Effect of various fractions of the hot-water extract of germinating rye seed in eliciting biting by larvae of the prairie grain wireworm, Ctenicera destructor Brown

1971 ◽  
Vol 49 (1) ◽  
pp. 5-9 ◽  
Author(s):  
G. R. F. Davis

Factors in the hot-water extract of germinating rye seed, which elicit biting responses from larvae of Ctenicera destructor Brown, occurred both in the amino acid fraction and in the carbohydrate fraction of this extract. Factors in the carbohydrate fraction responsible for adding to the activity of the amino acid fraction, when both were recombined, were probably fructose, glucose, and sucrose, although chromatographic separation may have eliminated feeding deterrents. Hot-water extracts of 8-day-old and 11-day-old seedlings of the same rye were considerably less active than that of germinating seed. Plant age differences were suggested to influence the susceptibility and resistance of plants and plant-growth stages to wireworm attack.

1962 ◽  
Vol 15 (4) ◽  
pp. 287-290
Author(s):  
Yuki Yabuki ◽  
Teruo Iwasaki ◽  
Kiyoshi Izumi

1966 ◽  
Vol 44 (6) ◽  
pp. 995-1001 ◽  
Author(s):  
G. R. F. Davis

Larvae of Ctenicera destructor were fed nine synthetic diets for 6 months at 68 ± 2 °F and 75% relative humidity. Seven of the nine diets were based on a diet developed for larvae of Heliothis sp. It was modified by adding putrescine or spermidine, or both; hot-water or 70% ethanol extract of tea leaves; residue from hot-water extraction of tea leaves; or hot-water extract of germinating rye seed. Two chemically defined diets based on a previously reported amino acid analysis of this wireworm were also formulated: one contained hot-water extract of germinating rye seed.More larvae survived on a diet supplemented with putrescine and spermidine, on one supplemented with hot-water or with ethanol extract of tea leaves, and on the unsupplemented chemically defined diet than on the other diets. They more than doubled their weight on a diet supplemented with putrescine and spermidine. Growth factors for C. destructor present in tea-leaf extracts and residue probably differ from those for Adoxophyes orana. Inhibitory and growth-promoting factors for C. destructor are apparently present in hot-water extract of germinating rye seed. Putrescine or spermidine, or both, or some related metabolic product, may be necessary for growth and development of C. destructor. Frequency of molting was not always indicative of dietary efficiency.


2009 ◽  
Vol 38 (8) ◽  
pp. 977-982 ◽  
Author(s):  
Yoo-Seok Jeong ◽  
Hee-Kyoung Jung ◽  
Kwang-Sup Youn ◽  
Myoung-Ok Kim ◽  
Joo-Heon Hong

2010 ◽  
Vol 118 (2) ◽  
pp. 239-244 ◽  
Author(s):  
Tetsuro Ogawa ◽  
Hiromasa Tabata ◽  
Takuya Katsube ◽  
Yukari Ohta ◽  
Yukikazu Yamasaki ◽  
...  

2017 ◽  
Vol 6 ◽  
Author(s):  
Ryusei Uchio ◽  
Yohei Higashi ◽  
Yusuke Kohama ◽  
Kengo Kawasaki ◽  
Takashi Hirao ◽  
...  

AbstractTurmeric (Curcuma longa) is a widely used spice that has various biological effects, and aqueous extracts of turmeric exhibit potent antioxidant activity and anti-inflammatory activity. Bisacurone, a component of turmeric extract, is known to have similar effects. Oxidative stress and inflammatory cytokines play an important role in ethanol-induced liver injury. This study was performed to evaluate the influence of a hot water extract of C. longa (WEC) or bisacurone on acute ethanol-induced liver injury. C57BL/6 mice were orally administered WEC (20 mg/kg body weight; BW) or bisacurone (60 µg/kg BW) at 30 min before a single dose of ethanol was given by oral administration (3·0 g/kg BW). Plasma levels of aspartate aminotransferase and alanine aminotransferase were markedly increased in ethanol-treated mice, while the increase of these enzymes was significantly suppressed by prior administration of WEC. The increase of alanine aminotransferase was also significantly suppressed by pretreatment with bisacurone. Compared with control mice, animals given WEC had higher hepatic tissue levels of superoxide dismutase and glutathione, as well as lower hepatic tissue levels of thiobarbituric acid-reactive substances, TNF-α protein and IL-6 mRNA. These results suggest that oral administration of WEC may have a protective effect against ethanol-induced liver injury by suppressing hepatic oxidation and inflammation, at least partly through the effects of bisacurone.


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