Framingham-Based Cardiovascular Risk Estimates Among People With Episodic Migraine in the US Population: Results from the American Migraine Prevalence and Prevention (AMPP) Study

2017 ◽  
Vol 57 (10) ◽  
pp. 1507-1521 ◽  
Author(s):  
Richard B. Lipton ◽  
Michael L. Reed ◽  
Tobias Kurth ◽  
Kristina M. Fanning ◽  
Dawn C. Buse
2013 ◽  
Vol 53 (8) ◽  
pp. 1300-1311 ◽  
Author(s):  
Richard B. Lipton ◽  
Dawn C. Buse ◽  
Daniel Serrano ◽  
Starr Holland ◽  
Michael L. Reed

2011 ◽  
Vol 52 (2) ◽  
pp. 213-223 ◽  
Author(s):  
Min Kyung Chu ◽  
Dawn C. Buse ◽  
Marcelo E. Bigal ◽  
Daniel Serrano ◽  
Richard B. Lipton

2016 ◽  
Vol 2016 ◽  
pp. 1-13 ◽  
Author(s):  
Hamid Ferdosi ◽  
Elisabeth K. Dissen ◽  
Nana Ama Afari-Dwamena ◽  
Ji Li ◽  
Rusan Chen ◽  
...  

Background.To examine whether the US EPA (2010) lung cancer risk estimate derived from the high arsenic exposures (10–934 µg/L) in southwest Taiwan accurately predicts the US experience from low arsenic exposures (3–59 µg/L).Methods. Analyses have been limited to US counties solely dependent on underground sources for their drinking water supply with median arsenic levels of ≥3 µg/L.Results. Cancer risks (slopes) were found to be indistinguishable from zero for males and females. The addition of arsenic level did not significantly increase the explanatory power of the models. Stratified, or categorical, analysis yielded relative risks that hover about 1.00. The unit risk estimates were nonpositive and not significantly different from zero, and the maximum (95% UCL) unit risk estimates for lung cancer were lower than those in US EPA (2010).Conclusions. These data do not demonstrate an increased risk of lung cancer associated with median drinking water arsenic levels in the range of 3–59 µg/L. The upper-bound estimates of the risks are lower than the risks predicted from the SW Taiwan data and do not support those predictions. These results are consistent with a recent metaregression that indicated no increased lung cancer risk for arsenic exposures below 100–150 µg/L.


US Neurology ◽  
2009 ◽  
Vol 05 (01) ◽  
pp. 72
Author(s):  
Susan L Hutchinson ◽  
Stewart J Tepper ◽  
◽  

The road ahead for migraine treatment depends on targeting pharmacological therapies to known pathophysiological mechananisms. Acute medications of the future may include calcitonin gene-related peptide antagonists, various glutamate antagonists, and serotonin 1F receptor agonists. New formulations of older medications include effervescent diclofenac, an orally inhaled dihydroergotamine, and sumatriptan iontophoretic patch and needless injection, all either approved in the US or in phase III trials. Preventive future treatments may include the use of botulinum neurotoxin type A for chronic daily headache and gabapentin enacarbil for episodic migraine, in addition to hormonal treatments and a potential for neuromodulators, but the preventive pipeline is far less developed. In the US, the United Council for Neurologic Subspecialty Board certification for headache medicine should raise quality of care and research in the future.


2005 ◽  
Vol 37 (4) ◽  
pp. 1868-1870 ◽  
Author(s):  
B.K. Krämer ◽  
C. Böger ◽  
B. Krüger ◽  
J. Marienhagen ◽  
M. Pietrzyk ◽  
...  

Global Heart ◽  
2014 ◽  
Vol 9 (1) ◽  
pp. e112
Author(s):  
Abiodun M. Adeoye ◽  
Mirabel Nwosu ◽  
Adewole Adebiyi ◽  
Olulola Oladapo ◽  
Ayodele Falase

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