High Frequency of Gross Deletions in 5′ LTR/gagandnefGenes in Patients Infected with CRF02_AG of HIV Type 1 Who Survived for Over 20 Years: An Association with Korean Red Ginseng

2009 ◽  
Vol 25 (5) ◽  
pp. 535-541 ◽  
Author(s):  
Young-Keol Cho ◽  
You-Sun Jung ◽  
Heungsup Sung ◽  
Mi-Kyung Sim ◽  
Yoo-Kyum Kim
Keyword(s):  
High Frequency ◽  
Red Ginseng ◽  
Korean Red Ginseng ◽  
Hiv Type 1

Korean red ginseng (Panax ginseng) ameliorates type 1 diabetes and restores immune cell compartments

2012 ◽  
Vol 144 (2) ◽  
pp. 225-233 ◽  
Author(s):  
Young Joo Hong ◽  
Nayoung Kim ◽  
Karim Lee ◽  
Chung Hee Sonn ◽  
Jung Eun Lee ◽  
...  
Keyword(s):  
Type 1 Diabetes ◽  
Panax Ginseng ◽  
Immune Cell ◽  
Red Ginseng ◽  
Korean Red Ginseng ◽  
Cell Compartments

High Frequency of DefectivevpuCompared withtatandrevGenes in Brain from Patients with HIV Type 1-Associated Dementia

2007 ◽  
Vol 23 (4) ◽  
pp. 575-580 ◽  
Author(s):  
Elaine R. Thomas ◽  
Rebecca L. Dunfee ◽  
Jennifer Stanton ◽  
Derek Bogdan ◽  
Kevin Kunstman ◽  
...  
Keyword(s):  
High Frequency ◽  
Hiv Type 1

scholarly journals High Frequency of Recombinant Genomes in HIV Type 1 Samples from Brazilian Southeastern and Southern Regions

2002 ◽  
Vol 18 (17) ◽  
pp. 1261-1269 ◽  
Author(s):  
Monick Lindenmeyer Guimarães ◽  
Aline dos Santos Moreira ◽  
Regina Loureiro ◽  
Bernardo Galvão-Castro ◽  
Mariza Gonçalves Morgado
Keyword(s):  
High Frequency ◽  
Hiv Type 1

scholarly journals The Efficacy of Red Ginseng in Type 1 and Type 2 Diabetes in Animals

2013 ◽  
Vol 2013 ◽  
pp. 1-7 ◽  
Author(s):  
Bin Na Hong ◽  
Min Gun Ji ◽  
Tong Ho Kang
Keyword(s):  
Type 2 Diabetes ◽  
Signal Intensity ◽  
Diabetic Mice ◽  
Red Ginseng ◽  
Korean Red Ginseng ◽  
Diabetes In Animals ◽  
Brainstem Response ◽  
Type 2 Diabetic

Diabetes mellitus (DM) is one of the most modern chronic metabolic diseases in the world. Moreover, DM is one of the major causes of modern neurological diseases. In the present study, the therapeutic actions of Korean red ginseng were evaluated in type 1 and type 2 diabetic mouse models using auditory electrophysiological measurement. The comprehensive results from auditory brainstem response (ABR), auditory middle latency response (AMLR), and transient evoked otoacoustic emission (TEOAE) demonstrate auditory functional damage caused by type 1 or 2 DM. Korean red ginseng improved the hearing threshold shift, delayed latencies and signal intensity decrease in type 2 diabetic mice. Type 1 diabetic mice showed a partial improvement in decreasing amplitude and signal intensity, not significantly. We suggest that the Korean red ginseng has a more potent efficacy in hearing loss in insulin resistance type 2 diabetes than in type 1 diabetes.

High Frequency of Grossly Deleted nef Genes in HIV-1 Infected Long-Term Slow Progressors Treated with Korean Red Ginseng

2006 ◽  
Vol 4 (4) ◽  
pp. 447-457 ◽  
Author(s):  
Heungsup Sung ◽  
Hee Lee ◽  
Ji Lim ◽  
You Jung ◽  
Sun Oh ◽  
...  
Keyword(s):  
High Frequency ◽  
Red Ginseng ◽  
Korean Red Ginseng ◽  
Slow Progressors ◽  
Hiv 1

scholarly journals Effects of Korean Red Ginseng and HAART onvifGene in 10 Long-Term Slow Progressors over 20 Years: High Frequency of Deletions and G-to-A Hypermutation

2013 ◽  
Vol 2013 ◽  
pp. 1-12 ◽  
Author(s):  
Young Keol Cho ◽  
Ba Reum Kim ◽  
Mee Soo Chang ◽  
Jung Eun Kim
Keyword(s):  
Patient Group ◽  
Control Patient ◽  
High Frequency ◽  
Stop Codon ◽  
Premature Stop Codon ◽  
Current Data ◽  
Red Ginseng ◽  
Korean Red Ginseng ◽  
Slow Progressors

To investigate if Korean red ginseng (KRG) affectsvifgene, we determinedvifgene over 20 years in 10 long-term slowly progressing patients (LTSP) who were treated with KRG alone and then KRG plus HAART. We also compared these data with those of 21 control patients who did not receive KRG. Control patient group harbored only one premature stop codon (PSC) (0.9%), whereas the 10 LTSP revealed 78 defective genes (18.1%) (P<0.001). The frequency of small in-frame deletions was found to be significantly higher in patients who received KRG alone (10.5%) than 0% in the pre-KRG or control patients (P<0.01). Regarding HAART,vifgenes containing PSCs were more frequently detected in patients receiving KRG plus HAART than patients receiving KRG alone or control patients (P<0.01). In conclusion, our current data suggest that the high frequency of deletions and PSC in thevifgene is associated with KRG intake and HAART, respectively.

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