Are reports of temporary threshold shift-like symptoms by humans with normal hearing associated with hidden hearing loss?

2017 ◽  
Vol 141 (5) ◽  
pp. 3814-3814 ◽  
Author(s):  
Anthony J. Brammer ◽  
Gongqiang Yu ◽  
James J. Grady ◽  
Kourosh Parham ◽  
Martin G. Cherniack ◽  
...  
1978 ◽  
Vol 21 (2) ◽  
pp. 295-308
Author(s):  
Terry L. Wiley ◽  
Raymond S. Karlovich

Contralateral acoustic-reflex measurements were taken for 10 normal-hearing subjects using a pulsed broadband noise as the reflex-activating signal. Acoustic impedance was measured at selected times during the on (response maximum) and off (response minimum) portions of the pulsed activator over a 2-min interval as a function of activator period and duty cycle. Major findings were that response maxima increased as a function of time for longer duty cycles and that response minima increased as a function of time for all duty cycles. It is hypothesized that these findings are attributable to the recovery characteristics of the stapedius muscle. An explanation of portions of the results from previous temporary threshold shift experiments on the basis of acoustic-reflex dynamics is proposed.


2020 ◽  
pp. 1671-1673
Author(s):  
David Koh ◽  
Tar-Ching Aw

Noise can affect hearing in the occupational setting but can have other effects where exposures are non-occupational. For clinical purposes, noise is measured in decibels weighted according to the sensitivity of the human ear (dB(A)). Regardless of source, the effects of overexposure to noise are similar. Initially there is a temporary threshold shift, where reversibility of hearing loss is possible with removal away from further noise. Noise-induced hearing loss occurs following prolonged or intense exposure, with poor prospects for improvement of hearing. The classical audiogram for noise-induced hearing loss shows a 4 kHz dip. Non-auditory effects of prolonged noise exposure include annoyance, sleep disturbance, hypertension, and cardiovascular disease, stress, and impaired cognitive performance. Prevention of noise-induced hearing loss is by reducing exposure to noise at source minimizing exposure time, using hearing protection, and participating in surveillance.


1974 ◽  
Vol 39 (2) ◽  
pp. 163-168 ◽  
Author(s):  
Lynne Marshall ◽  
John F. Brandt

Temporary threshold shift resulting from exposure to one and five toy cap gun pistol shots was investigated using 11 normal-hearing adult subjects and one subject with a noise-induced hearing loss. The subjects fired the cap gun at arm’s length, and absolute thresholds at 4000 Hz were obtained before and after noise exposure by a fixed-frequency Bekesy technique. After exposure to one gunshot, five subjects showed a small TTS, five demonstrated no TTS, and two (including the subject with the hearing loss) exhibited negative TTS. No TTS occurred in any of the subjects after exposure to five shots. It was postulated that the small amount of TTS was due to the unexpectedly low sound pressure level produced by the cap gun and to the contraction of the middle ear muscles in some subjects prior to firing.


1974 ◽  
Vol 17 (1) ◽  
pp. 41-50 ◽  
Author(s):  
Raymond S. Karlovich ◽  
Terry L. Wiley

The test ear of each of nine normal-hearing subjects was exposed for three minutes to a 1000-Hz tone at 110 dB SPL. Either a 4000-Hz tone at 105 dB SPL or a broad-band noise at 100 dB SPL was presented to the contralateral ear during exposure. Four different temporal patterns were used for each contralateral signal: (1) continuous, (2) 18 seconds on/18 seconds off, (3) 1.8 seconds on/1.8 seconds off, and (4) 0.18 seconds on/0.18 seconds off. A control condition, consisting of the absence of contralateral stimulation, also was used. Pre- and postexposure thresholds for the test ear were tracked at a signal one-half octave above the exposure frequency. Resultant data indicated that reduction in temporary threshold shift was greatest for conditions involving rapidly pulsed (1.8 and 0.18 seconds on-off) contralateral signals. We hypothesized that these data were reflective of the dynamic properties of the acoustic reflex. Specifically, we posited that the acoustic reflex manifests less adaptation in response to rapid signal-repetition rates and relatively more adaptation to sustained or slowly pulsed signals.


1996 ◽  
Vol 99 (4) ◽  
pp. 2563-2574
Author(s):  
Yea‐Wen Shiau ◽  
Ernest M. Weiler ◽  
Laura Kretschmer ◽  
Angel Dell’aira Ball ◽  
Mary Anne Baker

2010 ◽  
pp. 1432-1434
Author(s):  
Syed M. Ahmed ◽  
Tar-Ching Aw

For clinical purposes, noise is measured in decibels weighted according to the sensitivity of the human ear (dB(A)). Regardless of source, the effects of overexposure to noise are similar. Initially there is a temporary threshold shift, where reversibility of hearing loss is possible with removal away from further noise. Permanent threshold shift occurs following prolonged and/or intense exposure, with poor prospects for improvement of hearing. The classical audiogram for noise-induced hearing loss shows a 4 kHz dip. Prevention is by reducing exposure to noise at source, and in the United Kingdom a limit for exposure has been set at 87 dB(A) averaged over an 8-h day or 140 dB(A) for any instantaneous impulse noise....


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