scholarly journals Rates of Seed Treatment Insecticides and Control of Stewart's Wilt in Sweet Corn

Plant Disease ◽  
2005 ◽  
Vol 89 (3) ◽  
pp. 262-268 ◽  
Author(s):  
J. K. Pataky ◽  
P. M. Michener ◽  
N. D. Freeman ◽  
J. M. Whalen ◽  
J. A. Hawk ◽  
...  

Neonicotinoid insecticides applied as seed treatments reduce the incidence of Stewart's wilt. The objectives of this study were to examine the efficacy of different rates of seed treatment insecticides to control Stewart's wilt on susceptible sweet corn hybrids and to compare the economic value of Stewart's wilt control in sweet corn grown for processing and fresh market. Clothianidin (Poncho), imidacloprid (Gaucho), and thiamethoxam (Cruiser) applied to seed at rates ranging from 0.125 to 1.25 mg a.i. per kernel were evaluated in 11 field trials in Illinois and Delaware from 2000 to 2003. Incidence of Stewart's wilt was significantly lower when seed was treated with insecticides than when plants were grown from nontreated seed in all but one trial. The level of control usually was between 50 and 90%. Small but statistically significant differences in incidence of systemically infected plants occurred among rates of insecticides in all trials except those in 2001. Usually, incidence of systemic Stewart's wilt was lower when higher rates of insecticides were applied; however, increasing the rate of insecticides from 0.125 mg a.i. to 1.25 mg a.i. per kernel had a relatively small effect on the level of Stewart's wilt control compared with the difference between treated and nontreated sweet corn seed. Based on a regression analysis, the lowest rates of the insecticides provided 64 to 72% control. The level of control increased about 1.85% with each additional 0.1 mg a.i. of insecticide per kernel from 0.125 mg a.i. to 1.25 mg a.i. Clothianidin provided an 8 or 9% higher level of control than thiamethoxam or imidacloprid at the same rate. Recommendations for application of seed treatment insecticides to processing and fresh market sweet corn differed somewhat due to substantial differences in the value of the crops. Based on estimated costs of $6 to $12 per 0.4 ha for the seed treatments, the economic break even point (i.e., cost of control = value from control) occurred in the range of 3 to 6% Stewart's wilt incidence for processing sweet corn valued at $325 per 0.4 ha and at about 1% Stewart's wilt incidence for fresh market sweet corn valued at $1,625 per 0.4 ha. Relatively small differences in levels of control conferred by commercially available rates of clothianidin (0.25 mg a.i. per kernel) and thiamethoxam (0.125 mg a.i. per kernel) were of little consequence in processing sweet corn but had considerable economic value in fresh market sweet corn.

Plant Disease ◽  
2000 ◽  
Vol 84 (10) ◽  
pp. 1104-1108 ◽  
Author(s):  
J. K. Pataky ◽  
P. M. Michener ◽  
N. D. Freeman ◽  
R. A. Weinzierl ◽  
R. H. Teyker

Corn flea beetles, Chaetocnema pulicaria, vector Erwinia stewartii (synamorph Pantoea stewartii), which causes Stewart's bacterial wilt of corn (Zea mays). A seed treatment insecticide, imidacloprid, killed flea beetles and reduced the number of feeding wounds and Stewart's wilt symptoms per leaf in greenhouse studies. The objective of our research was to evaluate the ability of imidacloprid and thiamethoxam seed treatments to control Stewart's wilt on sweet corn hybrids under field conditions with naturally occurring populations of the corn flea beetle. Six field trials were planted at four locations in 1998. Eleven field trials were planted at nine locations in 1999. The treatment design was a factorial of sweet corn hybrids and seed treatments. Stewart's wilt incidence ranged from 0 to 54% in the 1998 trials. Incidence of Stewart's wilt in nontreated plots of the susceptible hybrid Jubilee ranged from 2% at the 8-leaf stage to 77% 1 week after mid-silk in the 1999 trials. Seed treatment insecticides reduced the incidence of Stewart's wilt by ≈50 to 85% relative to nontreated controls. The level of control was ≈75 to 85% in seven trials planted before 1 June 1999, when incidence of Stewart's wilt on nontreated Jubilee ranged from 4 to 71%. The level of control was ≈50 to 70% in the three trials planted after 1 July 1999, when incidence of Stewart's wilt on nontreated Jubilee ranged from 44 to 73%. Although comparisons varied, the level of control gained from seed treatment insecticides was similar to the next higher level of host resistance. Seed treatment insecticides appear to control Stewart's wilt during very early growth of corn plants, when foliar applications of insecticides are ineffective and the effectiveness of host resistance varies depending on the proximity of flea beetle feeding sites to the plant's growing point.


