Absorbability of the calcium in a high-calcium mineral water

1994 ◽  
Vol 4 (6) ◽  
pp. 323-324 ◽  
Author(s):  
R. P. Heaney ◽  
M. S. Dowell
1996 ◽  
Vol 59 (4) ◽  
pp. 238-239 ◽  
Author(s):  
C. Cepollaro ◽  
G. Orlandi ◽  
S. Gonnelli ◽  
G. Ferrucci ◽  
J. C. Arditti ◽  
...  

2001 ◽  
Vol 67 (1) ◽  
pp. 49-53 ◽  
Author(s):  
Giorgio Coen ◽  
Daniela Sardella ◽  
Giovanni Barbera ◽  
Michele Ferrannini ◽  
Carmela Comegna ◽  
...  

2005 ◽  
Vol 16 (10) ◽  
pp. 1203-1209 ◽  
Author(s):  
Pierre J. Meunier ◽  
Cecile Jenvrin ◽  
Françoise Munoz ◽  
Viviane de la Gueronnière ◽  
Patrick Garnero ◽  
...  

1996 ◽  
Vol 91 (3) ◽  
pp. 313-318 ◽  
Author(s):  
Martino Marangella ◽  
Corrado Vitale ◽  
Michele Petrarulo ◽  
Lidia Rovera ◽  
Franca Dutto

1. To assess whether the mineral content of drinking water influences both risk of stone formation and bone metabolism in idiopathic calcium nephrolithiasis, 21 patients were switched from their usual home diets to a 10 mmol calcium, low-oxalate, protein-controlled diet, supplemented with 21 of three different types of mineral water. Drinking water added 1, 6 and 20 mmol of calcium and 0.5, 10 and 50 mmol of bicarbonate respectively to the controlled diet. 2. The three controlled study periods lasted 1 month each and were separated by a 20 day washout interval. Blood and urine chemistries, including intact parathyroid hormone, calcitriol and two markers of bone resorption, were performed at the end of each study period. The stone-forming risk was assessed by calculating urine saturation with calcium oxalate (βCaOx), calcium phosphate (βbsh) and uric acid (βUA). 3. The addition of any mineral water produced the expected increase in urine output and was associated with similar decreases in βCaOx and βUA, whereas βbsh varied marginally. These equal decreases in βCaOx, however, resulted from peculiar changes in calcium, oxalate and citrate excretion during each study period. The increase in overall calcium intake due to different drinking water induced modest increases in calcium excretion, whereas oxalate excretion tended to decrease. The changes in oxalate excretion during any one study period compared with another were significantly related to those in calcium intake. Citrate excretion was significantly higher with the high-calcium, alkaline water. 4. Parathyroid hormone, calcitriol and markers of bone resorption increased when patients were changed from the high-calcium, alkaline to the low-calcium drinking water. 5. We suggest that overall calcium intake may be tailored by supplying calcium in drinking water. Adverse effects on bone turnover with low-calcium diets can be prevented by giving high-calcium, alkaline drinking water, and the stone-forming risk can be decreased as effectively as with low-calcium drinking water.


Author(s):  
Martin Poenie ◽  
Akwasi Minta ◽  
Charles Vorndran

The use of fura-2 as an intracellular calcium indicator is complicated by problems of rapid dye leakage and intracellular compartmentalization which is due to a probenecid sensitive anion transporter. In addition there is increasing evidence for localized microdomains of high calcium signals which may not be faithfully reported by fura-2.We have developed a new family of fura-2 analogs aimed at addressing some of these problems. These new indicators are based on a modified bapta which can be readily derivatized to produce fura-2 analogs with a variety of new properties. The modifications do not affect the chromophore and have little impact on the spectral and metal binding properties of the indicator. One of these new derivatives known as FPE3 is a zwitterionic analog of fura-2 that can be loaded into cells as an acetoxymethyl ester and whose retention in cells is much improved. The improved retention of FPE3 is important for both cuvettebased measurements of cell suspensions and for calcium imaging.


2017 ◽  
Vol 23 (2) ◽  
pp. 31-39
Author(s):  
Pyong-In Yi ◽  
Jung-hwan Kwon ◽  
So-hui Ko ◽  
Sung-chul Hong ◽  
Yong-jae Lee ◽  
...  

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