Neurologic disease in reindeer (Rangifer tarandus tarandus) introduced into Ontario

1971 ◽  
Vol 49 (2) ◽  
pp. 159-166 ◽  
Author(s):  
Roy C. Anderson
Keyword(s):  
Odocoileus Virginianus ◽  
Rangifer Tarandus ◽  
Neurologic Disease ◽  
Rangifer Tarandus Tarandus ◽  
Pathologic Findings ◽  
History Of

In May, 1969, a small herd of reindeer (Rangifer tarandus tarandus) from Norway was placed in a recently enclosed area formerly inhabited by white-tailed deer (Odocoileus virginianus). The animals subsequently developed signs of neurologic disease. An analysis of clinical and pathologic findings, as well as a consideration of the history of the introduction, indicates the reindeer became infected in June with meningeal worm (Pneumostrongylus tenuis Dougherty, 1945) harbored by molluscs which had survived during the winter in the enclosure.

A protostrongylid nematode (Strongylida: Protostrongylidae) in woodland caribou (Rangifer tarandus caribou)

1976 ◽  
Vol 54 (5) ◽  
pp. 680-684 ◽  
Author(s):  
M. W. Lankester ◽  
V. J. Crichton ◽  
H. R. Timmermann
Keyword(s):  
Rangifer Tarandus ◽  
Neurologic Disease ◽  
Rangifer Tarandus Caribou ◽  
Woodland Caribou ◽  
Northwestern Ontario ◽  
Rangifer Tarandus Tarandus

First-stage protostrongylid larvae found in faeces of woodland caribou (Rangifer tarandus caribou) in northwestern Ontario and Manitoba may be larvae of Elaphostrongylus sp., a well-known agent of neurologic disease in reindeer (Rangifer tarandus tarandus) of Eurasia.

scholarly journals Clinical outbreak of babesiosis caused by Babesia capreoli in captive reindeer ( Rangifer tarandus tarandus ) in the Netherlands

2017 ◽  
Vol 8 (5) ◽  
pp. 799-801 ◽  
Author(s):  
Jan H. Bos ◽  
Fokko C. Klip ◽  
Hein Sprong ◽  
Els M. Broens ◽  
Marja J.L. Kik
Keyword(s):  
The Netherlands ◽  
Rangifer Tarandus ◽  
Rangifer Tarandus Tarandus

CAT SCRATCH SYNDROME

PEDIATRICS ◽  
1952 ◽  
Vol 10 (3) ◽  
pp. 311-318
Author(s):  
WILLIAM J. WATERS ◽  
SEYMOUR S. KALTER ◽  
JOHN T. PRIOR
Keyword(s):  
Skin Test ◽  
Laboratory Tests ◽  
Complement Fixation Test ◽  
Complement Fixation ◽  
Clinical Laboratory ◽  
Clinical Course ◽  
Diagnostic Value ◽  
Clinical Findings ◽  
Pathologic Findings ◽  
History Of

The clinical, laboratory and pathologic findings of a series of cases of cat scratch syndrome have been reviewed. In spite of a variable clinical course, certain features associated with a selected group of laboratory tests appear to be constant enough to be of diagnostic value. A history of contact with a cat and/or scratch which is usually associated with a peripheral skin lesion, lack of lymphangitis, presence of regional lymphadenopathy with tenderness to palpation are the most constant clinical findings. Fever, so frequently emphasized as a characteristic clinical sign, may be extremely variable in type and duration or entirely absent. A skin test with cat scratch antigen has been positive in all cases. Lacking this antigen, a negative Frei skin test in conjunction with a positive complement fixation test (Lygranum C. F.) is suggestive evidence for the diagnosis. With positive evidence from the above data, biopsy of an affected gland with its relatively nonspecific pathologic picture is not considered essential for the establishment of the diagnosis of cat scratch syndrome.

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