Plant Disease ◽  
2010 ◽  
Vol 94 (9) ◽  
pp. 1111-1117
Author(s):  
M. D. Meyer ◽  
J. K. Pataky ◽  
D. K. Joos ◽  
R. W. Esgar ◽  
B. R. Henry

Many sweet corn (Zea mays) hybrids commercially available today have higher levels of resistance to Stewart's disease (caused by Pantoea stewartii subsp. stewartii) than the cultivars from which Stevens developed the first forecast of this disease in the 1930s. Incorporating levels of host resistance into forecasts of the seedling wilt phase of Stewart's disease (i.e., Stewart's wilt) could improve control decisions for sweet corn which are made prior to planting. Incidence of systemic infection of seedlings was assessed on 27 sweet corn hybrids with a range of reactions to P. stewartii. In total, 741 observations were collected from 1998 to 2009 in 79 field trials at 15 locations throughout Illinois and one each in Kentucky and Delaware. Relative frequency distributions of the incidence of systemic Stewart's wilt were developed for combinations of hybrids with different levels of resistance and ranges of winter temperature from Stewart's wilt forecasts. The probability of exceeding thresholds of 1 or 5% incidence that warrant the use of seed-treatment insecticides on sweet corn grown for fresh market or processing, respectively, was determined from these frequency distributions. Levels of host resistance affected the incidence of systemic seedling wilt within ranges of winter temperatures used by Stewart's wilt forecasts. For moderate and resistant hybrids, frequency distributions of Stewart's wilt incidence and mean incidence ranging from 0.7 to 1.8% did not differ among three winter temperature ranges above –2.8°C. Conversely, distributions of Stewart's wilt incidence on susceptible hybrids differed among each of the four ranges of winter temperature from the Stevens-Boewe forecast (i.e., >0.6, –1.1 to 0.6, –2.8 to –1.1, and <–2.8°C), with mean incidence ranging from 0.5 to 8.5%. Occurrence of Stewart's wilt also differed among trials varying in number of winter months above –4.4°C, the criterion used by the Iowa State forecast of this disease. Levels of host resistance to P. stewartii also affected the occurrence of Stewart's wilt as predicted by the Iowa State method. The probability of exceeding economic thresholds of 1 or 5% incidence of systemic Stewart's wilt depended on levels of host resistance and winter temperature. Stewart's wilt is unlikely to exceed economic thresholds when the mean winter temperature is below –4.4°C. When mean winter temperature was above –2.8°C, the probability of exceeding 1% incidence of systemic Stewart's wilt was 0.59 for susceptible sweet corn hybrids and 0.28 for moderate and resistant hybrids. When mean winter temperature was below –2.8°C, the probability of exceeding 1% incidence of systemic Stewart's wilt was 0.22 for susceptible hybrids and 0.04 for moderate or resistant sweet corn hybrids. The probability of exceeding 5% incidence was less than 0.1, except when the mean winter temperature was above –2.8°C and susceptible hybrids were grown.


