Factors Affecting Renal Cortical Blood Flow. A Review

1975 ◽  
Vol 53 (1) ◽  
pp. 1-20 ◽  
Author(s):  
Serge Carrière
Keyword(s):  
Blood Flow ◽  
Cortical Blood Flow ◽  
Factors Affecting ◽  
Renal Cortical Blood Flow

Effects of Isosmolar Contrast Media on the Renal Cortical Blood Flow in Mice

1996 ◽  
Vol 37 (4) ◽  
pp. 587-590 ◽  
Author(s):  
Barry Högström ◽  
S.-O. Hietala ◽  
P. Rooth
Keyword(s):  
Blood Flow ◽  
Contrast Media ◽  
Cortical Blood Flow ◽  
Renal Cortical Blood Flow

Modulation of Renal Cortical Blood Flow During Static Exercise in Humans

1997 ◽  
Vol 80 (1) ◽  
pp. 62-68 ◽  
Author(s):  
Holly R. Middlekauff ◽  
Egbert U. Nitzsche ◽  
Alison H. Nguyen ◽  
Carl K. Hoh ◽  
G. Gary Gibbs
Keyword(s):  
Blood Flow ◽  
Static Exercise ◽  
Cortical Blood Flow ◽  
Renal Cortical Blood Flow ◽  
Exercise In Humans

Autoradiographic Identification of Kidney Angiotensin IV Binding Sites and Angiotensin IV-Induced Renal Cortical Blood Flow Changes in Rats

Peptides ◽  
1998 ◽  
Vol 19 (2) ◽  
pp. 269-277 ◽  
Author(s):  
J.K.M. Coleman ◽  
L.T. Krebs ◽  
T.A. Hamilton ◽  
B. Ong ◽  
K.A. Lawrence ◽  
...  
Keyword(s):  
Blood Flow ◽  
Binding Sites ◽  
Angiotensin Iv ◽  
Cortical Blood Flow ◽  
Renal Cortical Blood Flow ◽  
Flow Changes

Comparison of Nonpulsatile and Pulsatile Extracorporeal Circulation on Renal Cortical Blood Flow

1980 ◽  
Vol 29 (1) ◽  
pp. 57-62 ◽  
Author(s):  
James D. Sink ◽  
W. Randolph Chitwood ◽  
Ronald C. Hill ◽  
Andrew S. Wechsler
Keyword(s):  
Blood Flow ◽  
Extracorporeal Circulation ◽  
Cortical Blood Flow ◽  
Renal Cortical Blood Flow

scholarly journals Mechanism of the Redistribution of Renal Cortical Blood Flow during Hemorrhagic Hypotension in the Dog

1973 ◽  
Vol 52 (1) ◽  
pp. 39-47 ◽  
Author(s):  
Jay H. Stein ◽  
Sampanta Boonjarern ◽  
Richard C. Mauk ◽  
Thomas F. Ferris
Keyword(s):  
Blood Flow ◽  
Cortical Blood Flow ◽  
Hemorrhagic Hypotension ◽  
Renal Cortical Blood Flow

scholarly journals Increased activity and expression of Ca2+-dependent NOS in renal cortex of ANG II-infused hypertensive rats

1999 ◽  
Vol 277 (5) ◽  
pp. F797-F804 ◽  
Author(s):  
So Yeon Chin ◽  
Kailash N. Pandey ◽  
Shang-Jin Shi ◽  
Hiroyuki Kobori ◽  
Carol Moreno ◽  
...  
Keyword(s):  
Nitric Oxide ◽  
Blood Flow ◽  
Renal Cortex ◽  
Renal Medulla ◽  
Ang Ii ◽  
Sprague Dawley ◽  
Hypertensive Rats ◽  
Differential Distribution ◽  
Cortical Blood Flow ◽  
Renal Cortical Blood Flow

We have previously demonstrated that nitric oxide (NO) exerts a greater modulatory influence on renal cortical blood flow in ANG II-infused hypertensive rats compared with normotensive rats. In the present study, we determined nitric oxide synthase (NOS) activities and protein levels in the renal cortex and medulla of normotensive and ANG II-infused hypertensive rats. Enzyme activity was determined by measuring the rate of formation ofl-[14C]citrulline froml-[14C]arginine. Western blot analysis was performed to determine the regional expression of endothelial (eNOS), neuronal (nNOS), and inducible (iNOS) isoforms in the renal cortex and medulla of control and ANG II-infused rats. Male Sprague-Dawley rats were prepared by the infusion of ANG II at a rate of 65 ng/min via osmotic minipumps implanted subcutaneously for 13 days and compared with sham-operated rats. Systolic arterial pressures were 127 ± 2 and 182 ± 3 mmHg in control ( n = 13) and ANG II-infused rats ( n = 13), respectively. The Ca2+-dependent NOS activity, expressed as picomoles of citrulline formed per minute per gram wet weight, was higher in the renal cortex of ANG II-infused rats (91 ± 11) than in control rats (42 ± 12). Likewise, both eNOS and nNOS were markedly elevated in the renal cortex of the ANG II-treated rats. In both groups of rats, Ca2+-dependent NOS activity was higher in the renal medulla than in the cortex; however, no differences in medullary NOS activity were observed between the groups. Also, no differences in medullary eNOS levels were observed between the groups; however, medullary nNOS was decreased by 45% in the ANG II-infused rats. For the Ca2+-independent NOS activities, the renal cortex exhibited a greater activity in the control rats (174 ± 23) than in ANG II-infused rats (101 ± 10). Similarly, cortical iNOS was greater by 47% in the control rats than in ANG II-treated rats. No differences in the activity were found for the renal medulla between the groups. There was no detectable signal for iNOS in the renal medulla for both groups. These data indicate that there is a differential distribution of NOS activity, with the Ca2+-dependent activity and protein expression higher in the renal cortex of ANG II-infused rats compared with control rats, and support the hypothesis that increased constitutive NOS activity exerts a protective effect in ANG II-induced hypertension to maintain adequate renal cortical blood flow.

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