Acute Effects of a Class IA Antiarrhythmic Drug on the Ventricular Evoked Response Amplitude in Patients with Cardiac Pacemakers

Cardiology ◽  
2000 ◽  
Vol 94 (2) ◽  
pp. 103-105 ◽  
Author(s):  
Andreas Schuchert ◽  
Thomas Meinertz
1974 ◽  
Vol 4 (1) ◽  
pp. 19-27 ◽  
Author(s):  
Joan Mushin ◽  
Raymond Levy

SYNOPSISCortical somatosensory evoked responses to stimuli of various intensities were recorded in a group of patients with a diagnosis of ‘psychogenic pain’ and in three separate control groups. Differences emerged in the relationship between stimulus intensity and response amplitude. It is suggested that patients with psychogenic pain tended to deal with small stimuli in the way in which the other groups of patients dealt with large stimuli.


2013 ◽  
Vol 9 (1) ◽  
pp. 44 ◽  
Author(s):  
Hidehiko Okamoto ◽  
Henning Teismann ◽  
Sumru Keceli ◽  
Christo Pantev ◽  
Ryusuke Kakigi

1978 ◽  
Vol 21 (2) ◽  
pp. 408-416 ◽  
Author(s):  
John Cobb ◽  
Paul Skinner ◽  
John Burns

Two experiments studied the effects of signal rise time and frequency on the brainstem auditory evoked response. In Experiment 1, five different rise times were presented, fast (10 µsec), 0.5, 1, 2.5, and 5 msec at a center frequency of 1000 Hz at three sensation levels, 20, 40, and 60 dB. As rise time was increased, response amplitude and detectability decreased and response latency increased. In Experiment 2, tonal pips were presented at 250, 500, 1000, 2000, 4000, and 8000 Hz at 40-dB sensation level. Although the effects of frequency and signal rise time were confounded, no frequency effect was apparent.


1973 ◽  
Vol 16 (2) ◽  
pp. 297-308 ◽  
Author(s):  
David A. Nelson ◽  
Frank M. Lassman

Averaged auditory evoked vertex responses were obtained from eight normal-hearing subjects in response to 32 monaural 1000-Hz tone bursts at 30 combinations of recovery period and stimulus intensity. From curves describing N 1 -P 2 peak-to-peak amplitudes, an equation was derived that describes the combined effects of recovery period and stimulus intensity on evoked response amplitude. The results show evoked response amplitude to be a dual function of both recovery period and stimulus intensity. At a given stimulus intensity, evoked response amplitude increases as a logarithmic function of recovery period. At a given recovery period, evoked response amplitude increases as a power function of stimulus intensity. The combined effects of recovery period and stimulus intensity produce equal ratio changes in the slope of the recovery function with equal ratio changes in stimulus intensity.


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