The ‘Myth’ of Involutional Melancholia

1982 ◽  
Vol 8 (3) ◽  
pp. 140-143 ◽  
Author(s):  
P. Rinieris
1942 ◽  
Vol 88 (373) ◽  
pp. 559-565 ◽  
Author(s):  
R. E. Hemphill ◽  
Max Reiss

During the past three years some clinical applications of corticotrophic hormone have been studied, and in this paper is described a form of involutional melancholia in which hypopituitarism and secondary hypoadrenalism are factors. Nine such cases were treated with corticotrophic hormone; in addition two cases of pituitary cachexia in young women were similarly treated.


1943 ◽  
Vol 89 (375) ◽  
pp. 274-277
Author(s):  
R. Gibson

In the development of involutional melancholia a variety of causes have from time to time been implicated, ranging from glandular upset of ovarian or pituitary origin to the stress of air-raids. In the bulk of cases the pathogenesis would appear to depend on the interaction of a constellation of predisposing and precipitating factors. In the present study a tentative attempt has been made to indicate a possible ethnic element in the aetiology of this psychosis.


1964 ◽  
Vol 110 (465) ◽  
pp. 244-254 ◽  
Author(s):  
G. Hopkinson

The genetic evidence concerning affective illness of later life is still conflicting and the relationship of such conditions to the manic-depressive psychosis unclear. Kallman (1955) believed that, genetically, involutional melancholia bore a closer relationship to schizophrenia than to the manic-depressive psychosis. An increased risk for schizophrenia amongst the relatives of such patients was not observed by Kay (1959) and Stenstedt (1952). Both these writers do however describe a lower loading for manic-depressive psychosis than would be found amongst the relations of manic-depressive patients, though a much higher incidence than in the general population. Both Stenstedt and Kay assumed that they were dealing with a heterogeneous group of patients containing both psychotic and neurotic depressions.


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