HortScience ◽  
1994 ◽  
Vol 29 (4) ◽  
pp. 277-278 ◽  
Author(s):  
Carlos A. Parera ◽  
Daniel J. Cantliffe

Poor emergence and low seedling vigor are characteristics of many supersweet sweet corn (Zea mays L.) cultivars carrying the shrunken-2 (sh2) gene. Four sh2 sweet corn cultivar seeds [`How Sweet It Is' (HSII), `Crisp N' Sweet 711' (CNS-711), `Sweet Belle' (SB), and `Dazzle' (DZ)] were solid-matrix-primed (SMP), SMP with sodium hypochlorite (SMPcl), treated with a fungicide combination (F) (Imazalil + Captan + Apron + Thiram), or primed with the aforementioned fungicides (SMPf). The seed treatments were tested in the laboratory and the field. Seed imbibition and leachate electrical conductivity were lower in SMP seeds than in nonprimed seeds. In the field, emergence percentage and rate of CNS-711 and SB (high-vigor seeds) were not improved by the seed treatments compared to the nontreated seeds. Emergence percentage and rate of HSII and DZ (considered low-vigor seeds) were improved as a result of SMPcl, SMPf, or F treatments compared to nonprimed seeds. Compared to the F treatment, the SMPcl presowing treatment increased DZ seedling emergence rate and percentage. The combined SMP and seed disinfection via NaOCl seems to be a promising fungicide seed-treatment substitute that improves the stand establishment and seedling vigor of sh2 sweet corn cultivars. Chemical names used: 1-[2-(2,4-dichlorophenyl)-2-(2-propenyloxy)ethyl]-1 H imidazole (Imazalil); N-[(trichloromethyl)thio]-4-cyclohexene-1,2-dicarboximide(Captan); N- (2,6-dimethylphenyl)- N -(methoxyacetyl)alanine methyl ester (Apron); tetramethylthiuram disulfide (Thiram).


HortScience ◽  
1990 ◽  
Vol 25 (9) ◽  
pp. 1110f-1110
Author(s):  
Nancy W. Callan ◽  
James B. Miller ◽  
Don E. Mathre

Shrunken-2 supersweet (sh2) sweet corn is susceptible to preemergence damping-off caused by Pythium ultimum, especially when planted into cold soil. Bio-priming, a seed treatment which combines the establishment of a bioprotectant on the seed with preplant seed hydration, was developed to protect seeds from damping-off.In a series of field experiments conducted in Montana's Bitterroot and Gallatin Valleys, bio-priming or seed bacterization with Pseudomonas fluorescens AB254 protected sweet corn from P. ultimum damping-off. Bio-priming corn seed with P. fluorescens AB254 was comparable to treatment with the fungicide metalaxyl in increasing seedling emergence. Seedlings from bio-primed seeds emerged from the soil more rapidly than from nontreated seeds and were larger at three weeks postplanting. Seeds of sh 2 and sugary enhancer (se) sweet corn, as well as that of several sh 2 cultivars, were protected from damping-off by bio-priming.


2017 ◽  
Vol 32 (1) ◽  
pp. 41-49 ◽  
Author(s):  
Farag Mahmoud ◽  
Mohamed Osman ◽  
Kariman Mahmoud

Aphids are the most important pests on wheat in Egypt and worldwide. Field trials were conducted to assess the efficacy of the neonicotinoid insecticides imidacloprid (Nufidor 60% FS; at the doses of 2.1, 1.05 and 0.525 g a.i./kg of seeds) and thiamethoxam (Cruiser 70% WS; at the rates of 14.4, 7.2 and 3.6 g a.i./kg of seeds) as seed treatments, and thiamethoxam (Actara 25% WG; at the rates of 0.1, 0.05 and 0.025 g a.i./l) as foliar application, against three wheat aphids: bird cherry oat aphid, Rhopalosiphum padi (L.), greenbug, Schizaphis graminum (Rondani), and corn leaf aphid, Rhopalosiphum maidis (Fitch). Their effects on the aphids? natural enemies: lacewings, Chrysoperla carnea Stephens, ladybird beetles, Coccinella spp., and syrphid flies Syrphus spp. were assessed as well. The trials were conducted on the farm of the Faculty of Agriculture, Suez Canal University, Ismailia Governorate, during 2013/14 and 2014/15 seasons. Our results showed significant differences between the tested insecticides at all concentrations and clearly indicated that the recommended doses of all insecticides were more efficient against aphids on wheat plants than half- and quarter-doses. Also, the data revealed significant differences between the two seed treatments at all doses before and after foliar application, as compared to the control at all time intervals of inspection regarding the mean number of aphids on wheat plants, from the 3rd until 13th week after sowing, except in the 8th week after seed treatment before foliar application. The results clearly indicated that the weekly reduction of infestation and the general efficacy was higher at the recommended doses of Nufidor, Cruiser and Actara than the half and quarter doses in both seasons. Also, reduction in infestation decreased over the following weeks until the 8th week, when Actara insecticide was sprayed. Data revealed that there were no significant differences between treatments and control in the mean number of Chrysoperla carnea and Syrphus spp., while a significant difference in the mean number of coccinellids was observed on wheat plants treated with imidacloprid seed treatment before foliar application 6 and 7 weeks after sowing, and also after foliar application with thiamethoxam 9, 10, 11, 12 and 13 weeks after sowing. Regarding thiamethoxam seed treatment, significant differences were revealed in the ladybird population on wheat plants 3, 4, 7 and 8 weeks before foliar application, and only in the 10th week after spraying with thiamethoxam.


