scholarly journals Two-channel quantum wire with an adatom impurity: Role of the van Hove singularity in the quasibound state in continuum, decay rate amplification, and the Fano effect

2009 ◽  
Vol 80 (11) ◽  
Author(s):  
S. Garmon ◽  
H. Nakamura ◽  
N. Hatano ◽  
T. Petrosky
1995 ◽  
Vol 09 (09) ◽  
pp. 1067-1080 ◽  
Author(s):  
M.L. HORBACH ◽  
H. KAJÜTER

Experimental indications for the role of the van Hove singularity (vHs) in the electronic density of states of high Tc superconductors are discussed. It is argued that (i) like the resistivity, the measured temperature and doping dependences of the normal state electronic specific heat of YBCO are consistent with the existence of a vHs and (ii) the doping dependence of Tc in the underdoped and optimally doped regimes may be accounted for mainly by a vHs. Further, we discuss the suppression of the quasiparticle scattering rate below Tc, and the coherence lengths in the hole-doped materials and NCCO. Assuming that the same mechanism for superconductivity operates in the electron-doped and the hole-doped cuprates, we argue that the bosonic mode that causes the superconductivity is strongly influenced by the doping in the overdoped regime. We further argue that this boson involves an energy scale larger than that of phonons and is only weakly coupled to the charge carriers.


1991 ◽  
Vol 43 (4) ◽  
pp. 3075-3084 ◽  
Author(s):  
D. M. Newns ◽  
P. C. Pattnaik ◽  
C. C. Tsuei

1999 ◽  
Vol 201-202 ◽  
pp. 814-818 ◽  
Author(s):  
Richard Nötzel ◽  
Hans-Peter Schönherr ◽  
Zhichuan Niu ◽  
Lutz Däweritz ◽  
Klaus H Ploog

2019 ◽  
Vol 20 (19) ◽  
pp. 4868
Author(s):  
Ruo-Ciao Cheng ◽  
Pi-Cheng Cheng ◽  
Yi-Chi Wang ◽  
Rong-Chi Huang

Transmembrane Ca2+ influx is essential to the proper functioning of the central clock in the suprachiasmatic nucleus (SCN). In the rat SCN neurons, the clearance of somatic Ca2+ following depolarization-induced Ca2+ transients involves Ca2+ extrusion via Na+/Ca2+ exchanger (NCX) and mitochondrial Ca2+ buffering. Here we show an important role of intracellular Na+ in the regulation of [Ca2+]i in these neurons. The effect of Na+ loading on [Ca2+]i was determined with the Na+ ionophore monensin and the cardiac glycoside ouabain to block Na+/K+-ATPase (NKA). Ratiometric Na+ and Ca2+ imaging was used to measure the change in [Na+]i and [Ca2+]i, and cell-attached recordings to investigate the effects of monensin and ouabain on spontaneous firing. Our results show that in spite of opposite effects on spontaneous firing and basal [Ca2+], both monensin and ouabain induced Na+ loading, and increased the peak amplitude, slowed the fast decay rate, and enhanced the slow decay phase of 20 mM K+-evoked Ca2+ transients. Furthermore, both ouabain and monensin preferentially enhanced nimodipine-insensitive Ca2+ transients. Together, our results indicate that in the SCN neurons the NKA plays an important role in regulating [Ca2+]i, in particular, associated with nimodipine-insensitive Ca2+ channels.


2003 ◽  
Vol 52-54 ◽  
pp. 707-712 ◽  
Author(s):  
Yoonsuck Choe ◽  
Risto Miikkulainen

Author(s):  
J.P. Silveira ◽  
M.U. Gonzalez ◽  
J.M. Garcia ◽  
L. Gonzalez ◽  
Y. Gonzalez ◽  
...  

Sign in / Sign up

Export Citation Format

Share Document