Neuroimaging Studies of Cerebral “visceral Larva Migrans” Syndrome

1994 ◽  
Vol 4 (1) ◽  
pp. 39-40 ◽  
Author(s):  
Sally B Zachariah ◽  
Babu Zachariah ◽  
Rachel Varghese
Keyword(s):  
Larva Migrans ◽  
Visceral Larva Migrans ◽  
Visceral Larva Migrans Syndrome

Precipitin reactions in monkeys (Macaca sinica) experimentally infected with Toxocara canis and in children with visceral larva migrans syndrome

1970 ◽  
Vol 80 (3) ◽  
pp. 407-IN12 ◽  
Author(s):  
S.T. Fernando ◽  
B. Vasudevan ◽  
M.H.M. Hamza ◽  
I.K.T. Panditha-Gunawardene ◽  
H.T. Samarasinghe
Keyword(s):  
Toxocara Canis ◽  
Larva Migrans ◽  
Visceral Larva Migrans ◽  
Macaca Sinica ◽  
Visceral Larva Migrans Syndrome

scholarly journals The co-occurrence of Toxocara ocular and visceral larva migrans syndrome: a case series

Cases Journal ◽  
2009 ◽  
Vol 2 (1) ◽  
pp. 6881 ◽  
Author(s):  
Małgorzata Paul ◽  
Jerzy Stefaniak ◽  
Hanna Twardosz-Pawlik ◽  
Krystyna Pecold
Keyword(s):  
Case Series ◽  
Larva Migrans ◽  
Visceral Larva Migrans ◽  
Visceral Larva Migrans Syndrome

scholarly journals IL-5 drives eosinophils from bone marrow to blood and tissues in a guinea-pig model of visceral larva migrans syndrome

1996 ◽  
Vol 5 (1) ◽  
pp. 24-31 ◽  
Author(s):  
L. H. Faccioli ◽  
V. F. Mokwa ◽  
C. L. Silva ◽  
G. M. Rocha ◽  
J. I. Araujo ◽  
...  
Keyword(s):  
Bone Marrow ◽  
Guinea Pig ◽  
Lung Parenchyma ◽  
Lavage Fluid ◽  
Toxocara Canis ◽  
Pig Model ◽  
Larva Migrans ◽  
Visceral Larva Migrans ◽  
Guinea Pig Model ◽  
Visceral Larva Migrans Syndrome

This study was undertaken to evaluate the role of IL-5 in eosinophil migration and in the maintenance of eosinophilia in a guinea-pig model of visceral larva migrans syndrome. The results show that the infection of animals withToxocara canisinduced an early increase in serum IL-5 levels that might be essential for eosinophil differentiation and proliferation and for the development of eosinophilia. When infected guinea-pigs were treated with mAb anti-IL-5 (TRFK-5) given at the same time or 1 or 3 days after infection, there was a high percentage of reduction of eosinophil counts 18 days after infection. However, when the mAb was administered during the peak of eosinophilia, there was high inhibition in blood, no inhibition in bronchoalveolar lavage fluid (BALF) or peritoneum and an increase in eosinophil numbers in bone marrow. Thus, a basic level of IL-5 may be essential to drive eosinophils from bone marrow to blood and tissues, and for the maintenance of eosinophilia in infected animals. We may also conclude that when eosinophils have already migrated to the lungs, TRFK-5 has no power to inhibit eosinophilia, which is also under control of local lung cells producing IL-5. In this way, only one later TRFK-5 treatment may not be sufficient to modify the lung parenchyma microenvironment, sinceT. canisantigens had already stimulated some cell populations to produce IL-5.

