scholarly journals Hydrogen Sulfide Promotes Surface Insertion of Hippocampal AMPA Receptor GluR1 Subunit via Phosphorylating at Serine-831/Serine-845 Sites Through a Sulfhydration-Dependent Mechanism

2016 ◽  
Vol 22 (9) ◽  
pp. 789-798 ◽  
Author(s):  
Yuan-Long Li ◽  
Jun Zhou ◽  
Hai Zhang ◽  
Yi Luo ◽  
Li-Hong Long ◽  
...  
Keyword(s):  
Hydrogen Sulfide ◽  
Ampa Receptor ◽  
Glur1 Subunit ◽  
Dependent Mechanism

β 3 adrenergic receptor activation relaxes human corpus cavernosum and penile artery through a hydrogen sulfide/cGMP-dependent mechanism

2017 ◽  
Vol 124 ◽  
pp. 100-104 ◽  
Author(s):  
Emma Mitidieri ◽  
Teresa Tramontano ◽  
Danila Gurgone ◽  
Ciro Imbimbo ◽  
Vincenzo Mirone ◽  
...  
Keyword(s):  
Hydrogen Sulfide ◽  
Adrenergic Receptor ◽  
Corpus Cavernosum ◽  
Receptor Activation ◽  
Penile Artery ◽  
Dependent Mechanism

scholarly journals Hydrogen Sulfide Improves Survival After Cardiac Arrest and Cardiopulmonary Resuscitation via a Nitric Oxide Synthase 3–Dependent Mechanism in Mice

Circulation ◽  
2009 ◽  
Vol 120 (10) ◽  
pp. 888-896 ◽  
Author(s):  
Shizuka Minamishima ◽  
Masahiko Bougaki ◽  
Patrick Y. Sips ◽  
Jia De Yu ◽  
Yoji Andrew Minamishima ◽  
...  
Keyword(s):  
Nitric Oxide ◽  
Cardiac Arrest ◽  
Hydrogen Sulfide ◽  
Nitric Oxide Synthase ◽  
Cardiopulmonary Resuscitation ◽  
Nitric Oxide Synthase 3 ◽  
Oxide Synthase ◽  
Dependent Mechanism

scholarly journals Modulation of AMPA receptor GluR1 subunit phosphorylation in neurons by the intravenous anaesthetic propofol

2008 ◽  
Vol 100 (5) ◽  
pp. 676-682 ◽  
Author(s):  
M. Haines ◽  
L.M. Mao ◽  
L. Yang ◽  
A. Arora ◽  
E.E. Fibuch ◽  
...  
Keyword(s):  
Ampa Receptor ◽  
Intravenous Anaesthetic ◽  
Glur1 Subunit

REGULATION OF AMPA RECEPTOR SURFACE EXPRESSION BY A NSF-DEPENDENT MECHANISM IN HIPPOCAMPAL NEURONS IN CULTURE

1999 ◽  
Vol 27 (3) ◽  
pp. A117-A117
Author(s):  
J. Noel ◽  
G. Scott Ralph ◽  
L. Pickard ◽  
E. Molnar ◽  
J. B. Uney ◽  
...  
Keyword(s):  
Ampa Receptor ◽  
Hippocampal Neurons ◽  
Surface Expression ◽  
Receptor Surface Expression ◽  
Dependent Mechanism

Exendin-4 promotes the membrane trafficking of the AMPA receptor GluR1 subunit and ADAM10 in the mouse neocortex

2014 ◽  
Vol 190-191 ◽  
pp. 1-11 ◽  
Author(s):  
Nobuaki Ohtake ◽  
Mieko Saito ◽  
Masaaki Eto ◽  
Kenjiro Seki
Keyword(s):  
Ampa Receptor ◽  
Membrane Trafficking ◽  
Glur1 Subunit

scholarly journals Hydrogen sulfide triggers late-phase preconditioning in postischemic small intestine by an NO- and p38 MAPK-dependent mechanism

2009 ◽  
Vol 296 (3) ◽  
pp. H868-H876 ◽  
Author(s):  
Mozow Yusof ◽  
Kazuhiro Kamada ◽  
Theodore Kalogeris ◽  
F. Spencer Gaskin ◽  
Ronald J. Korthuis
Keyword(s):  
Hydrogen Sulfide ◽  
P38 Mapk ◽  
Ischemia Reperfusion ◽  
Late Phase ◽  
Mitogen Activated Protein Kinase ◽  
Sodium Hydrosulfide ◽  
Pharmacological Inhibition ◽  
Mapk Inhibitors ◽  
Dependent Mechanism

Hydrogen sulfide (H2S) is one of three endogenous gases, along with carbon monoxide (CO) and nitric oxide (NO), that exert a variety of important vascular actions in vivo. Although it has been demonstrated that CO or NO can trigger the development of a preconditioned phenotype in postischemic tissues, it is unclear whether H2S may also induce protection in organs subsequently exposed to ischemia-reperfusion (I/R). In light of these observations, we postulated that preconditioning with the exogenous H2S donor sodium hydrosulfide (NaHS-PC) would inhibit leukocyte rolling (LR) and adhesion (LA) induced by I/R. We used intravital microscopic techniques to demonstrate that NaHS-PC 24 h, but not 1 h, before I/R causes postcapillary venules to shift to an anti-inflammatory phenotype in wild-type (WT) mice such that these vessels fail to support LR and LA during reperfusion. The protective effect of NaHS-PC on LR was largely abolished by coincident pharmacological inhibition of NO synthase (NOS) in WT animals and was absent in endothelial NOS-deficient (eNOS−/−) mice. A similar pattern of response was noted in WT mice treated concomitantly with NaHS plus p38 mitogen-activated protein kinase (MAPK) inhibitors (SB 203580 or SK-86002). Whereas the reduction in LA induced by antecedent NaHS was attenuated by pharmacological inhibition of NOS or p38 MAPK in WT mice, the antiadhesive effect of NaHS was still evident in eNOS−/− mice. Thus NaHS-PC prevents LR and LA by triggering the activation of an eNOS- and p38 MAPK-dependent mechanism. However, the role of eNOS in the antiadhesive effect of NaHS-PC was less prominent than its effect to reduce LR.

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