Cramp Training Induces a Long-Lasting Increase of the Cramp Threshold Frequency in Healthy Subjects

2016 ◽  
Vol 21 (8) ◽  
pp. 809-814 ◽  
Author(s):  
Michael Behringer ◽  
Volker Spieth ◽  
Johannes Caspar Konrad Montag ◽  
Steffen Willwacher ◽  
Molly Leigh McCourt ◽  
...  
Keyword(s):  
Healthy Subjects ◽  
Threshold Frequency ◽  
Cramp Threshold

scholarly journals Are electrically induced muscle cramps able to increase the cramp threshold frequency, when induced once a week?

2015 ◽  
Vol 7 (3) ◽  
Author(s):  
Michael Behringer ◽  
Tobias Walter Link ◽  
Johannes Caspar Konrad Montag ◽  
Molly Leigh McCourt ◽  
Joachim Mester
Keyword(s):  
Creatine Kinase ◽  
Muscle Damage ◽  
Intervention Group ◽  
Term Effect ◽  
Short Term ◽  
Muscle Cramps ◽  
Threshold Frequency ◽  
Single Bout ◽  
The Individual ◽  
Cramp Threshold

The cramp threshold frequency (CTF) is known to be positively correlated with the individual cramp susceptibility. Here we assessed CTF changes after two bouts of electrically induced muscle cramps (EIMCs). The EIMCs (6×5 sec) were unilaterally induced twice (separated by one week) in the gastrocnemius of an intervention group (n=8), while 5 participants served as control. The CTF increased from 25.1±4.6 Hz at baseline to 31.4±9.0 Hz and 31.7±8.5 Hz 24 h after bout 1 and 2 (P<0.05). Thereafter, the CTF declined following both bouts to reach values of 28.0±6.7 Hz and 29.1±7.7 Hz after 72 h after bout 1 and 2. Creatine kinase (CK) activity and perceived discomfort during cramps was lower after bout 2 (P<0.05). CTF, CK, and discomfort did not change in CG. That is, a single bout of EIMCs induces a 24 h CTF increment and a second bout sustains this effect, while perceived discomfort and muscle damage decreases. This short term effect may help athletes to reduce the cramp susceptibility for an important match.

scholarly journals Initial Electrical Stimulation Frequency and Cramp Threshold Frequency and Force

2012 ◽  
Vol 47 (6) ◽  
pp. 643-647 ◽  
Author(s):  
Kevin C. Miller ◽  
Kenneth L. Knight
Keyword(s):  
Electrical Stimulation ◽  
Outcome Measure ◽  
Tibial Nerve ◽  
Stimulation Frequency ◽  
Maximal Voluntary Isometric Contraction ◽  
Main Outcome Measure ◽  
Initial Frequency ◽  
Threshold Frequency ◽  
Cramp Threshold ◽  
Over Time

Context In the electrically induced cramp model, the tibial nerve is stimulated at an initial frequency of 4 Hz with increases in 2-Hz increments until the flexor hallucis brevis cramps. The frequency at which cramping occurs (ie, threshold frequency [TF]) can vary considerably. A potential limitation is that multiple subthreshold stimulations before TF might induce fatigue, which is operationally defined as a decrease in maximal voluntary isometric contraction (MVIC) force, thereby biasing TF. Objective To determine if TF is similar when initially stimulated at 4 Hz or 14 Hz and if MVIC force is different among stimulation frequencies or over time (precramp, 1 minute postcramp, and 5 minutes postcramp). Design Crossover study. Setting Laboratory. Patients or Other Participants Twenty participants (13 males: age = 20.6 ± 2.9 years, height = 184.4 ± 5.7 cm, mass = 76.3 ± 7.1 kg; 7 females: age = 20.4 ± 3.5 years, height = 166.6 ± 6.0 cm, mass = 62.4 ± 10.0 kg) who were prone to cramps. Intervention(s) Participants performed 20 practice MVICs. After a 5-minute rest, three 2-second MVICs were recorded and averaged for the precramp measurement. Participants were stimulated at either 4 Hz or 14 Hz, and the frequency was increased in 2-Hz increments from each initial frequency until cramp. The MVIC force was reevaluated at 1 minute and 5 minutes postcramp. Main Outcome Measure(s) The TF and MVIC force. Results Initial stimulation frequency did not affect TF (4 Hz = 16.2 ± 3.8 Hz, 14 Hz = 17.1 ± 5.0 Hz; t19=1.2, P = .24). Two participants had inaccurate TFs when initially stimulated at 14 Hz; they cramped at 10 and 12 Hz in the 4-Hz condition. The MVIC force did not differ between initial frequencies (F1,19 = 0.9, P = .36) but did differ over time (F2,38 = 5.1, P = .01). Force was lower at 1 minute postcramp (25.1 ± 10.1 N) than at precramp (28.7 ± 7.8 N; P < .05) but returned to baseline at 5 minutes postcramp (26.7 ± 8.9 N; P > .05). Conclusions The preferred initial stimulation frequency might be 4 Hz because it did not alter or overestimate TF. The MVIC force was lower at 1 minute postcramp, suggesting the induced cramp rather than the varying electrical frequencies affected force. A 1- to 5-minute rest should be provided postcramp induction if multiple cramps are induced.