MRI studies of spinal visceral larva migrans syndrome

2006 ◽  
Vol 249 (1) ◽  
pp. 7-12 ◽  
Author(s):  
Fujio Umehara ◽  
Hideki Ookatsu ◽  
Daisuke Hayashi ◽  
Akifumi Uchida ◽  
Yukari Douchi ◽  
...  
Keyword(s):  
Larva Migrans ◽  
Visceral Larva Migrans ◽  
Mri Studies ◽  
Visceral Larva Migrans Syndrome

Functional defects of neutrophils in visceral larva migrans syndrome

Infection ◽  
1980 ◽  
Vol 8 (4) ◽  
pp. 171-173 ◽  
Author(s):  
G. Maggiore ◽  
F. Sacchi ◽  
M. Marconi ◽  
E. Bianchi ◽  
A. Dei Cas ◽  
...  
Keyword(s):  
Larva Migrans ◽  
Visceral Larva Migrans ◽  
Functional Defects ◽  
Visceral Larva Migrans Syndrome

Visceral larva migrans syndrome: analysis of serum cytokine levels in children with hepatic lesions confirmed in radiological findings

2014 ◽  
Vol 36 (12) ◽  
pp. 668-673 ◽  
Author(s):  
K. Mazur-Melewska ◽  
K. Jończyk ◽  
A. Modlińska-Cwalińska ◽  
M. Figlerowicz ◽  
W. Służewski
Keyword(s):  
Larva Migrans ◽  
Serum Cytokine ◽  
Visceral Larva Migrans ◽  
Radiological Findings ◽  
Cytokine Levels ◽  
Hepatic Lesions ◽  
Syndrome Analysis ◽  
Visceral Larva Migrans Syndrome

scholarly journals Human case of visceral larva migrans syndrome: pulmonary and hepatic involvement

2016 ◽  
Vol 53 (4) ◽  
pp. 372-377 ◽  
Author(s):  
A. M. Almatary ◽  
H. Y. Bakir
Keyword(s):  
Toxocara Canis ◽  
Human Case ◽  
Larva Migrans ◽  
Toxocara Cati ◽  
Visceral Larva Migrans ◽  
Abdominal Ct ◽  
Hepatic Involvement ◽  
First Case ◽  
Host Tissues ◽  
Visceral Larva Migrans Syndrome

SummaryVisceral Larva Migrans (VLM) syndrome is commonly caused by larvae of roundwormsToxocara canisorToxocara cati. Human toxocarosis is a soil-transmitted zoonosis, which may result in partial or general pathological changes in host tissues. We reported a case of 14-year-old boy presented with severe dry cough without dyspnea, mild chest and abdominal pain with general fatigue. Examination of peripheral blood showed marked increase in eosinophils. The chest radiography showed an infiltrative shadow in the lung fields. Chest CT demonstrated multiple opacities in both lungs. Abdominal CT showed multiple low attenuation areas in the liver. Ultrasound guided liver biopsy revealed granulomas with severe eosinophilic infiltration. The boy was treated with albendazole and responded radically. It is worth mentioning that this is the first case of hepato-pulmonary VLM syndrome in Egypt.

VISCERAL LARVA MIGRANS SYNDROME: CLINICAL CHARACTERISTICS AND IMMUNOLOGIC STUDIES IN 51 PATIENTS

PEDIATRICS ◽  
1965 ◽  
Vol 36 (4) ◽  
pp. 523-536
Author(s):  
Carolyn C. Huntley ◽  
Margarita C. Costas ◽  
Anne Lyerly
Keyword(s):  
Patient Group ◽  
Clinical Characteristics ◽  
Larva Migrans ◽  
Visceral Larva Migrans ◽  
Physical Finding ◽  
Cell Agglutination ◽  
Heat Stable ◽  
Blood Group Substances ◽  
B Blood Group ◽  
Visceral Larva Migrans Syndrome

Fifty-one preschool children with hypereosinophilia and signs of visceral involvement were studied. The most common symptoms were pica (90%), pulmonary symptoms (86%), and fever (80%). Fourteen patients (28%) had convulsions. Hepatomegaly (encountered in 65%) was the most prominent physical finding. Anemia was present in almost half of the patients. Albumin concentrations were decreased and globulin concentrations increased in the patient group. Total gamma-globulins and gamma1 M globulins were significantly elevated. Precipitating antibody to A and B blood group substances and/or helminth antigens was found in one-half of the patients. Isohemagglutinin titers were elevated (above 1:256) in two-thirds of the patients. Sheep cell agglutination titers were slightly elevated in the patient group, and heat stable antiglobulins were found in 14 of 29 patients tested. The significance of the hypergammaglobulinemia, anemia, and central nervous system symptoms was discussed.

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