DCG recording with red light: discovery of a new electron-donor system

1990 ◽  
Author(s):  
Jeff Blyth
2020 ◽  
Vol 56 (11) ◽  
pp. 1721-1724 ◽  
Author(s):  
Yuqi Hou ◽  
Qingyun Liu ◽  
Jianzhang Zhao

Heavy atom-free dyads showing a red light-absorbing and exceptionally long-lived triplet state based on a spin–orbital charge transfer intersystem crossing mechanism.


2017 ◽  
Vol 16 (7) ◽  
pp. 1043-1048 ◽  
Author(s):  
Masaaki Yusa ◽  
Toshi Nagata
Keyword(s):  

Quinones were converted to hydroquinones by the action of red light (690 nm), with phthalocyanine as a photosensitizer and thiols as an electron donor.


2014 ◽  
Vol 26 (31) ◽  
pp. 315004 ◽  
Author(s):  
Stefano Prada ◽  
Livia Giordano ◽  
Gianfranco Pacchioni
Keyword(s):  

2019 ◽  
Vol 294 (34) ◽  
pp. 12708-12716 ◽  
Author(s):  
Rajib Sengupta ◽  
Lucia Coppo ◽  
Pradeep Mishra ◽  
Arne Holmgren

Weed Science ◽  
1972 ◽  
Vol 20 (4) ◽  
pp. 357-363 ◽  
Author(s):  
Charles E. Stanger ◽  
Arnold P. Appleby

Chloroplasts isolated from spinach(Spinacia oleraceaL.) leaves were used to study mechanisms of toxicity from 3-(3,4-dichlorophenyl)-1,1-dimethylurea (diuron). Light was needed to initiate diuron injury. The addition of ascorbate plus 2,6-dichlorophenylindolephenol (DPIP) as an electron donor system completely protected the chloroplasts from diuron-induced toxicity. The protective effect from the electron donor system occurred only in functional chloroplasts. Diuron caused rapid and extensive chlorophyll degradation at chlorophyll: diuron ratios of 200:1 and lower. At higher ratios the effect was much less measurable. The electron donor system gave complete protection in the presence of methylamine HCl, a known inhibitor of photophosphorylation, indicating that a deficiency of ATP was not the primary cause of diuron toxicity. Time-course studies showed that carotenoid pigments began to degrade before initiation of chlorophyll degradation. These results are interpreted as supporting a hypothesis that diuron induces phytotoxicity by catalyzing lethal photosensitized oxidations in the cell. This may occur as a result of (a) a greater concentration of oxidized chlorophyll caused by an interruption of electron flow and (b) an inhibition of NADPH formation which is necessary to maintain a functional carotenoid protective mechanism.


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