scholarly journals Lack of cholinergic innervation in gastric mucosa does not affect gastrin secretion or basal acid output in neurturin receptor GFRα2 deficient mice

2013 ◽  
Vol 591 (8) ◽  
pp. 2175-2188 ◽  
Author(s):  
Jussi Kupari ◽  
Jari Rossi ◽  
Karl-Heinz Herzig ◽  
Matti S. Airaksinen
Keyword(s):  
Gastric Mucosa ◽  
Acid Output ◽  
Cholinergic Innervation ◽  
Basal Acid Output ◽  
Gastrin Secretion ◽  
Basal Acid ◽  
Deficient Mice

scholarly journals Comparative study of the control of basal acid output from rodent isolated stomachs

1993 ◽  
Vol 109 (4) ◽  
pp. 941-945 ◽  
Author(s):  
Nicola J. Welsh ◽  
Nigel P. Shankley ◽  
James W. Black
Keyword(s):  
Comparative Study ◽  
Acid Output ◽  
Basal Acid Output ◽  
Basal Acid

Enhanced somatostatin secretion into the gastric juice with recovery of basal acid output afterHelicobacter pylorieradication in gastric ulcers

2003 ◽  
Vol 18 (5) ◽  
pp. 505-511 ◽  
Author(s):  
TOSHIHIKO HAYAKAWA ◽  
HIROSHI KANEKO ◽  
TOSHIHIRO KONAGAYA ◽  
KAORI SHINOZAKI ◽  
AKIHITO KASAHARA ◽  
...  
Keyword(s):  
Gastric Juice ◽  
Acid Output ◽  
Gastric Ulcers ◽  
Basal Acid Output ◽  
Somatostatin Secretion ◽  
Basal Acid

Exaggerated luminal secretion of basal and gastrin-stimulated somatostatin accompanied by recovery of basal acid output after H. pylori eradication in gastric ulcers

2001 ◽  
Vol 120 (5) ◽  
pp. A648
Author(s):  
Toshihiko Hayakawa ◽  
Hiroshi Kaneko ◽  
Toshihiro Konagaya ◽  
Kaori Shinozaki ◽  
Akihito Kasahara ◽  
...  
Keyword(s):  
Acid Output ◽  
Gastric Ulcers ◽  
Basal Acid Output ◽  
Basal Acid ◽  
H Pylori

Does corticosterone affect gastric mucosal cell growth during development?

1986 ◽  
Vol 250 (5) ◽  
pp. G633-G638
Author(s):  
C. C. Tseng ◽  
L. R. Johnson
Keyword(s):  
Gastric Mucosa ◽  
Protein Content ◽  
Acid Output ◽  
Biological Action ◽  
Mucosal Cell ◽  
Body Weights ◽  
Basal Acid ◽  
Oxyntic Gland ◽  
Precocious Development ◽  
Stimulation Of

The effect of corticosterone on the growth of the gastric mucosa was investigated during development. The weight of the mucosa and DNA, RNA, and protein content were used as parameters of growth. The pepsinogen content of the oxyntic gland was examined as an indicator of the biological action of corticosterone on development. Corticosterone-treated animals had lower body weights than control animals on days 10-20. This difference was not seen on day 25. Corticosterone administration during the first and second postnatal weeks induced the precocious appearance of pepsinogen in the oxyntic gland mucosa and increased basal acid output. These effects disappeared by day 25. The weight of the mucosa and DNA, RNA, and protein content of the oxyntic gland and antrum increased with age, but corticosterone had no effect on any of these parameters. We conclude that the induction of precocious development of the gastric mucosa by corticosterone is not due to the stimulation of growth.

Enhanced basal and gastrin-stimulated somatosatin secretion into the gastric juice with recovery of basal acid output after H. pylori eradication in gastric ulcers

2000 ◽  
Vol 94 (1-3) ◽  
pp. 19
Author(s):  
Toshihiko Hayakawa ◽  
Toshihiko Hayakawa ◽  
Hiroshi Kaneko ◽  
Kaori Shinozaki ◽  
Akihito Kasahara ◽  
...  
Keyword(s):  
Gastric Juice ◽  
Acid Output ◽  
Gastric Ulcers ◽  
Basal Acid Output ◽  
Basal Acid ◽  
H Pylori
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