High‐intensity H− ion source with steady‐state plasma injection

1986 ◽  
Vol 57 (5) ◽  
pp. 827-830 ◽  
Author(s):  
A. I. Hershcovitch ◽  
V. J. Kovarik ◽  
K. Prelec
Keyword(s):  
Steady State ◽  
High Intensity ◽  
Ion Source ◽  
Plasma Injection ◽  
State Plasma

Particle balance in a steady state plasma in a dipole magnetic field

2019 ◽  
Vol 1 (4) ◽  
pp. 045005 ◽  
Author(s):  
Anuj Ram Baitha ◽  
Ayesha Nanda ◽  
Sargam Hunjan ◽  
Sudeep Bhattacharjee
Keyword(s):  
Magnetic Field ◽  
Steady State ◽  
Dipole Magnetic Field ◽  
Particle Balance ◽  
State Plasma

Time resolving diagnostic of the compensation process of pulsed ion beams at high-intensity light ion source

2000 ◽  
Vol 71 (2) ◽  
pp. 1107-1109 ◽  
Author(s):  
A. Jakob ◽  
P-Y. Beauvais ◽  
R. Gobin ◽  
H. Klein ◽  
J-L. LeMaire ◽  
...  
Keyword(s):  
High Intensity ◽  
Ion Beams ◽  
Ion Source ◽  
Compensation Process ◽  
High Intensity Light

Commissioning of the ECR ion source of the high intensity proton injector of the Facility for Antiproton and Ion Research (FAIR)

2018 ◽  
Vol 89 (5) ◽  
pp. 052303 ◽  
Author(s):  
O. Tuske ◽  
N. Chauvin ◽  
O. Delferriere ◽  
J. Fils ◽  
Y. Gauthier
Keyword(s):  
High Intensity ◽  
Ion Source ◽  
Ecr Ion Source ◽  
Proton Injector

Drug Interactions with Warfarin: Studies with Dichloralphenazone, Chloral Hydrate and Phenazone (Antipyrine)

1971 ◽  
Vol 40 (4) ◽  
pp. 351-364 ◽  
Author(s):  
A. Breckenridge ◽  
M. L'E. Orme ◽  
S. Thorgeirsson ◽  
D. S. Davies ◽  
R. V. Brooks
Keyword(s):  
Endoplasmic Reticulum ◽  
Steady State ◽  
Urinary Excretion ◽  
Binding Sites ◽  
Half Life ◽  
Chloral Hydrate ◽  
Maximal Velocity ◽  
Protein Binding Sites ◽  
Drug Oxidation ◽  
State Plasma

1. Administration of dichloralphenazone, a complex of chloral hydrate and phenazone (antipyrine) caused a fall in steady-state plasma warfarin concentration and loss of anticoagulant control in five subjects. 2. This effect of dichloralphenazone is due to stimulation of the drug-oxidizing enzymes of the liver endoplasmic reticulum by antipyrine, the non-hypnotic part of the complex. Administration of antipyrine caused a fall in steady-state plasma warfarin concentration in five subjects, a shortening of the plasma warfarin half-life, with increased urinary excretion of the metabolites of 14C-labelled warfarin in two subjects and increased urinary excretion of 6β-hydroxycortisol which is formed in the liver endoplasmic reticulum. 3. Administration of chloral hydrate, the hypnotic part of dichloralphenazone, caused no change in anticoagulant control but a fall in steady-state plasma warfarin concentration in five subjects. This is due to the accumulation of trichloroacetic acid which displaces warfarin from plasma protein binding sites. 4. Individual differences in the extent of enzyme induction have been shown to be related to the subjects' rates of drug oxidation. 5. In the rat administration of dichloralphenazone and antipyrine, but not chloral hydrate, caused shortening of pentobarbitone sleeping time and of the plasma [14C]pentobarbitone half-life, shortening of the zoxazolamine paralysis time and increase in the maximal velocity of N-demethylation of ethylmorphine.

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