scholarly journals Erratum to: Striatal and cerebellar vesicular acetylcholine transporter expression is disrupted in human DYT1 dystonia

Brain ◽  
2021 ◽  
Author(s):  
Joachim Mazere ◽  
Bixente Dilharreguy ◽  
Gwenaëlle Catheline ◽  
Marie Vidailhet ◽  
Marc Deffains ◽  
...  
Keyword(s):  
Vesicular Acetylcholine Transporter ◽  
Dyt1 Dystonia ◽  
Transporter Expression

scholarly journals Normal Striatal Vesicular Acetylcholine Transporter Expression in Tourette Syndrome

eNeuro ◽  
2017 ◽  
Vol 4 (4) ◽  
pp. ENEURO.0178-17.2017 ◽  
Author(s):  
Roger L. Albin ◽  
Christine Minderovic ◽  
Robert A. Koeppe
Keyword(s):  
Tourette Syndrome ◽  
Vesicular Acetylcholine Transporter ◽  
Transporter Expression

scholarly journals Vesicular Acetylcholine Transporter Alters Cholinergic Tone and Synaptic Plasticity in DYT1 Dystonia

2021 ◽  
Author(s):  
Annalisa Tassone ◽  
Giuseppina Martella ◽  
Maria Meringolo ◽  
Valentina Vanni ◽  
Giuseppe Sciamanna ◽  
...  
Keyword(s):  
Synaptic Plasticity ◽  
Vesicular Acetylcholine Transporter ◽  
Dyt1 Dystonia ◽  
Cholinergic Tone

LXR und ABC-Transporter-Expression im Trophoblasten bei IUGR

2017 ◽  
Vol 77 (04) ◽  
pp. 379-395
Author(s):  
L Schmieding ◽  
A Klein ◽  
N Maass ◽  
C Eckmann-Scholz ◽  
D Lütjohann ◽  
...  
Keyword(s):  
Abc Transporter ◽  
Transporter Expression

Effect of Neuroinflammation on ABC Transporters: Possible Contribution to Refractory Epilepsy

2018 ◽  
Vol 17 (10) ◽  
pp. 728-735 ◽  
Author(s):  
Xiaolin Deng ◽  
Yangmei Xie ◽  
Yinghui Chen
Keyword(s):  
Antiepileptic Drugs ◽  
Abc Transporters ◽  
Refractory Epilepsy ◽  
Vascular Endothelial Cells ◽  
Efflux Transporters ◽  
Vascular Endothelial ◽  
Intracellular Signalling Pathways ◽  
The Brain ◽  
Transporter Expression

Background & Objective: Epilepsy is a common and serious chronic neurological disorder that is mainly treated with antiepileptic drugs. Although current antiepileptic drugs used in clinical practice have advanced to the third generation, approximately one-third of patients are refractory to these treatments. More efficacious treatments for refractory epilepsy are therefore needed. A better understanding of the mechanism underlying refractory epilepsy is likely to facilitate the development of a more effective therapy. The abnormal expression and/or dysfunction of efflux transporters, particularly ABC transporters, might contribute to certain cases of refractory epilepsy. Inflammation in the brain has recently been shown to regulate the expression and/or function of ABC transporters in the cerebral vascular endothelial cells and glia of the blood-brain barrier by activating intracellular signalling pathways. Conclusion: Therefore, in this review, we will briefly summarize recent research advances regarding the possible role of neuroinflammation in regulating ABC transporter expression in epilepsy.

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