scholarly journals Apical Wilting and Petiole Xylem Vessel Diameter of the rms2 Branching Mutant of Pea are Shoot Controlled and Independent of a Long-Distance Signal Regulating Branching

2008 ◽  
Vol 49 (5) ◽  
pp. 791-800 ◽  
Author(s):  
I. C. Dodd ◽  
B. J. Ferguson ◽  
C. A. Beveridge
2016 ◽  
Vol 38 (1) ◽  
pp. 19-24 ◽  
Author(s):  
Robin K. Cameron ◽  
Philip Carella ◽  
Marisa Isaacs ◽  
Marc Champigny ◽  
Juliane Merl-Pham ◽  
...  

2006 ◽  
pp. 333-349
Author(s):  
Stefano Mancuso ◽  
Sergio Mugnai

2020 ◽  
Vol 90 (3) ◽  
Author(s):  
Mark Olson ◽  
Julieta A. Rosell ◽  
Cecilia Martínez‐Pérez ◽  
Calixto León‐Gómez ◽  
Alex Fajardo ◽  
...  

2012 ◽  
Vol 52 (1) ◽  
pp. 139-145 ◽  
Author(s):  
Mohamed El-Nady ◽  
Amany Hamza ◽  
Aly Derbalah

Echinochloa ColonumResistance to Bispyribac-Soduim in Egypt - Occurrence and IdentificationIdentification and mechanism ofEchinochloa colonum(L.) resistance to bispyribac-soduim via physiological and anatomical differences between susceptible and resistant biotypes was investigated. The physiological and anatomical differences that were take into account were growth reduction, chlorophyll content reduction, protein analysis, lamina thickness and xylem vessel diameter in both susceptible and resistant biotypes ofE. colonum.The results showed the growth reduction fifty (GR50) of resistant biotype was 10.2 times higher than that of the susceptible biotypeE. colonumtreated with bispyribac-soduim. The chlorophyll content was highly reduced in the susceptible biotype relative to the resistant one ofE. colonumtreated with bispyribac-soduim. An anatomical test showed significant differences in the cytology of susceptible and resistant biotypes ofE. colonumtreated with bispyribac-soduim with respect to lamina thickness and xylem vessel diameter. Furthermore, leaf protein analysis showed significant differences between the susceptible and resistant biotypes ofE. colonumin the number and the density of protein bands. The resistance ofE. colonumto bispyribac-soduim may be due to the faster metabolism of bispyribac-soduim below the physiologically active concentration or the insensitivity of its target enzyme, (acetolactate synthase). These results implied the occurrence ofE. colonumresistance to bispyribac-soduim in Egypt and provide conclusive evidence that a single resistance mechanism alone cannot explain insensitivity inE. colonumto bispyribac-soduim.


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