Characterizing the Catecholamine Content of Single Mammalian Vesicles by Collision–Adsorption Events at an Electrode

2015 ◽  
Vol 137 (13) ◽  
pp. 4344-4346 ◽  
Author(s):  
Johan Dunevall ◽  
Hoda Fathali ◽  
Neda Najafinobar ◽  
Jelena Lovric ◽  
Joakim Wigström ◽  
...  
Keyword(s):  
Catecholamine Content

Unexpected changes in the catecholamine content of platelets and plasma during exercise

Platelets ◽  
1999 ◽  
Vol 10 (5) ◽  
pp. 312-318 ◽  
Author(s):  
N.S. Peirce, C.D. Forster, S. Heptinstal
Keyword(s):  
Catecholamine Content

Chronic CO inhalation and carotid body catecholamines: testing of hypotheses

1989 ◽  
Vol 67 (1) ◽  
pp. 239-242 ◽  
Author(s):  
S. Lahiri ◽  
D. G. Penney ◽  
A. Mokashi ◽  
K. H. Albertine
Keyword(s):  
Blood Flow ◽  
Carotid Body ◽  
Direct Action ◽  
Systemic Effect ◽  
Hypoxic Hypoxia ◽  
Tissue Blood Flow ◽  
Catecholamine Content ◽  
Carotid Bodies ◽  
Significant Stimulation ◽  
Tissue Po2

The purpose of this study was twofold: one concerns carotid blood flow and tissue PO2 and the other the effect of chronic hypoxic hypoxia on enhanced catecholamine content. The rationale was that chronic CO inhalation would not mimic the effect of hypoxia on the carotid body if its tissue blood flow is sufficiently high to counteract the effect of CO on O2 delivery and, hence, on tissue PO2. The differential effects of CO on the carotid body and erythropoietin-producing tissue would also indicate that the effect of hypoxic hypoxia on the carotid body is the result of a direct action of a local low O2 stimulus rather than secondary to a systemic effect initiated by other O2-sensing tissues. To test these alternatives we studied the effects of chronic CO inhalation on carotid body catecholamine content and hematocrit in the rats, which were exposed to an inspired PCO of 0.4–0.5 Torr at an inspired PO2 of approximately 150 Torr for 22 days. The hematocrit of CO-exposed rats was 75 +/- 1.1% compared with 48 +/- 0.7% in controls. Dopamine and norepinephrine content of the carotid bodies (per pair) was 5.88 +/- 0.91 and 3.02 +/- 0.19 ng, respectively, in the CO-exposed rats compared with 6.20 +/- 1.0 and 3.29 +/- 0.6 ng, respectively, in the controls. Protein content of the carotid bodies (per pair) was 18.4 +/- 1.6 and 20.5 +/- 0.9 micrograms, respectively. Thus, despite a vigorous erythropoietic response, the CO-exposed rats failed to show any significant stimulation of carotid body in terms of the content of either catecholamine or protein. The results suggest that carotid body tissue PO2 is not compromised by moderate carboxyhemoglobinemia because of its high tissue blood flow and that the chronic effect of hypoxic hypoxia on carotid body is direct.

scholarly journals RELATIONSHIP BETWEEN BEHAVIORS AND CATECHOLAMINE CONTENT IN PREFRONTAL CORTEX AND HIPPOCAMPUS OF CHRONICALLY STRESSED RATS

2017 ◽  
Author(s):  
Nataša Popović ◽  
Snežana B. Pajović ◽  
Vesna Stojiljković ◽  
Ana Todorović ◽  
Snežana Pejić ◽  
...  
Keyword(s):  
Prefrontal Cortex ◽  
Catecholamine Content

scholarly journals The identification of a lipid-mobilizing factor from sheep midbrain

1972 ◽  
Vol 130 (2) ◽  
pp. 467-473
Author(s):  
P. P. Shah ◽  
A. R. Tee ◽  
P. D. English ◽  
B. T. Redman ◽  
J. Bunyan
Keyword(s):  
Mass Spectrum ◽  
Lipolytic Activity ◽  
Fluorimetric Determination ◽  
Catecholamine Content

The mass spectrum of a lipolytic substance from purified sheep midbrain extract indicated that it was isoprenaline, not N-n-propylnoradrenaline, noradrenaline or adrenaline. The identification of the lipid-mobilizing factor as isoprenaline was confirmed qualitatively by t.l.c. and g.l.c. and quantitatively by determination of its lipolytic activity and u.v. absorption and by fluorimetric determination of its catecholamine content.

Sign in / Sign up

Export Citation Format

Share Document