A Transitory Mechanism in Free-Operant Discrimination Learning

1975 ◽  
Author(s):  
Anthony A. Wright
1964 ◽  
Vol 35 (3) ◽  
pp. 737
Author(s):  
Mae Williamson Simmons

1969 ◽  
Vol 7 (3) ◽  
pp. 492-513
Author(s):  
Richard P. Toister ◽  
J.S. Birnbrauer

1978 ◽  
Vol 13 (1) ◽  
pp. 45-61 ◽  
Author(s):  
Craig Jensen ◽  
John C. Schmitt ◽  
C. James Scheirer ◽  
Therese L. Cochrane

1967 ◽  
Vol 24 (1) ◽  
pp. 75-82 ◽  
Author(s):  
Charles N. Uhl ◽  
B. Kent Parker ◽  
Philip B. Wooton

Rats were trained on a single-stimulus, successive discrimination in a free operant situation with continuous reinforcement of responding to S+. Ss were given 0, 4, or 8 days of overtraining (OT) after reaching the discrimination criterion. Half of the Ss were given a reversal shift (RS) and half a nonreversal shift (NRS). An irrelevant stimulus dimension was present at all times in Exp. 1, and it was absent in Exp. 2. OT did not affect RS or NRS learning in either experiment. NRS learning was faster than RS learning. These results were contrasted with other studies which have reported that OT facilitated RS learning and impeded NRS learning. Certain theoretical interpretations of discrimination learning, particularly Sutherland's treatment of centrally mediated attentional mechanisms, were critically discussed in light of the present findings.


1972 ◽  
Vol 24 (4) ◽  
pp. 524-535 ◽  
Author(s):  
Anthony Dickinson

In Experiment I rats were trained preoperatively on a successive, free-operant discrimination with fixed component durations until S— responding had been minimized. Septal damage resulted in a transitory loss of discriminative performance due to an elevation of S— responding. Experiment II showed a comparable septal deficit following training which employed a “correction” procedure that penalized S— responding. It is concluded that, even after extended training, the maintenance of a low level of S— responding by rats in these types of discriminations requires the operation of an active mechanism which can be disrupted by septal damage. The possibility that this disruption results from a lesion-induced change either in the primary reactions to frustrative non-reward or in associated processes is discussed.


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