Plasma Nitric Oxide Metabolites and Lipid Peroxide Levels in Preeclamptic Pregnant Women before and after Delivery

1999 ◽  
Vol 48 (4) ◽  
pp. 247-250 ◽  
Author(s):  
Ü. Mutlu-Türkoglu ◽  
G. Aykaç-Toker ◽  
L. Ibrahimoglu ◽  
E. Ademoglu ◽  
M. Uysal
Keyword(s):  
Nitric Oxide ◽  
Pregnant Women ◽  
Lipid Peroxide ◽  
Before And After ◽  
Nitric Oxide Metabolites

Nitric oxide metabolite in pregnant women before and after delivery

1997 ◽  
Vol 76 (4) ◽  
pp. 222-226 ◽  
Author(s):  
Toshiyuki Okutomi ◽  
Keiko Nomoto ◽  
Kunie Nakamura ◽  
Fumio Goto
Keyword(s):  
Nitric Oxide ◽  
Pregnant Women ◽  
Before And After

Erythrocyte Deformability and Nitric Oxide Metabolites in Athletes Before and After a Cardiopulmonary Test

2009 ◽  
Vol 19 (4) ◽  
pp. 306-310 ◽  
Author(s):  
Gregorio Caimi ◽  
Baldassare Canino ◽  
Gabriella Amodeo ◽  
Paolo Ingargiola ◽  
Daniela Lucido ◽  
...  
Keyword(s):  
Nitric Oxide ◽  
Erythrocyte Deformability ◽  
Before And After ◽  
Nitric Oxide Metabolites

Effects of 7 Days of Arginine-Alpha-Ketoglutarate Supplementation on Blood Flow, Plasma L-Arginine, Nitric Oxide Metabolites, and Asymmetric Dimethyl Arginine After Resistance Exercise

2011 ◽  
Vol 21 (4) ◽  
pp. 291-299 ◽  
Author(s):  
Darryn S. Willoughby ◽  
Tony Boucher ◽  
Jeremy Reid ◽  
Garson Skelton ◽  
Mandy Clark
Keyword(s):  
Nitric Oxide ◽  
Blood Flow ◽  
Resistance Exercise ◽  
Brachial Artery ◽  
Exercise Session ◽  
Artery Blood Flow ◽  
Before And After ◽  
Asymmetric Dimethyl Arginine ◽  
Nitric Oxide Metabolites ◽  
Dimethyl Arginine

Background:Arginine-alpha-ketoglutarate (AAKG) supplements are alleged to increase nitric oxide production, thereby resulting in vasodilation during resistance exercise. This study sought to determine the effects of AAKG supplementation on hemodynamics and brachial-artery blood flow and the circulating levels of L-arginine, nitric oxide metabolites (NOx; nitrate/nitrite), asymmetric dimethyl arginine (ADMA), and L-arginine:ADMA ratio after resistance exercise.Methods:Twenty-four physically active men underwent 7 days of AAKG supplementation with 12 g/day of either NO2 Platinum or placebo (PLC). Before and after supplementation, a resistance-exercise session involving the elbow flexors was performed involving 3 sets of 15 repetitions with 70–75% of 1-repetition maximum. Data were collected immediately before, immediately after (PST), and 30 min after (30PST) each exercise session. Data were analyzed with factorial ANOVA (p < .05).Results:Heart rate, blood pressure, and blood flow were increased in both groups at PST (p = .001) but not different between groups. Plasma L-arginine was increased in the NO2 group (p = .001). NOx was shown to increase in both groups at PST (p = .001) and at 30PST (p = .001) but was not different between groups. ADMA was not affected between tests (p = .26) or time points (p = .31); however, the L-arginine:ADMA ratio was increased in the NO2 group (p = .03).Conclusion:NO2 Platinum increased plasma L-arginine levels; however, the effects observed in hemodynamics, brachial-artery blood flow, and NOx can only be attributed to the resistance exercise.

scholarly journals Reduced Levels of Nitric Oxide Metabolites in Cerebrospinal Fluid Are Associated with Equine Protozoal Myeloencephalitis

2002 ◽  
Vol 9 (3) ◽  
pp. 605-610 ◽  
Author(s):  
Chinedu J. Njoku ◽  
William J. A. Saville ◽  
Stephen M. Reed ◽  
Michael J. Oglesbee ◽  
Päivi J. Rajala-Schultz ◽  
...  
Keyword(s):  
Nitric Oxide ◽  
Cerebrospinal Fluid ◽  
Natural Infection ◽  
Clinical Signs ◽  
Sarcocystis Neurona ◽  
Immune Effector ◽  
Equine Protozoal Myeloencephalitis ◽  
Clinically Normal ◽  
Before And After ◽  
Nitric Oxide Metabolites

ABSTRACT Equine protozoal myeloencephalitis (EPM) is a disease of horses that is primarily associated with infection with the apicomplexan Sarcocystis neurona. Infection with this parasite alone is not sufficient to induce the disease, and the mechanism of neuropathogenesis associated with EPM has not been reported. Nitric oxide (NO) functions as a neurotransmitter, a vasodilator, and an immune effector and is produced in response to several parasitic protozoa. The purpose of this work was to determine if the concentration of NO metabolites (NO x −) in the cerebrospinal fluid (CSF) is correlated with the development of EPM. CSF NO x − levels were measured before and after transport-stressed, acclimated, or dexamethasone-treated horses (n = 3 per group) were experimentally infected with S. neurona sporocysts. CSF NO x − levels were also compared between horses that were diagnosed with EPM after natural infection with S. neurona and horses that did not have clinical signs of disease or that showed no evidence of infection with the parasite (n = 105). Among the experimentally infected animals, the mean CSF NO x − levels of the transport-stressed group, which had the most severe clinical signs, was reduced after infection, while these values were found to increase after infection in the remaining groups that had less severe signs of EPM. Under natural conditions, horses with EPM (n = 65) had a lower mean CSF NO x − concentration than clinically normal horses with antibodies (Abs) against S. neurona (n = 15) in CSF, and horses that developed ataxia (n = 81) had a significantly lower mean CSF NO x − concentration than horses that did not have neurologic signs (n = 24). In conclusion, lower CSF NO x − levels were associated with clinical EPM, suggesting that measurement of CSF NO x − levels could improve the accuracy of diagnostic tests that are based upon detection of S. neurona-specific Abs in CSF alone and that reduced NO levels could be causatively related to the development of EPM.

Nitric Oxide Metabolites Before and After Spinal Anesthesia with Lidocaine

1998 ◽  
Vol 89 (Supplement) ◽  
pp. 1414A
Author(s):  
Ashwani K. Chhibber ◽  
Michael P. Eaton ◽  
Stewart J. Lustik ◽  
Salvatore Mauro ◽  
Jeremy Hogan
Keyword(s):  
Nitric Oxide ◽  
Spinal Anesthesia ◽  
Before And After ◽  
Nitric Oxide Metabolites
Sign in / Sign up

Export Citation Format

Share Document