Urinary Composition and Lithogenic Risk in Normal Subjects following Oligomineral versus Bicarbonate-Alkaline High Calcium Mineral Water Intake

2001 ◽  
Vol 67 (1) ◽  
pp. 49-53 ◽  
Author(s):  
Giorgio Coen ◽  
Daniela Sardella ◽  
Giovanni Barbera ◽  
Michele Ferrannini ◽  
Carmela Comegna ◽  
...  
Keyword(s):  
Water Intake ◽  
Mineral Water ◽  
Normal Subjects ◽  
High Calcium ◽  
High Calcium Mineral Water

Effect of Calcium Supplementation as a High-Calcium Mineral Water on Bone Loss in Early Postmenopausal Women

1996 ◽  
Vol 59 (4) ◽  
pp. 238-239 ◽  
Author(s):  
C. Cepollaro ◽  
G. Orlandi ◽  
S. Gonnelli ◽  
G. Ferrucci ◽  
J. C. Arditti ◽  
...  
Keyword(s):  
Bone Loss ◽  
Postmenopausal Women ◽  
Mineral Water ◽  
Calcium Supplementation ◽  
Early Postmenopausal Women ◽  
High Calcium ◽  
High Calcium Mineral Water

Absorbability of the calcium in a high-calcium mineral water

1994 ◽  
Vol 4 (6) ◽  
pp. 323-324 ◽  
Author(s):  
R. P. Heaney ◽  
M. S. Dowell
Keyword(s):  
Mineral Water ◽  
High Calcium ◽  
High Calcium Mineral Water

Consumption of a high calcium mineral water lowers biochemical indices of bone remodeling in postmenopausal women with low calcium intake

2005 ◽  
Vol 16 (10) ◽  
pp. 1203-1209 ◽  
Author(s):  
Pierre J. Meunier ◽  
Cecile Jenvrin ◽  
Françoise Munoz ◽  
Viviane de la Gueronnière ◽  
Patrick Garnero ◽  
...  
Keyword(s):  
Postmenopausal Women ◽  
Bone Remodeling ◽  
Calcium Intake ◽  
Mineral Water ◽  
Low Calcium ◽  
High Calcium ◽  
Biochemical Indices ◽  
High Calcium Mineral Water

Drinking mineral water in the correction of metabolic syndrome in patients with cicatricial skin changes

2021 ◽  
pp. 56-61 ◽  
Author(s):  
V. K. Frolkov ◽  
S. N Nagornev ◽  
K. V. Kazantseva
Keyword(s):  
Insulin Resistance ◽  
Metabolic Syndrome ◽  
Water Intake ◽  
Mineral Water ◽  
Clinical Manifestations ◽  
Practical Interest ◽  
Resistance Index ◽  
Mineral Waters ◽  
Skin Changes ◽  
Lipid Metabolism Disorders

The article analyzes the efficiency of complex physiotherapy in combination with internal intake of medium-mineralized water “Essentuki № 17” in patients with cicatricial skin changes in the presence or absence of metabolic syndrome. It was found that internal mineral water intake supplementing physiotherapy effectively corrected carbohydrate and lipid metabolism disorders by reducing the insulin resistance index, which contributed to a greater regression of clinical manifestations of cicatricial skin changes in patients with metabolic syndrome. Meanwhile, in the absence of metabolic syndrome, the course intake of mineral water did not signifi cantly affect complex physiotherapy, although the index of insulin resistance and body weight of patients significantly decreased. It is suggested that, given the presence of nonspecifi c preventive potential of the course of drinking mineral waters intake, there is some theoretical and possibly practical interest in research on pre-course mineral water intake before performing cosmetic operations to accelerate the process of skin regeneration and the formation of scarring in a less pronounced form.

