Unraveling Dendrimer Translocation Across Cell Membrane Mimics

Langmuir ◽  
2012 ◽  
Vol 28 (36) ◽  
pp. 13025-13033 ◽  
Author(s):  
Anna Åkesson ◽  
Tania K. Lind ◽  
Robert Barker ◽  
Arwel Hughes ◽  
Marité Cárdenas
2017 ◽  
Vol 5 (1) ◽  
pp. 1700453 ◽  
Author(s):  
Ariane Peyret ◽  
Emmanuel Ibarboure ◽  
Jean-François Le Meins ◽  
Sebastien Lecommandoux

2010 ◽  
Vol 402 (1) ◽  
pp. 139-153 ◽  
Author(s):  
Stephanie Tristram-Nagle ◽  
Rob Chan ◽  
Edgar Kooijman ◽  
Pradeep Uppamoochikkal ◽  
Wei Qiang ◽  
...  

2011 ◽  
Vol 100 (3) ◽  
pp. 186a
Author(s):  
Stephanie Tristram-Nagle ◽  
Rob Chan ◽  
Edgar E. Kooijman ◽  
Wei Qiang ◽  
David P. Weliky ◽  
...  

Langmuir ◽  
2018 ◽  
Vol 34 (20) ◽  
pp. 5880-5888 ◽  
Author(s):  
Peter J. Beltramo ◽  
Laura Scheidegger ◽  
Jan Vermant

2019 ◽  
Vol 116 (3) ◽  
pp. 80a
Author(s):  
Peter Beltramo

Author(s):  
M. Ashraf ◽  
L. Landa ◽  
L. Nimmo ◽  
C. M. Bloor

Following coronary artery occlusion, the myocardial cells lose intracellular enzymes that appear in the serum 3 hrs later. By this time the cells in the ischemic zone have already undergone irreversible changes, and the cell membrane permeability is variably altered in the ischemic cells. At certain stages or intervals the cell membrane changes, allowing release of cytoplasmic enzymes. To correlate the changes in cell membrane permeability with the enzyme release, we used colloidal lanthanum (La+++) as a histological permeability marker in the isolated perfused hearts. The hearts removed from sprague-Dawley rats were perfused with standard Krebs-Henseleit medium gassed with 95% O2 + 5% CO2. The hypoxic medium contained mannitol instead of dextrose and was bubbled with 95% N2 + 5% CO2. The final osmolarity of the medium was 295 M osmol, pH 7. 4.


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