2019 ◽  
Vol 20 (3) ◽  
pp. 160-164
Author(s):  
Sudha GC Upadhaya ◽  
Venkataramana Chapara ◽  
Mukhlesur Rahman ◽  
Luis E. del Río Mendoza

The efficacy of five fungicide seed treatments as a management tool against blackleg on spring canola was evaluated under greenhouse and field conditions in North Dakota. Blackleg, caused by Leptosphaeria maculans, inflicts the greatest yield losses when infecting seedlings before they reach the six-leaf growth stage. In greenhouse studies, 10-day-old seedlings were inoculated with L. maculans spore suspensions and evaluated 12 days later and at maturity or inoculated 12, 20, or 28 days after planting and evaluated at maturity. In field trials conducted in 2017 and 2018, severity was assessed at maturity. In the greenhouse, all fungicide seed treatments reduced (P = 0.05) disease severity at the seedling stage, but only the protection provided by Obvius (fluxapyroxad + pyraclostrobin + metalaxyl) and Helix Vibrance (mefenoxam + fludioxonil + sedaxane + difenoconazole + thiamethoxam) reduced (P < 0.05) severity at the adult stage; however, none of them provide effective protection when plants were inoculated 20 days after planting or later. In field trials, none of the treatments significantly (P > 0.05) improved plant stand and yield or reduced disease incidence and severity. Although fungicide seed treatment is a valuable tool, it should not be used as the only method to manage blackleg disease.


2018 ◽  
Vol 2018 ◽  
pp. 1-7 ◽  
Author(s):  
N. R. Steppig ◽  
J. K. Norsworthy ◽  
R. C. Scott ◽  
G. M. Lorenz

With increased instances of weed resistance to applications of postemergence herbicides, the use of soil-applied herbicides that offer residual activity is becoming popular. Unfortunately, under some conditions, the use of residual herbicides can result in unintentional injury to crops. However, there are a number of ways to reduce these risks, including the use of in-crop herbicide safeners. Based on previous research conducted on rice, the potential may exist for crop injury from certain soil-applied herbicides to be reduced (safened) in seeds treated with insecticides. Field trials were conducted in Marianna, Arkansas, in 2015 and 2016, and near Colt, Arkansas, in 2016, to explore this possibility in soybean. Soybean seeds were treated with the insecticide thiamethoxam and subsequently the herbicides metribuzin, saflufenacil, pyroxasulfone, sulfentrazone, chlorimuron, flumioxazin, flumioxazin + pyroxasulfone + chlorimuron, mesotrione, and chlorsulfuron were applied immediately after planting. Of the nine herbicides evaluated, the insecticide reduced crop injury for flumioxazin, chlorsulfuron, saflufenacil, pyroxasulfone, and flumioxazin + pyroxasulfone + chlorimuron. The highest degree of injury reduction was seen 1 week after emergence (WAE) at Marianna, where injury from flumioxazin + pyroxasulfone + chlorimuron was reduced from 15% to 5%. Based on the results from this study, the insecticide seed treatment thiamethoxam may have the potential to safen soybean to applications of some soil-applied herbicides.