Free and total magnesium in lymphocytes of migraine patients — effect of magnesium-rich mineral water intake

2000 ◽  
Vol 295 (1-2) ◽  
pp. 63-75 ◽  
Author(s):  
Jean Thomas ◽  
Jean-Marc Millot ◽  
Stéphane Sebille ◽  
Anne-Marie Delabroise ◽  
Elisabeth Thomas ◽  
...  
Keyword(s):  
Water Intake ◽  
Mineral Water ◽  
Total Magnesium

scholarly journals Long-Term Potable Effects of Alkalescent Mineral Water on Intestinal Microbiota Shift and Physical Conditioning

2019 ◽  
Vol 2019 ◽  
pp. 1-10 ◽  
Author(s):  
Takaaki Yahiro ◽  
Takao Hara ◽  
Takashi Matsumoto ◽  
Emi Ikebe ◽  
Nichole Fife-Koshinomi ◽  
...  
Keyword(s):  
Gut Microbiota ◽  
Water Intake ◽  
Visceral Fat ◽  
Mineral Water ◽  
Terminal Point ◽  
Northwestern Part ◽  
Sequencing Analysis ◽  
Metagenomic Sequencing ◽  
Fat Accumulation ◽  
Visceral Fat Accumulation

Background. An alkalescent (pH 8.3) mineral water (AMW) of Hita basin, located in the northwestern part of Kyushu island in Japan, has been recognized for the unique quality of ingredients including highly concentrated silicic acid, sodium, potassium, and hydrogen carbonate. The biological effects of AMW intake were evaluated with a particular focus on its “antiobesity” properties through its modulation of the gut microbiota population. Methods. Two groups of C57BL6/J mice (8-week-old male) were maintained with a standard diet and tap water (control: TWC group) or AMW (AMW group) for 6 months and the following outputs were quantitated: (1) food and water intake, (2) body weight (weekly), (3) body fat measurements by CT scan (monthly), (4) sera biochemical values (TG, ALT, AST, and ALP), and (5) UCP-1 mRNA in fat tissues (terminal point). Two groups of ICR mice (7-week-old male) were maintained with the same method and their feces were collected at the 0, 1st, 3rd, and 6th month at which time the population rates of gut microbiota were quantitated using metagenomic sequencing analysis of 16S-rRNA. Results. Among all antiobesity testing items, even though a weekly dietary consumption was increased (p=0.012), both ratios of weight gain (p=1.21E−10) and visceral fat accumulation (p=0.029) were significantly reduced in the AMW group. Other criteria including water intake (p=0.727), the amounts of total (p=0.1602), and subcutaneous fat accumulation (p=0.052) were within the margin of error and UCP-1 gene expression level (p=0.171) in the AMW group was 3.89-fold higher than that of TWC. Among 8 major gut bacteria families, Lactobacillaceae (increased, p=0.029) and Clostridiaceae (decreased, p=0.029) showed significant shift in the whole population. Conclusion. We observed significantly reduced (1) weight gaining ratio (average −1.86%, up to −3.3%), (2) visceral fat accumulation ratio (average −4.30%, up to −9.1%), and (3) changes in gut microbiota population. All these consequences could support the “health benefit” functionality of AMW.

scholarly journals Calcium blood level modulates endogenous nitric oxide action: effects of parathroidectomy in patients with hyperparathyroidism

1998 ◽  
Vol 156 (2) ◽  
pp. 231-235 ◽  
Author(s):  
V Martina ◽  
GA Bruno ◽  
V Brancaleoni ◽  
E Zumpano ◽  
M Tagliabue ◽  
...  
Keyword(s):  
Nitric Oxide ◽  
Soluble Guanylate Cyclase ◽  
Normal Values ◽  
Normal Subjects ◽  
Cyclic Guanosine Monophosphate ◽  
Guanosine Monophosphate ◽  
Blood Levels ◽  
Free Calcium ◽  
No Production ◽  
High Calcium

