Synthesis of LH-RH by Rat Hypothalamic Tissue in vitro. II. Effect of Short-Term Orchiectomy and Estradiol Benzoate Therapy

1977 ◽  
Vol 24 (5-6) ◽  
pp. 325-332 ◽  
Author(s):  
R.W. Hall ◽  
E. Steinberger
Keyword(s):  
Estradiol Benzoate ◽  
Short Term ◽  
Hypothalamic Tissue ◽  
Lh Rh

scholarly journals Use of estradiol benzoate to induce ovulation in a short-term protocol for fixed-time AI in sheep

2015 ◽  
Vol 36 (3) ◽  
pp. 1419 ◽  
Author(s):  
Caliê Castilho ◽  
Marcelo Ferreira de Almeida ◽  
Ines Cristina Giometti ◽  
Marcelo Zoccolaro Costa ◽  
Luís Roberto Almeida Gabriel Filho ◽  
...  
Keyword(s):  
Fixed Time ◽  
Estradiol Benzoate ◽  
Short Term

The Effect of Estradiol Benzoate on the Pituitary Responsiveness to LH-RH in Male and Female Rats

1974 ◽  
Vol 15 (3-4) ◽  
pp. 182-188 ◽  
Author(s):  
J.A.M.J. van Dieten ◽  
J. Steijsiger ◽  
J. Dullaart ◽  
G.P. van Rees
Keyword(s):  
Estradiol Benzoate ◽  
Female Rats ◽  
Male And Female ◽  
Male And Female Rats ◽  
Lh Rh

Effect of estradiol benzoate on the response of pituitary gland to LH-RH

1981 ◽  
Vol 14 (11) ◽  
pp. xxxvii
Author(s):  
H. Maia ◽  
I.C. Barbosa ◽  
E. Tironi ◽  
E.M. Coutinho
Keyword(s):  
Pituitary Gland ◽  
Estradiol Benzoate ◽  
Lh Rh

scholarly journals Erythropoietin Inhibits Basal and Stimulated Corticotropin-Releasing Hormone Release from the Rat Hypothalamus via a Nontranscriptional Mechanism

Endocrinology ◽  
2007 ◽  
Vol 148 (10) ◽  
pp. 4711-4715 ◽  
Author(s):  
Giuseppe Tringali ◽  
Giacomo Pozzoli ◽  
Lucia Lisi ◽  
Pierluigi Navarra
Keyword(s):  
Gene Expression ◽  
Growth Factor ◽  
Hpa Axis ◽  
Statistical Significance ◽  
Vascular Endothelial ◽  
Short Term ◽  
Pathological Conditions ◽  
Hypothalamic Tissue ◽  
Endothelial Growth Factor ◽  
Hpa Function

Brain hypoxia-ischemia induces a local increase in the levels of erythropoietin (EPO) and vascular endothelial growth factor (VEGF); this condition is also associated with acute activation of the hypothalamo-pituitary-adrenal (HPA) axis, suggesting that increased levels of EPO and VEGF in the hypothalamus may play a role in the control of HPA function. Thus, in this study we used rat hypothalamic explants to investigate whether EPO and VEGF can directly modulate CRH release; the latter was assessed by RIA measurement of the peptide in the incubation medium and hypothalamic tissue. EPO and VEGF effects were studied in short-term (1–3 h) experiments under basal conditions or after stimulation with 56 mM KCl or 10 μM veratridine. We observed that EPO (1–10 nm) significantly reduced CRH release and, in parallel, increased intrahypothalamic CRH content. VEGF tended to reduce CRH release without reaching statistical significance. Moreover, EPO, but not VEGF, inhibited KCl- and veratridine-stimulated CRH release and counteracted the parallel decrease in intrahypothalamic CRH induced by the two secretagogues. EPO effects were not mediated by modification of CRH gene expression, either in the absence or the presence of KCl or veratridine; in this paradigm, KCl and veratridine per se did not modify CRH gene expression. Our findings suggest that EPO contributes to the regulation of the HPA axis activation; in pathological conditions such as brain ischemia, this growth factor may control the HPA axis function, preventing possible detrimental effects of HPA overactivation.

Effect of Ovariectomy and Estradiol Benzoate Treatment on LH-RH Induced LH and FSH Release in the Female Rabbit

1975 ◽  
Vol 6 (5-6) ◽  
pp. 372-379 ◽  
Author(s):  
L. Dufy-Barbe ◽  
P. Franchimont ◽  
B. Dufy ◽  
J.D. Vincent
Keyword(s):  
Estradiol Benzoate ◽  
Female Rabbit ◽  
Lh Rh
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