Gene Expression of Neurotrophins and Their High-Affinity Trk Receptors in Cultured Human Müller Cells

2002 ◽  
Vol 34 (1) ◽  
pp. 38-42 ◽  
Author(s):  
Hidehiro Oku ◽  
Tsunehiko Ikeda ◽  
Youichi Honma ◽  
Chie Sotozono ◽  
Kohji Nishida ◽  
...  
2020 ◽  
Vol 191 ◽  
pp. 107903 ◽  
Author(s):  
Javier Cáceres-del-Carpio ◽  
M. Tarek Moustafa ◽  
Jaime Toledo-Corral ◽  
Mohamed A. Hamid ◽  
Shari R. Atilano ◽  
...  

PLoS ONE ◽  
2013 ◽  
Vol 8 (5) ◽  
pp. e63467 ◽  
Author(s):  
Xin Wang ◽  
Jiawen Fan ◽  
Meng Zhang ◽  
Zhongcui Sun ◽  
Gezhi Xu

PLoS ONE ◽  
2012 ◽  
Vol 7 (11) ◽  
pp. e50417 ◽  
Author(s):  
Diana Katsman ◽  
Emma J. Stackpole ◽  
Daniel R. Domin ◽  
Debora B. Farber

2018 ◽  
Vol 17 (4) ◽  
pp. 255-260 ◽  
Author(s):  
Feng Gao ◽  
Lin-Jie Xu ◽  
Yuan Zhao ◽  
Xing-Huai Sun ◽  
Zhongfeng Wang

Background & Objective: Müller cell is the major type of glial cell in the vertebrate retina. Müller cells express various types of K+ channels, such as inwardly rectifying K+ (Kir) channels, big conductance Ca2+-activated K+ (BKCa) channels, delayed rectifier K+ channels (KDR), and transient A-type K+ channels. These K+ channels play important roles in maintaining physiological functions of Müller cells. Under some retinal pathological conditions, the changed expression and functions of K+ channels may contribute to retinal pathogenesis. Conclusion: In this article, we reviewed the physiological properties of K+ channels in retinal Müller cells and the functional changes of these channels in retinal disorders.


2021 ◽  
Vol 137 ◽  
pp. 111274
Author(s):  
Yuanyuan Tu ◽  
E Song ◽  
Zhenzhen Wang ◽  
Na Ji ◽  
Linling Zhu ◽  
...  

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