An autopsy case of frontotemporal lobar degeneration with the appearance of fused in sarcoma inclusions (basophilic inclusion body disease) clinically presenting corticobasal syndrome

2015 ◽  
Vol 36 (1) ◽  
pp. 77-87 ◽  
Author(s):  
Arifumi Matsumoto ◽  
Hiroyoshi Suzuki ◽  
Reiko Fukatsu ◽  
Hiroshi Shimizu ◽  
Yasushi Suzuki ◽  
...  
Keyword(s):  
Inclusion Body ◽  
Frontotemporal Lobar Degeneration ◽  
Corticobasal Syndrome ◽  
Autopsy Case ◽  
Fused In Sarcoma ◽  
Inclusion Body Disease ◽  
Basophilic Inclusion

scholarly journals FUS pathology in basophilic inclusion body disease

2009 ◽  
Vol 118 (5) ◽  
pp. 617-627 ◽  
Author(s):  
David G. Munoz ◽  
Manuela Neumann ◽  
Hirofumi Kusaka ◽  
Osamu Yokota ◽  
Kenji Ishihara ◽  
...  
Keyword(s):  
Inclusion Body ◽  
Inclusion Body Disease ◽  
Basophilic Inclusion

scholarly journals Basophilic inclusion body disease with novel ubiquitinated extracellular, cytoplasmic, and intranuclear inclusions

2007 ◽  
Vol 21 (5) ◽  
Author(s):  
Eileen H Bigio ◽  
Anna Engberg ◽  
Manjari Mishra ◽  
Nancy Johnson ◽  
Marsel Mesulam
Keyword(s):  
Inclusion Body ◽  
Intranuclear Inclusions ◽  
Inclusion Body Disease ◽  
Basophilic Inclusion

Optineurin is co-localized with FUS in basophilic inclusions of ALS with FUS mutation and in basophilic inclusion body disease

2011 ◽  
Vol 121 (4) ◽  
pp. 555-557 ◽  
Author(s):  
Hidefumi Ito ◽  
Kengo Fujita ◽  
Masataka Nakamura ◽  
Reika Wate ◽  
Satoshi Kaneko ◽  
...  
Keyword(s):  
Inclusion Body ◽  
Inclusion Body Disease ◽  
Basophilic Inclusion

Basophilic inclusion body disease and neuronal intermediate filament inclusion disease: a comparative clinicopathological study

2007 ◽  
Vol 115 (5) ◽  
pp. 561-575 ◽  
Author(s):  
Osamu Yokota ◽  
Kuniaki Tsuchiya ◽  
Seishi Terada ◽  
Hideki Ishizu ◽  
Hirotake Uchikado ◽  
...  
Keyword(s):  
Inclusion Body ◽  
Intermediate Filament ◽  
Clinicopathological Study ◽  
Inclusion Body Disease ◽  
Basophilic Inclusion

Mumps Virus Infection of the Pregnant Hamster

Development ◽  
1963 ◽  
Vol 11 (4) ◽  
pp. 659-665
Author(s):  
Vergil H. Ferm ◽  
Lawrence Kilham
Keyword(s):  
Virus Infection ◽  
Human Milk ◽  
Inclusion Body ◽  
Congenital Malformations ◽  
Post Partum ◽  
Mumps Virus ◽  
Human Pregnancy ◽  
Effect Relation ◽  
Inclusion Body Disease ◽  
Selection Of

The objective of our present studies has been to follow the course of mumps virus when injected intravenously into pregnant hamsters during early stages of gestation, in order to determine possible relations to fetal disease and/or malformations. Several considerations prompted the selection of mumps virus for these investigations. One was that, while rubella (Gregg, 1941) and cytomegalic inclusion body disease (Weller & Hanshaw, 1962) have been the only two viruses shown to have a definite cause-effect relation in the etiology of human congenital malformations, there has been a continuing suspicion that mumps virus may also act as a teratogenic agent in human pregnancy (Kaye & Reaney, 1962; Blattner & Heys, 1961; Hyatt, 1961). A second reason was that mumps virus has a natural pathogenicity for hamsters (Kilham & Overman, 1953). In addition, this agent is capable of infecting women at term, the strain used in present experiments having been obtained from human milk a few days post-partum (Kilham, 1951).

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