Platelet cyclic guanosine monophosphate (cGMP) is produced by soluble guanylate cyclase (sGC), the activity of which is modulated by the activity of nitric oxide (NO) constitutive synthase (cNOS) which, in turn, is activated by a calcium/calmodulin complex. In primary hyperparathyroidism (H-PTH) an increase in platelet free calcium levels is present. In this study we evaluate the platelet cGMP levels, as an expression of NO production, in the presence of 3-isobutyl-1-methylxanthine (IBMX) alone (IBMXcGMP) and after stimulation by ionomycine (IONO; IONOcGMP) and sodium nitroprusside (SNP; SNPcGMP), in eight subjects affected by H-PTH before and after removal of adenoma. Platelet cGMP levels were also measured in seven normal subjects. IBMXcGMP and IONOcGMP were elevated in H-PTH patients compared with normal subjects (1.9 +/- 0.3 vs 0.8 +/- 0.2 fmol/10(6) platelets and 2.7 +/- 0.4 vs 1.4 +/- 0.3; P < 0.02 and P < 0.05 respectively) but SNPcGMP was unaffected (3.9 +/- 0.6 vs 2.5 +/- 0.5). After parathyroidectomy, blood levels of intact parathyroid hormone (i-PTH), total calcium (t-Ca), IBMXcGMP and IONOcGMP all decreased (177.5 +/- 23.9 vs 45.0 +/- 8.8 pg/ml, P < 0.005; 6.5 +/- 0.5 vs 4.6 +/- 0.1 mEq/1, P < 0.005; 1.9 +/- 0.3 vs 0.8 +/- 0.2, P < 0.005; 2.7 +/- 0.4 vs 1.8 +/ 0.3, P < 0.05 respectively), while SNPcGMP was not modified (3.9 +/- 0.6 vs 4.3 +/- 0.9). t-Ca and i-PTH were directly correlated with IBMXcGMP (P < 0.02, rs = 0.613; P < 0.02, rs = 0.576 respectively) and i-PTH was also correlated with t-Ca (P < 0.001), rs = 0.840). In conclusion: (1) levels of IBMXcGMP and IONOcGMP are high in subjects with H-PTH; (2) after surgery both IBMXcGMP and IONOcGMP decrease to normal values. As IBMXcGMP expresses basal cGMP and IONOcGMP expresses the cGMP after cNOS stimulation, it can be speculated that the increase in NO production could be a mechanism to downregulate the vasoconstriction which may be caused by the high calcium levels in smooth muscle cells. After surgery, together with the normalization of calcium levels, NO production also returned to normal values.

Effects of sulphur-containing mineral water intake on oxidative status and markers for inflammation in healthy subjects

2019 ◽  
pp. 1-10
Author(s):  
Todorka D. Sokrateva ◽  
Bogdan H. Roussev ◽  
Milka A. Nashar ◽  
Yoana D. Kiselova-Kaneva ◽  
Galya M. Mihaylova ◽  
...  
Keyword(s):  
Water Intake ◽  
Healthy Subjects ◽  
Mineral Water ◽  
Oxidative Status

Effect of citrate on the urinary excretion of calcium and oxalate: relevance to calcium oxalate nephrolithiasis.

1989 ◽  
Vol 35 (1) ◽  
pp. 23-28 ◽  
Author(s):  
D M Cowley ◽  
B C McWhinney ◽  
J M Brown ◽  
A H Chalmers
Keyword(s):  
Calcium Oxalate ◽  
Normal Subjects ◽  
Urinary Calcium ◽  
Urinary Calcium Excretion ◽  
Calcium Excretion ◽  
Molar Ratios ◽  
Oxalate Absorption ◽  
Stone Formers ◽  
Calcium Loading ◽  
High Calcium

Abstract Studies in 24 recurrent oxalate stone-formers have shown that values for urinary calcium excretion for this group on at-home diets vary significantly (P less than 0.001) more than values for creatinine excretions. By placing stone-formers on controlled in-hospital diets and measuring their calcium excretions, we were able to predict probable outpatient hypercalciuria (greater than 7.5 mmol/day) with a sensitivity of 95% and a specificity of 95%. In this study, the renal loss of calcium during low-calcium diets was proportional to the absorptive hypercalciuria during high-calcium diets. Calcium loading experiments in fasted stone-formers and normal subjects indicated that citrate, at citrate:calcium molar ratios ranging from 0.12 to 1, stimulated urinary calcium excretion more than did calcium carbonate loading alone. In addition, citrate also significantly (P less than 0.05) increased the excretion of urinary oxalate by two normal subjects for a given load of calcium oxalate. Malabsorption of citrate and possibly other hydroxycarboxylic acids may thus predispose to oxalate nephrolithiasis by promoting calcium and oxalate absorption.

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