Central Hearing Loss with a Bilateral Inferior Colliculus Lesion

2003 ◽  
Vol 8 (2) ◽  
pp. 111-113 ◽  
Author(s):  
Dick L. Hoistad ◽  
Timothy C. Hain
Keyword(s):  
Hearing Loss ◽  
Inferior Colliculus ◽  
Central Hearing Loss

scholarly journals Reorganization of receptive fields following hearing loss in inferior colliculus neurons

Neuroscience ◽  
2007 ◽  
Vol 147 (2) ◽  
pp. 532-545 ◽  
Author(s):  
K. Barsz ◽  
W.W. Wilson ◽  
J.P. Walton
Keyword(s):  
Hearing Loss ◽  
Inferior Colliculus ◽  
Receptive Fields

scholarly journals Acute and Long-Term Effects of Noise Exposure on the Neuronal Spontaneous Activity in Cochlear Nucleus and Inferior Colliculus Brain Slices

2014 ◽  
Vol 2014 ◽  
pp. 1-8 ◽  
Author(s):  
Moritz Gröschel ◽  
Jana Ryll ◽  
Romy Götze ◽  
Arne Ernst ◽  
Dietmar Basta
Keyword(s):  
Hearing Loss ◽  
Spontaneous Activity ◽  
Inferior Colliculus ◽  
Cochlear Nucleus ◽  
Noise Exposure ◽  
Brain Slices ◽  
Noise Induced Hearing Loss ◽  
Long Term Effects ◽  
Noise Trauma

Noise exposure leads to an immediate hearing loss and is followed by a long-lasting permanent threshold shift, accompanied by changes of cellular properties within the central auditory pathway. Electrophysiological recordings have demonstrated an upregulation of spontaneous neuronal activity. It is still discussed if the observed effects are related to changes of peripheral input or evoked within the central auditory system. The present study should describe the intrinsic temporal patterns of single-unit activity upon noise-induced hearing loss of the dorsal and ventral cochlear nucleus (DCN and VCN) and the inferior colliculus (IC) in adult mouse brain slices. Recordings showed a slight, but significant, elevation in spontaneous firing rates in DCN and VCN immediately after noise trauma, whereas no differences were found in IC. One week postexposure, neuronal responses remained unchanged compared to controls. At 14 days after noise trauma, intrinsic long-term hyperactivity in brain slices of the DCN and the IC was detected for the first time. Therefore, increase in spontaneous activity seems to develop within the period of two weeks, but not before day 7. The results give insight into the complex temporal neurophysiological alterations after noise trauma, leading to a better understanding of central mechanisms in noise-induced hearing loss.

scholarly journals Clinical and pathophysiological study of patients with vertigo at SAIMS, Indore

2019 ◽  
Vol 5 (4) ◽  
pp. 1009
Author(s):  
Digant Patni ◽  
Alok Tyagi ◽  
Vishal R. Munjal
Keyword(s):  
Hearing Loss ◽  
Pure Tone ◽  
Pure Tone Audiometry ◽  
Descriptive Study ◽  
Medical Sciences ◽  
Myogenic Potential ◽  
Mixed Hearing Loss ◽  
Sri Aurobindo ◽  
Number Of Patients ◽  
Central Hearing Loss

<p class="abstract"><strong>Background:</strong> This was a prospective, descriptive study performed by collecting and analyzing the results of vestibular exams, evoked myogenic potential tests, pure tone audiometry test and impedence test performed in the Otorhinolaryngology Department of Sri Aurobindo Institute of Medical Sciences, Indore.</p><p class="abstract"><strong>Methods:</strong> This was a prospective, descriptive study performed by collecting and analyzing the results of vestibular exams, evoked myogenic potential tests, pure tone audiometry test and impedence test performed in the Otorhinolaryngology Department of Sri Aurobindo Institute of Medical Sciences, Indore.  </p><p class="abstract"><strong>Results:</strong> In age group of 20 to 60, maximum number of patients were in group of 31-40 (16), followed by 51-60 (15), followed by 20-30 (24.) Maximum positive seen in Romberg (07), followed by nystagmus (06), followed by Dix-Hallpike (06). The patients in our study (30 out of 50) were suffering from hypertension. Two patients were of Meniere’s disease and rest 16 were of SNHL, 08 were of SNHL and BPPV combined. The rest of the cases were of mixed hearing loss and other central hearing loss causes will have be ruled out.</p><p class="abstract"><strong>Conclusions:</strong> The main aim of study is to study clinicopathological aspects in patients with vertigo. We also studied incidence of SNHL in our study. It has been established by this study that hypertension is one the major causes of vertigo in patients whereas VEMP has not proven to be very beneficial in our study to see peripheral vertigo patients. Most of the patients with BPPV also had a normal cVEMP which showed that it was not a very reliable instrument in BPPV.</p>

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