Significant and serious dehydration does not affect skeletal muscle cramp threshold frequency

2012 ◽  
Vol 47 (11) ◽  
pp. 710-714 ◽  
Author(s):  
Kyle W Braulick ◽  
Kevin C Miller ◽  
Jay M Albrecht ◽  
Jared M Tucker ◽  
James E Deal
Keyword(s):  
Skeletal Muscle ◽  
Muscle Cramp ◽  
Threshold Frequency ◽  
Cramp Threshold

scholarly journals Acute Passive Static Stretching and Cramp Threshold Frequency

2017 ◽  
Vol 52 (10) ◽  
pp. 918-924 ◽  
Author(s):  
Gino Panza ◽  
Justin Stadler ◽  
Donal Murray ◽  
Nicholas Lerma ◽  
Tomas Barrett ◽  
...  
Keyword(s):  
Outcome Measure ◽  
Clinical Problem ◽  
Static Stretching ◽  
Static Stretch ◽  
Muscle Cramps ◽  
Threshold Frequency ◽  
Independent Variable ◽  
Exercise Associated Muscle Cramps ◽  
Cramp Threshold ◽  
Common Clinical Problem

Context:  Exercise-associated muscle cramps are a common clinical problem for athletes. Objective:  To determine whether acute passive static stretching altered cramp threshold frequency (CTF) of electrically induced muscle cramps. Design:  Crossover study. Setting:  Laboratory. Patients or Other Participants:  Seventeen healthy college-aged individuals. Intervention(s):  Stretching or no stretching. Main Outcome Measure(s):  The independent variable was the static stretch versus the no-stretch condition, and the dependent variable was the CTF. Results:  The CTF increased in both the control (pretest: 18.12 ± 6.46 Hz, posttest: 19.65 ± 7.25 Hz; P = .033) and stretching (pretest: 18.94 ± 5.96 Hz, posttest: 20.47 ± 7.12 Hz; P = .049) groups. No difference between the groups was found (t15 = 0.035, P = .97). Conclusions:  Acute passive static stretching did not seem to increase the CTF.

ELECTRICALLY INDUCED MUSCLE CRAMP THRESHOLD FREQUENCY INCREASES FOLLOWING LOCAL MUSCLE FATIGUE.

2003 ◽  
Vol 35 (Supplement 1) ◽  
pp. S282
Author(s):  
M B. Stone ◽  
J E. Edwards ◽  
J P. Babington ◽  
C D. Ingersoll ◽  
M L. Cordova
Keyword(s):  
Muscle Fatigue ◽  
Muscle Cramp ◽  
Threshold Frequency ◽  
Cramp Threshold ◽  
Local Muscle Fatigue

The Effects of Acute Passive Static Stretching on Cramp Threshold Frequency

2014 ◽  
Vol 46 ◽  
pp. 194
Author(s):  
Gino S. Panza ◽  
Justin Stadler ◽  
Donal Murray ◽  
Nicholas Lerma ◽  
Tomas Barret ◽  
...  
Keyword(s):  
Static Stretching ◽  
Threshold Frequency ◽  
Cramp Threshold

Investigation of Muscle Cramps in Patients With Cirrhosis Using Electrical Stimulation Cramp Threshold Frequency

2018 ◽  
Vol 113 (Supplement) ◽  
pp. S577-S579
Author(s):  
Rishi Bolla ◽  
Cynthia Bodkin ◽  
Marwan Ghabril ◽  
Naga Chalasani ◽  
Raj Vuppalanchi
Keyword(s):  
Electrical Stimulation ◽  
Muscle Cramps ◽  
Threshold Frequency ◽  
Cramp Threshold

Reflex Responses in Upper Limb Muscles to Cutaneous Stimuli

1993 ◽  
Vol 20 (04) ◽  
pp. 271-278 ◽  
Author(s):  
R. Chen
Keyword(s):  
Stimulus Intensity ◽  
Upper Limb ◽  
Healthy Subjects ◽  
Voluntary Contraction ◽  
Cutaneous Reflexes ◽  
Focal Lesions ◽  
Normal Ranges ◽  
Conduction Velocities ◽  
Limb Muscles ◽  
Biphasic Responses

ABSTRACT:Cutaneous reflexes in the upper limb were elicited by stimulating digital nerves and recorded by averaging rectified EMG from proximal and distal upper limb muscles during voluntary contraction. Distal muscles often showed a triphasic response: an inhibition with onset about 50 ms (Il) followed by a facilitation with onset about 60 ms (E2) followed by another inhibition with onset about 80 ms (12). Proximal muscles generally showed biphasic responses beginning with facilitation or inhibition with onset at about 40 ms. Normal ranges for the amplitude of these components were established from recordings on 22 arms of 11 healthy subjects. An attempt was made to determine the alterent fibers responsible for the various components by varying the stimulus intensity, by causing ischemic block of larger fibers and by estimating the afferent conduction velocities. The central pathways mediating these reflexes were examined by estimating central delays and by studying patients with focal lesions

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