Hyperbaric oxygen therapy accelerates wound healing in diabetic mice by decreasing active matrix metalloproteinase‐9

2019 ◽  
Vol 28 (2) ◽  
pp. 194-201 ◽  
Author(s):  
Trung T. Nguyen ◽  
Jeffrey I. Jones ◽  
William R. Wolter ◽  
Rocio L. Pérez ◽  
Valerie A. Schroeder ◽  
...  
2015 ◽  
Vol 23 (1) ◽  
pp. 98-103 ◽  
Author(s):  
Vivekananda Gupta Sunkari ◽  
Folke Lind ◽  
Ileana Ruxandra Botusan ◽  
Abad Kashif ◽  
Zhao-Jun Liu ◽  
...  

2000 ◽  
Vol 275 (4) ◽  
pp. 2661-2668 ◽  
Author(s):  
Matthew W. Olson ◽  
M. Margarida Bernardo ◽  
Martin Pietila ◽  
David C. Gervasi ◽  
Marta Toth ◽  
...  

Diabetes Care ◽  
2009 ◽  
Vol 32 (11) ◽  
pp. e138-e138 ◽  
Author(s):  
M. Muller ◽  
C. Trocme ◽  
F. Morel ◽  
S. Halimi ◽  
P. Y. Benhamou

2014 ◽  
Vol 133 (2) ◽  
pp. 208e-215e ◽  
Author(s):  
Phillip B. Dauwe ◽  
Benson J. Pulikkottil ◽  
Lawrence Lavery ◽  
James M. Stuzin ◽  
Rod J. Rohrich

2009 ◽  
Vol 106 (3) ◽  
pp. 988-995 ◽  
Author(s):  
Stephen R. Thom

The goal of this review is to outline advances addressing the role that reactive species of oxygen and nitrogen play in therapeutic mechanisms of hyperbaric oxygen. The review will be organized around major categories of problems or processes where controlled clinical trials have demonstrated clinical efficacy for hyperbaric oxygen therapy. Reactive species are now recognized to play a major role in cell signal transduction cascades, and the discussion will focus on how hyperbaric oxygen acts through these pathways to mediate wound healing and ameliorate postischemic and inflammatory injuries.


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