Association of phthalate exposure with precocious and delayed pubertal timing in girls and boys: a systematic review and meta-analysis

2020 ◽  
Vol 22 (4) ◽  
pp. 873-894 ◽  
Author(s):  
Mohsen Golestanzadeh ◽  
Roya Riahi ◽  
Roya Kelishadi
Keyword(s):  
Systematic Review ◽  
Pubertal Timing ◽  
Meta Analysis ◽  
Phthalate Exposure

Herein, recent studies were evaluated to obtain regulation results in relation to puberty status and phthalate exposure in children.

Phthalate exposure and risk of diabetes mellitus: Implications from a systematic review and meta-analysis

2022 ◽  
Vol 204 ◽  
pp. 112109
Author(s):  
Hong Zhang ◽  
Yujie Ben ◽  
Yonghe Han ◽  
Yong Zhang ◽  
Yu Li ◽  
...  
Keyword(s):  
Diabetes Mellitus ◽  
Systematic Review ◽  
Meta Analysis ◽  
Phthalate Exposure

scholarly journals Biological Aging in Childhood and Adolescence Following Experiences of Threat and Deprivation: A Systematic Review and Meta-Analysis

2019 ◽  
Author(s):  
N.L. Colich ◽  
M.L. Rosen ◽  
E.S. Williams ◽  
K.A. McLaughlin
Keyword(s):  
Systematic Review ◽  
Prefrontal Cortex ◽  
Low Socioeconomic Status ◽  
Life History Theory ◽  
Pubertal Timing ◽  
Meta Analysis ◽  
Pubertal Development ◽  
Cellular Aging ◽  
Biological Aging ◽  
Childhood And Adolescence

AbstractLife history theory argues that exposure to early-life adversity (ELA) accelerates development, although existing evidence for this varies. We present a meta-analysis and systematic review testing the hypothesis that ELA involving threat (e.g., violence exposure) will be associated with accelerated biological aging across multiple metrics, whereas exposure to deprivation (e.g., neglect, institutional rearing) and low-socioeconomic status (SES) will not. We meta-analyze 46 studies (n=64,925) examining associations of ELA with pubertal timing and cellular aging (telomere length and DNA methylation age), systematically review 19 studies (n=2276) examining ELA and neural markers of accelerated development (cortical thickness and amygdala-prefrontal cortex functional connectivity) and evaluate whether associations of ELA with biological aging vary according to the nature of adversity experienced. ELA overall was associated with accelerated pubertal timing (d=-0.12) and cellular aging (d=-0.32). Moderator analysis revealed that ELA characterized by threat (d=-0.26), but not deprivation or SES, was associated with accelerated pubertal development. Similarly, exposure to threat-related ELA was associated with accelerated cellular aging (d=-0.43), but not deprivation or SES. Systematic review revealed associations between ELA and accelerated cortical thinning, with threat-related ELA consistently associated with thinning in ventromedial prefrontal cortex, and deprivation and SES associated with thinning in frontoparietal, default, and visual networks. There was no consistent association of ELA with amygdala-PFC connectivity. These findings suggest specificity in the types of early environmental experiences associated with accelerated biological aging and highlight the importance of evaluating how accelerated aging contributes to health disparities and whether this process can be mitigated through early intervention.

scholarly journals Phthalate exposure and neurodevelopment: A systematic review and meta-analysis of human epidemiological evidence

2020 ◽  
Vol 137 ◽  
pp. 105408 ◽  
Author(s):  
Elizabeth G. Radke ◽  
Joseph M. Braun ◽  
Rebecca M. Nachman ◽  
Glinda S. Cooper
Keyword(s):  
Systematic Review ◽  
Meta Analysis ◽  
Epidemiological Evidence ◽  
Phthalate Exposure

The effects of low-ratio n-6/n-3 PUFA on biomarkers of inflammation: a systematic review and meta-analysis

2021 ◽  
Author(s):  
Yali Wei ◽  
Yan Meng ◽  
Na Li ◽  
Qian Wang ◽  
Liyong Chen
Keyword(s):  
Systematic Review ◽  
Fatty Acid ◽  
Polyunsaturated Fatty Acid ◽  
Meta Analysis ◽  
Chain Polyunsaturated Fatty Acid ◽  
Inflammation Markers ◽  
Pufa Supplementation ◽  
Long Chain

The purpose of the systematic review and meta-analysis was to determine if low-ratio n-6/n-3 long-chain polyunsaturated fatty acid (PUFA) supplementation affects serum inflammation markers based on current studies.

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