Comparison between otoacoustic and auditory brainstem response latencies supports slow backward propagation of otoacoustic emissions

2008 ◽  
Vol 123 (3) ◽  
pp. 1495-1503 ◽  
Author(s):  
Arturo Moleti ◽  
Renata Sisto
Keyword(s):  
Auditory Brainstem Response ◽  
Otoacoustic Emissions ◽  
Auditory Brainstem ◽  
Response Latencies ◽  
Brainstem Response ◽  
Backward Propagation

scholarly journals The Auditory Brainstem Response: Latencies Obtained in Children While under General Anesthesia

2012 ◽  
Vol 23 (01) ◽  
pp. 057-063 ◽  
Author(s):  
Linda W. Norrix ◽  
Stacey Trepanier ◽  
Matthew Atlas ◽  
Darlyne Kim
Keyword(s):  
General Anesthesia ◽  
Auditory Brainstem Response ◽  
Auditory Brainstem ◽  
Control Group ◽  
Neural Function ◽  
General Anesthetic ◽  
Response Latencies ◽  
Hearing Sensitivity ◽  
Brainstem Response ◽  
Group Delays

Background: The auditory brainstem response (ABR) test is frequently employed to estimate hearing sensitivity and assess the integrity of the ascending auditory system. In persons who cannot participate in conventional tests of hearing, a short-acting general anesthetic is used, recordings are obtained, and the results are compared with normative data. However, several factors (e.g., anesthesia, temperature changes) can contribute to delayed absolute and interpeak latencies, making it difficult to evaluate the integrity of the person's auditory brainstem function. Purpose: In this study, we investigated the latencies of ABR responses in children who received general anesthesia. Research Design: Between subject. Study Sample: Twelve children between the ages of 29 and 52 mo, most of whom exhibited a developmental delay but normal peripheral auditory function, comprised the anesthesia group. Twelve participants between the ages of 13 and 26 yr with normal hearing thresholds comprised the control group. Data Collection and Analysis: ABRs from a single ear from children, recorded under general anesthesia, were retrospectively analyzed and compared to those obtained from a control group with no anesthesia. ABRs were generated using 80 dB nHL rarefaction click stimuli. T-tests, corrected for alpha slippage, were employed to examine latency differences between groups. Results: There were significant delays in latencies for children evaluated under general anesthesia compared to the control group. Delays were observed for wave V and the interpeak intervals I–III, III–V, and I–V. Conclusions: Our data suggest that caution is needed in interpreting neural function from ABR data recorded while a child is under general anesthesia.

Auditory steady state response in auditory neuropathy

2010 ◽  
Vol 124 (9) ◽  
pp. 950-956 ◽  
Author(s):  
A A Emara ◽  
T A Gabr
Keyword(s):  
Steady State ◽  
Auditory Brainstem Response ◽  
Otoacoustic Emissions ◽  
Hearing Threshold ◽  
Auditory Brainstem ◽  
Auditory Neuropathy ◽  
Steady State Response ◽  
State Response ◽  
Auditory Steady State Response ◽  
Brainstem Response

AbstractReview:Auditory neuropathy is a disorder characterised by preservation of outer hair cell function, with normal otoacoustic emissions and/or cochlear microphonics, but an absent or distorted auditory brainstem response.Purpose:This study aimed to objectively assess hearing thresholds in patients with auditory neuropathy, using the auditory steady state response.Materials and methods:Thirteen patients with auditory neuropathy and 15 normal hearing subjects were examined. Audiological evaluation included basic audiological tests, otoacoustic emissions, auditory brainstem response and auditory steady state response.Results:In the auditory neuropathy patients, the auditory brainstem response was absent in 11 patients, while the auditory steady state response was absent in only three.Conclusion:The auditory steady state response may serve as a valuable objective measure for assessing the hearing threshold across different frequencies in patients with auditory neuropathy. We recommend that auditory steady state response be used to complete the evaluation of patients with auditory neuropathy.

scholarly journals Auditory brainstem response demonstrates that reduced peripheral auditory input is associated with self-report of tinnitus

2019 ◽  
Author(s):  
Naomi Bramhall ◽  
Garnett McMillan ◽  
Frederick Gallun ◽  
Dawn Konrad-Martin
Keyword(s):  
Animal Models ◽  
Auditory Brainstem Response ◽  
Otoacoustic Emissions ◽  
Auditory Brainstem ◽  
Self Report ◽  
Auditory Input ◽  
Distortion Product ◽  
Brainstem Response ◽  
Temporal Bones ◽  
The Relationship

Tinnitus is one of the predicted perceptual consequences of cochlear synaptopathy, a type of age-, noise-, or drug-induced auditory damage that has been demonstrated in animal models to cause homeostatic changes in central auditory gain. Although synaptopathy has been observed in human temporal bones, assessment of this condition in living humans is limited to indirect non-invasive measures such as the auditory brainstem response (ABR). In animal models, synaptopathy is associated with a reduction in ABR wave I amplitude at suprathreshold stimulus levels. Several human studies have explored the relationship between wave I amplitude and tinnitus, with conflicting results. This study investigates the hypothesis that reduced peripheral auditory input due to synaptic/neuronal loss is associated with tinnitus. ABR wave I amplitude data from 193 individuals (43 with tinnitus (22%), 150 without tinnitus (78%)), who participated in up to three out of four different studies, were included in a logistic regression analysis to estimate the relationship between wave I amplitude and tinnitus at a variety of stimulus levels and frequencies. Statistical adjustment for sex and distortion product otoacoustic emissions was included in the analysis. The results suggest that smaller ABR wave I amplitudes are associated with an increased probability of reporting tinnitus.

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