2007 ◽  
Vol 87 (1) ◽  
pp. 167-174 ◽  
Author(s):  
A. G. Xue ◽  
E. Cober ◽  
M. J. Morrison ◽  
H. D. Voldeng ◽  
B. L. Ma

Field trials were conducted with soybean at two sites each year from 2001 to 2003 in Ottawa, ON, to determine the effect of seed treatments with various combinations of seven formulated fungicides and the bioagent Yield Shield (Bacillus pumilus GB34) under Rhizoctonia solani inoculated conditions. Controls were untreated seed planted into both non-inoculated (natural) soil and soil inoculated with R. solani. Compared with the non-inoculated control, inoculation significantly increased root rot severity and reduced emergence by 27%, and yield by 31%. Under the inoculated conditions, none of the seed treatments significantly increased emergence or yield in all of the six trials when compared with the control. Allegiance (metalaxyl) plus Vitaflo-280 (carbathiin plus thiram) and Vitaflo-280 alone were the most effective seed treatments, increasing emergence in by 20 and 19% and yield by 21 and 26%, which were significantly better than the control in four and five trials for emergence and three and four trials for yield, respectively. Allegiance plus HEC5725 (HEC5725), Apron Maxx RTA (fludioxonil plus metalaxyl), and Maxim 480FS (fludioxonil) increased both emergence and yield in two trials and TFL RTU (metalaxyl plus triflox ystrobin) plus Yield Shield in one trial. There was no difference between seed treatment with Allegiance and the untreated control for all parameters, confirming that metalaxyl is ineffective to R. solani. It is concluded that carbathiin, thiram, HEC5725, fludioxonil an trifloxystrobin are effective active ingredients protecting soybean from soil-borne R. solani and increasing plant emergence and yield. Key words: Rhizoctonia solani, seed treatment, soybean, Glycine max, fungicide, bioagent


2002 ◽  
Vol 21 (1) ◽  
pp. 25-31 ◽  
Author(s):  
Thomas P Kuhar ◽  
Lydia J Stivers-Young ◽  
Michael P Hoffmann ◽  
Alan G Taylor

2017 ◽  
Vol 68 (6) ◽  
pp. 527 ◽  
Author(s):  
Sarah Mansfield ◽  
Richard J. Chynoweth ◽  
Mark R. H. Hurst ◽  
Alasdair Noble ◽  
Sue M. Zydenbos ◽  
...  

Insecticidal seed treatments are used commonly worldwide to protect seedlings against root feeding insects. Organophosphate insecticides that have been used for seed treatments are being phased out and replaced with neonicotinoid insecticides. Concerns about the environmental impact of neonicotinoids have prompted a search for alternatives. Microbial insecticides are a biological alternative for seed treatments to target root feeding insects. Six field trials with organophosphate granules (diazinon, chlorpyrifos), neonicotinoid seed treatment (clothianidin) and microbial (Serratia entomophila) seed treatment targeting grass grub, a New Zealand scarab pest, were conducted in wheat crops at several sites over 4 years (2012–2015). Sites were selected each year that had potentially damaging populations of grass grub present during the trials. Untreated seeds led to significant losses of plants and wheat yield due to lower seedling establishment and ongoing plant losses from grass grub damage. Insecticide and microbial treatments increased plant survival in all trials compared with untreated seeds. Better plant survival was associated with higher yields from the insecticide treatments in four out of six trials. Neonicotinoid seed treatment alone gave similar yield increases to combined neonicotinoid seed treatment and organophosphate granules. Microbial seed treatment with S. entomophila gave similar yield increases to insecticide treatments in two out of six trials. Seed treatment with S. entomophila is an alternative for grass grub control; however, development of a commercial product requires effective scale-up of production, further research to improve efficacy, and viability of the live bacteria needs to be maintained on coated seed.


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