scholarly journals A Low-Nitrogen Low-Phosphorus Vegan Diet for Patients with Chronic Renal Failure

Nephron ◽  
1996 ◽  
Vol 74 (2) ◽  
pp. 390-394 ◽  
Author(s):  
Giuliano Barsotti ◽  
Ester Morelli ◽  
Adamasco Cupisti ◽  
Mario Meola ◽  
Lucia Dani ◽  
...  
Keyword(s):  
Renal Failure ◽  
Chronic Renal Failure ◽  
Vegan Diet ◽  
Low Phosphorus ◽  
Low Nitrogen

Effects of a Low-Phosphorus, Low-Nitrogen Diet Supplemented with Essential Amino Acids and Ketoanalogues on Serum Beta-Endorphin in Chronic Renal Failure

Nephron ◽  
1989 ◽  
Vol 53 (2) ◽  
pp. 129-132 ◽  
Author(s):  
F. Ciardella ◽  
A. Cupisti ◽  
G. Catapano ◽  
A. Guidi ◽  
A. Pasquinucci ◽  
...  
Keyword(s):  
Amino Acids ◽  
Renal Failure ◽  
Chronic Renal Failure ◽  
Essential Amino Acids ◽  
Beta Endorphin ◽  
Low Phosphorus ◽  
Low Nitrogen

Hypertriglyceridemia and Low-Phosphorus, Low-Nitrogen Diet in Patients with Chronic Renal Failure

Nephron ◽  
1987 ◽  
Vol 46 (3) ◽  
pp. 335-336 ◽  
Author(s):  
M. Aparicio ◽  
L. Potaux ◽  
V. de Precigout ◽  
M.H. Lafage ◽  
H. Gin ◽  
...  
Keyword(s):  
Renal Failure ◽  
Chronic Renal Failure ◽  
Low Phosphorus ◽  
Low Nitrogen

Renal and intestinal Pi transport adaptation to low phosphorus diet in uremic rats.

1993 ◽  
Vol 3 (12) ◽  
pp. 1930-1937
Author(s):  
M Loghman-Adham
Keyword(s):  
Renal Failure ◽  
Chronic Renal Failure ◽  
Adaptive Response ◽  
Kinetic Studies ◽  
Normal Diet ◽  
Adaptive Responses ◽  
Normal Response ◽  
Dietary Phosphorus ◽  
Pi Transport ◽  
Low Phosphorus

The normal response of the kidney and intestine to a low-phosphorus diet (LPD) is an increased rate of Na(+)-dependent Pi transport by their brush border membranes (BBM). Dietary phosphorus restriction is used to reduce Pi accumulation in chronic renal failure. It is not known, however, if the uremic state alters the adaptive responses to an LPD. The adaptive response of the renal and intestinal BBM vesicles (BBMV) to LPD in acutely uremic (NX) and sham-operated (SH) control rats placed on a normal diet or an LPD was studied. In renal BBMV, the initial Na+ gradient-dependent Pi transport was lower in NX than in SH rats. Na(+)-independent uptake was unchanged. Thyroparathyroidectomy did not reverse the reduced Pi transport in NX rats. Kinetic studies showed a reduction of the apparent Vmax for Pi in BBMV from NX compared with SH rats (738 +/- 69 and 1,078 +/- 90 pmol/5 s.mg for NX and SH rats, respectively; P < 0.05; N = 5) with no change in the apparent Km. In intestinal BBMV, the initial Na+ gradient-dependent Pi transport was not different between SH and NX rats. There was also no difference in Pi transport kinetics between SH and NX rats. The adaptive response to an LPD persisted in renal and intestinal BBMV from NX rats and was comparable to that observed in SH rats: +54% for SH versus +48% for NX rats in kidney BBMV and +70.2% for SH versus +71.8% for NX rats in intestinal BBMV.(ABSTRACT TRUNCATED AT 250 WORDS)

scholarly journals Progression of chronic renal failure on substituting a ketoacid supplement for an amino acid supplement.

1992 ◽  
Vol 2 (7) ◽  
pp. 1178-1185 ◽  
Author(s):  
M Walser ◽  
S Hill ◽  
L Ward
Keyword(s):  
Renal Failure ◽  
Chronic Renal Failure ◽  
Amino Acid ◽  
Essential Amino Acid ◽  
Plasma Lipids ◽  
Urinary Cortisol ◽  
Amino Acid Supplement ◽  
Low Protein ◽  
Acid Supplement ◽  
Low Phosphorus

Twelve patients with severe chronic renal failure (average initial GFR, 13 mL/min) were monitored for 4 to 23 months while receiving an essential amino acid supplement and were then switched to a ketoacid supplement for 6 to 40 months, while continuously receiving a very low-protein (0.3 g/kg), low-phosphorus (7 to 9 mg/kg) diet. Urinary urea N excretion indicated that actual dietary protein intake averaged 0.46 g/kg. Progression, estimated as the linear regression slope of radioisotopically determined GFR on time, slowed from -0.46 +/- 0.31 (SD) to -0.24 +/- 0.15 mL/min/month (P = 0.029). Serum urea N, creatinine, phosphate, and uric acid rose significantly as GFR fell; blood pressure, plasma lipids, and urinary urea excretion were unchanged. Urinary 17-hydroxy-corticosteroid excretion decreased 18%, but this change was only marginally significant (P = 0.087). There was no change in plasma or urinary cortisol or urinary aldosterone. Viewed in light of previous evidence that progression seldom slows when treatment remains constant, the results suggest that this ketoacid supplement slows progression by approximately half, compared with an essential amino acid supplement, with no change in diet.

Low protein and low phosphorus diet in patients with chronic renal failure: Influence on glucose tolerance and tissue insulin sensitivity

Metabolism ◽  
1987 ◽  
Vol 36 (11) ◽  
pp. 1080-1085 ◽  
Author(s):  
H. Gin ◽  
M. Aparicio ◽  
L. Potaux ◽  
V. de Precigout ◽  
J.L. Bouchet ◽  
...  
Keyword(s):  
Renal Failure ◽  
Chronic Renal Failure ◽  
Insulin Sensitivity ◽  
Glucose Tolerance ◽  
Low Protein ◽  
Low Phosphorus

Incorporation of Urea 15N in Amino Acids of Patients with Chronic Renal Failure on Low Nitrogen Diet

1968 ◽  
Vol 21 (5) ◽  
pp. 394-404 ◽  
Author(s):  
C. GIORDANO ◽  
C. DE PASCALE ◽  
C. BALESTRIERI ◽  
D. CITTADINI ◽  
A. CRESCENZI
Keyword(s):  
Amino Acids ◽  
Renal Failure ◽  
Chronic Renal Failure ◽  
Low Nitrogen
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