acids ◽  
Atopic Dermatitis ◽  
Strong Evidence ◽  
Specific Immunotherapy ◽  
Essential Fatty Acids ◽  
Evidence Base ◽  
Allergen Specific Immunotherapy ◽  
Canine Atopic Dermatitis ◽  
Topical Emollient ◽  
Risk Of Relapse

Therapy in canine atopic dermatitis should be multimodal and should be individualised for each patient. Therapies can be described as foundation or supplementary. In all but the mildest cases, dogs require a foundation therapy for the control of their atopic dermatitis. These include allergen-specific immunotherapy, ciclosporin, glucocorticoids, lokivetmab or oclacitinib. All of these five therapeutic options have a strong evidence base to recommend their use in canine atopic dermatitis. Supplementary therapies, which include drugs such as antihistamines, essential fatty acids, topical emollient and antiseptics, help in treatment to improve barrier function and correct skin biome dysbiosis, but can rarely control canine atopic dermatitis in isolation. When combined with foundation therapy they can reduce the risk of relapse and the level of foundation therapy that is required. https://doi.org/10.12968/coan.2019.0020

Essential fatty acids and atopic dermatitis

1991 ◽  
Vol 2 (S1) ◽  
pp. 23-30 ◽  
Author(s):  
Stephen Wright ◽  
Dr. S. Wright
Keyword(s):  
Fatty Acids ◽  
Atopic Dermatitis ◽  
Essential Fatty Acids

scholarly journals Hsa-mir-144-3p expression is increased in umbilical cord serum of infants with atopic dermatitis

2019 ◽  
Vol 143 (1) ◽  
pp. 447-450.e11 ◽  
Author(s):  
Eishika Dissanayake ◽  
Yuzaburo Inoue ◽  
Shingo Ochiai ◽  
Akifumi Eguchi ◽  
Taiji Nakano ◽  
...  
Keyword(s):  
Atopic Dermatitis ◽  
Umbilical Cord ◽  
Cord Serum

Elevated umbilical cord serum TARC/CCL17 levels predict the development of atopic dermatitis in infancy

2010 ◽  
Vol 41 (2) ◽  
pp. 186-191 ◽  
Author(s):  
H. Miyahara ◽  
N. Okazaki ◽  
T. Nagakura ◽  
S. Korematsu ◽  
T. Izumi
Keyword(s):  
Atopic Dermatitis ◽  
Umbilical Cord ◽  
Cord Serum

Neonatal nutritional strategy of a viviparous elasmobranch with extremely low reproductive output

2020 ◽  
Vol 638 ◽  
pp. 107-121 ◽  
Author(s):  
BS Rangel ◽  
NE Hussey ◽  
Y Niella ◽  
LA Martinelli ◽  
AD Gomes ◽  
...  
Keyword(s):  
Fatty Acids ◽  
Reproductive Strategies ◽  
Trophic Ecology ◽  
Essential Fatty Acids ◽  
Physiological State ◽  
Maternal Investment ◽  
Early Life History ◽  
Nutritional State ◽  
Disc Width ◽  
Biomarker Analysis

Throughout evolutionary history, elasmobranchs have developed diverse reproductive strategies. Little focused work, however, has addressed how neonatal nutritional state is affected by differing degrees of maternal investment associated with these markedly different reproductive strategies. To investigate the effect of maternal investment on the nutritional quality of pups during the early life history of an extremely viviparous elasmobranch, quantitative biomarker analysis including lipids, fatty acids and stable isotopes was conducted. Using the cownose ray Rhinoptera bonasus (histotrophic viviparous) as a model, we found that pups were initially born in a positive nutritional state, enriched in physiologically important essential fatty acids and nitrogen and carbon stable isotope values (δ15N and δ13C), a result of maternal intrauterine transfer. A systematic decrease in some fatty acids and δ15N values, as well as a decrease in cholesterol with growth, confirmed that these substrates were derived from maternal resources and used in initial metabolic processes following birth. An observed increase in condition factor, plasma essential fatty acids and triglyceride:cholesterol ratio with increasing body size identified a progression towards successful independent foraging with pups not displaying marked nutritional deficiency or fasting phases. Our multi-tracer approach allowed the identification of 2 size classes of young rays (<50 and <70 cm disc width) that displayed distinct physiological states. Since prenatal maternal investment is critical for offspring condition and to promote successful foraging post birth, understanding the trophic ecology and physiological state of pups during their first year is critical to guide management and conservation within nursery grounds.

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