Visceral larva migrans syndrome: analysis of serum cytokine levels in children with hepatic lesions confirmed in radiological findings

2014 ◽  
Vol 36 (12) ◽  
pp. 668-673 ◽  
Author(s):  
K. Mazur-Melewska ◽  
K. Jończyk ◽  
A. Modlińska-Cwalińska ◽  
M. Figlerowicz ◽  
W. Służewski
1970 ◽  
Vol 80 (3) ◽  
pp. 407-IN12 ◽  
Author(s):  
S.T. Fernando ◽  
B. Vasudevan ◽  
M.H.M. Hamza ◽  
I.K.T. Panditha-Gunawardene ◽  
H.T. Samarasinghe

Cases Journal ◽  
2009 ◽  
Vol 2 (1) ◽  
pp. 6881 ◽  
Author(s):  
Małgorzata Paul ◽  
Jerzy Stefaniak ◽  
Hanna Twardosz-Pawlik ◽  
Krystyna Pecold

1996 ◽  
Vol 5 (1) ◽  
pp. 24-31 ◽  
Author(s):  
L. H. Faccioli ◽  
V. F. Mokwa ◽  
C. L. Silva ◽  
G. M. Rocha ◽  
J. I. Araujo ◽  
...  

This study was undertaken to evaluate the role of IL-5 in eosinophil migration and in the maintenance of eosinophilia in a guinea-pig model of visceral larva migrans syndrome. The results show that the infection of animals withToxocara canisinduced an early increase in serum IL-5 levels that might be essential for eosinophil differentiation and proliferation and for the development of eosinophilia. When infected guinea-pigs were treated with mAb anti-IL-5 (TRFK-5) given at the same time or 1 or 3 days after infection, there was a high percentage of reduction of eosinophil counts 18 days after infection. However, when the mAb was administered during the peak of eosinophilia, there was high inhibition in blood, no inhibition in bronchoalveolar lavage fluid (BALF) or peritoneum and an increase in eosinophil numbers in bone marrow. Thus, a basic level of IL-5 may be essential to drive eosinophils from bone marrow to blood and tissues, and for the maintenance of eosinophilia in infected animals. We may also conclude that when eosinophils have already migrated to the lungs, TRFK-5 has no power to inhibit eosinophilia, which is also under control of local lung cells producing IL-5. In this way, only one later TRFK-5 treatment may not be sufficient to modify the lung parenchyma microenvironment, sinceT. canisantigens had already stimulated some cell populations to produce IL-5.


2006 ◽  
Vol 249 (1) ◽  
pp. 7-12 ◽  
Author(s):  
Fujio Umehara ◽  
Hideki Ookatsu ◽  
Daisuke Hayashi ◽  
Akifumi Uchida ◽  
Yukari Douchi ◽  
...  

Author(s):  
Neeti Nagar ◽  
Neha K. Madan ◽  
Richa Mittal ◽  
Pradeep Kumar Debata ◽  
Sunil Ranga

VLM is a zoonotic disease caused by the migration of third-stage larvae of nematodes through the tissue of human viscera. Among various etiological agents such as Baylisascaris procyonis, Capillaria hepatica, Ascaris sum, and some Ancylostoma species, Toxocara is a major cause of VLM. Poor hygiene, contact with dogs and geophagia increases the risk of toxocariasis.Young adults and children who are in close contact with animals are at a higher risk. Here we present a case of 7 years male child presenting with fever, abdominal pain and vomiting. The clinical presentation, biochemical and radiological findings supported the diagnosis of VLM which was corroborated in the cytological examination. Here we report a rarest encounter of VLM in the cytology smear.


1994 ◽  
Vol 4 (1) ◽  
pp. 39-40 ◽  
Author(s):  
Sally B Zachariah ◽  
Babu Zachariah ◽  
Rachel Varghese

Infection ◽  
1980 ◽  
Vol 8 (4) ◽  
pp. 171-173 ◽  
Author(s):  
G. Maggiore ◽  
F. Sacchi ◽  
M. Marconi ◽  
E. Bianchi ◽  
A. Dei Cas ◽  
...  

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